Iziguli eziningi zihlola ngokucophelela ukutholakala kwazo kokunye okukhishwe kwamanye amadokhumenti ezokwelashwa. Imvamisa, abanesifo sikashukela abahlushwa yizifo zenhliziyo bayamangala lapho, ngaphezu kwe-arterial hypertension ne-angina pectoris, bebona ukutholakala kwe-atherosulinosis yemithambo ye-coronary.
I-Angina pectoris - lokhu kuyaqondakala, lesi sifo sihambisana nobuhlungu esifubeni; i-arterial hypertension - umfutho wegazi uyenyuka. Kodwa, yini i-coronary sclerosis, futhi yini imiphumela yalokhu kutholwa?
I-Atherossteosis yisifo esingapheli ngenxa yenkambo lapho kufakwa khona i-cholesterol plaque ezindongeni zemithambo yegazi. Ukudonswa kwamafutha kuphazamisa ukuhamba kwegazi okujwayelekile, kuholele ekuqhekekeni kwemithambo yegazi, okusongela ukubheduka kwe-myocardial, ukufa kwesifo senhliziyo.
Cabanga i-etiology ye-coronary artery atherosulinosis yenhliziyo, lesi sifo siziveza kanjani? Yikuphi ukwelashwa nokuvimbela?
Izigaba kanye nokuhlukaniswa kwe-coronary artery atherossteosis
Isifo sokuqaqamba kwamalunga emithambo yegazi sibonakala njengesifo esivame ukulingana ngokumelene nesifo sikashukela i-mellitus. Le pathology ibonakala ngokwakhiwa kwama-atherosselotic plaque ezindongeni zemithambo ye-coronary - anikezela ngegazi enhliziyweni. Ukuntuleka kokwelashwa kungaholela ezinkingeni ezinkulu zempilo nokufa.
Ngokuvamile, lesi sifo sitholakala ezigulini ezineminyaka engaphezu kwengama-45 ubudala. Kepha muva nje, ochwepheshe bezokwelapha baphawule ukuthambekela kokuvuselelwa kwenhliziyo - abesilisa nabesifazane abaningi babhekana nalesi sifo kuze kube yiminyaka engamashumi amathathu.
Ukuthuthukiswa kwe-atherosclerosis kungenxa yokuqongelelwa kwama-deposits okunamafutha ngaphakathi kwemikhumbi. Amathafa akhiwa ngento enjengamafutha, ikakhulukazi ama-lipoprotein aphansi kakhulu kanye nobukhulu obuphansi kakhulu. Amaplaque akhula kancane ngosayizi aze aqale ukungena ku-lumen of the coronary artery. Lokhu kuphazamisa ukuhamba kwegazi okugcwele kuze kube yilapho kuyeka ngokuphelele ukuphuma kwegazi.
I-coronary artery stenosis iholela ku-myocardial hypoxia, ukusebenza kahle kwemisipha yenhliziyo kwisifo sikashukela, i-IHD iba nesifo senhliziyo. Izigaba ze-atherosclerosis zemithambo yenhliziyo:
- Esigabeni sokuqala, ukuphuma kwegazi kuncipha kancane, kuvela ama-microcracks ku-endothelium yemithambo yegazi. Lokhu kuguqulwa kuholela ekwakhekeni kwama-atherosselotic plaque emthanjeni wemithambo - kukhula ibala lamafutha. Ngemuva kwalokho buthaka kwemisebenzi yesithintelo somzimba iholela ekwandeni kokuqina kokuqina kwemithambo, i-plaque iqala ukwanda ngosayizi, iphenduka ibe yi-lipid strip;
- Esigabeni sesibili, ama-plaque akhula. Kulesi sigaba sokuthuthukiswa kwalesi sifo, ukwakheka kwezigaxa zegazi akunakwa ngaphandle, okungaqhamuka bese kuvimbe ngokuphelele i-lumen;
- Esigabeni sokugcina, amadiphozithi we-cholesterol ancitshisiwe, ngoba usawoti we-calcium usabekwa. Kukhona i-stenosis yemithambo yegazi, ukuguquguquka kwayo.
Ngokuya ngezinga le-stenosis, i-atherossteosis ihlukaniswa ngokuthi ibe yi-non-stenotic (unciphise ngama-50%) kanye ne-stenotic (unciphise ngo-50% noma ngaphezulu, izimpawu zesifo sezivele zikhona).
Empeleni, ukwahlukaniswa okunjalo akubalulekile emtholampilo, ngoba abanesifo sikashukela bafuna usizo lodokotela lapho kutholakala izimpawu ezinzima zesifo senhliziyo esivele sikhona.
Izimbangela ze-coronary sclerosis
I-atherosclerosis yemithambo yenhliziyo iba ngenxa yemiphumela emibi yezici zangaphandle nezangaphakathi. Ochwepheshe bezokwelapha baveze izinto ezingaphezu kuka-200 ezingaba "ukusunduza" ekwakhiweni kwesifo esingalapheki.
Enye yezimbangela ezivame kakhulu ukwanda kwama-lipoprotein aphansi kushukela. Isimo siyanda uma onesifo sikashukela enomlando we-hypertension - ukwanda okuqhubekayo komfutho wegazi.
Into ethokozisayo ifaka phakathi umsebenzi ophansi wezimoto. I-Hypodynamia icasule ukwephulwa kwezinqubo ze-metabolic ne-metabolic, i-metabolism ye-lipids, i-carbohydrate nezinto ze-protein emzimbeni ziyaphazamiseka.
I-Etiology ye-atherosclerosis yemithambo ye-coronary yenhliziyo:
- Ukubhema. Lo mkhuba oyingozi uholela ekukhiqizweni okwandayo kwe-nitric oxide, okuphazamisa ukuhamba kwegazi, ukubhujiswa kwemithambo ye-coronary kubonisiwe;
- Ukudla okungenampilo, ikakhulukazi, ukusetshenziswa kwenqwaba yokudla okugcwele amafutha emfuyo;
- Isandulela sofuzo;
- Izinguquko ezihlobene nobudala emzimbeni. Imvamisa, i-atherosclerosis itholakala ezigulini ezineminyaka engaphezu kwengama-45 ubudala;
- Ukukhuluphala ngokweqile I-Type 2 diabetesics ikhuluphele, okunyusa ubungozi be-coronary sclerosis amahlandla ama-3;
- Ukusebenzisa kabi utshwala. I-Ethanol iphazamisa ukuhamba kwegazi, isebenza njengesici ekuqokeleleni kwamazinga aphansi we-lipoprotein emikhunjini.
Ngokwezibalo zezokwelapha, kwabesifazane besikhathi sokuzala, isifo sokuqina kwamathambo emithanjeni yamathambo asitholakali kakhulu. Lokhu kungenxa yokukhiqizwa kwe-estrogen - i-hormone yabesifazane evikela imithambo yegazi.
Kepha kokuya esikhathini, ubungozi buyakhuphuka, obuhambisana nokuphulwa kwesizinda se-hormonal.
Ukubonakaliswa kwemitholampilo kwe-stenosis ye-arterial
Esikhathini sokuqala kwenqubo ye-pathological, azikho izimpawu zesifo. Ukuxilonga lesi sifo cishe akunakwenzeka. Njengoba iqhubeka kancane kancane, kuvela izimpawu lapho izinkinga sezivele zikhona.
Kungakho ochwepheshe bezokwelapha bancomela ukuthi abanesifo sikashukela bahlolwe njalo ngonyaka ukuhlonza izifo zisencane. Izimpawu zokuqala zifaka izinhlungu endaweni yesifuba - izinhlungu zinikeza emuva noma ehlombe lasobunxele. Ngokuphikisana nesizinda sobuhlungu, ukuphefumula komoya kwenzeka.
Imvamisa, abanesifo sikashukela bakhononda ngaba nenhliziyo, ukuphefumula, isiyezi. Kodwa-ke, emidwebeni eminingi, lezi zimpawu kuthiwa zenziwa yisifo sikashukela, esibambezela ukwelashwa isikhathi esingapheli. Ngokuqhubeka kwesifo, kuboniswa izibonakaliso ezilandelayo zomtholampilo:
- I-Angina pectoris - lesi simo sihambisana nobuhlungu be-episodic endaweni yesifuba ekhula ngenxa yokusebenza ngokomzimba noma ukucindezela ngokomzwelo.
- I-Cardiossteosis - ischemia esibi yomsipha wenhliziyo, okuholela ekwakhekeni kwezindawo ze-fibrosis kulo lonke i-myocardium. I-Pathology yephula umsebenzi wesivumelwano senhliziyo.
- I-Arrhasmia ibonakaliswa ngenxa yomonakalo emisipha yenhliziyo, kukhona ukuncipha kokuhanjiswa kwesimilo.
Lapho i-atherosclerotic plaque igqashuka emthanjeni we-coronary, kuba nenhliziyo yesifo sikashukela. Imvamisa, lesi simo senzeka kusuka ku-4,00 kuye ku-10 :00 ekuseni, lapho ukugcwala kwe-adrenaline kukhuphuka ohlelweni lokujikeleza kwegazi.
Ku-50% yamacala, kuvela izimpawu ezingenhla, okuyi-harbinger yokuquleka.
Ukwelashwa okuqondayo nokuhlinzwa
Ukwelashwa kwezidakamizwa kufanele kuklanyelwe ukuze ukwelashwa kusebenze emikhombeni eminingana ngesikhathi esisodwa. Okokuqala, kuyadingeka ukuthonya i-pathological uqobo - inqubo ye-atherosulinotic emzimbeni, kanye nezinga lomtholampilo wesifo, ukuvimbela ukuqhubeka kwesifo senhliziyo.
Amaqhinga okwelapha anqunywa yisigaba salesi sifo. Emabangeni okuqala, aboshukela bayelulekwa ukuthi bashintshe indlela yabo yokuphila. Kuyadingeka ukushiya ngokuphelele imikhuba eyingozi - ukuphuza utshwala, ukubhema. Kubalulekile ukujwayezisa umsoco, ukulandela ukudla - ukunciphisa ukusetshenziswa kwamafutha ezilwane, ukwenqaba ukudla okunamafutha / okuthosiwe / okunezipayisi.
Ukulungisa inqubo ye-metabolic kanye ne-metabolic, ochwepheshe bezenhliziyo banquma ukusebenza okuhle komzimba. Umdlalo ukhethwa kucatshangelwa ama-anamnesis, ubudala, inhlala-kahle yesiguli. Ukuze ukhuluphele, kufanele wehlise isisindo.
Ukwelashwa kwe-atherosclerosis of aronal artery, imithi ingabekwa:
- Imithi, umphumela we-pharmacological ogxile ekunciphiseni umfutho we-oksijeni womoya, osiza ekuphambukiseni izimpawu ezingezinhle zesifo senhliziyo. Nikeza abavimbeli besiteshi se-calcium, ama-angiotensin-abaguqula ama-enzyme inhibitors, ama-antiplatelet agents;
- Imithi evimbela ukuthuthukiswa kwe-atherosclerosis. Faka izibhebhe zeqembu lama-statin. Banciphisa ukugcwala kwe-cholesterol ye-LDL, vimbela ukwakheka kwama-atherosclerotic plaque kushukela.
Ukwelashwa kwe-atherosclerosis kubandakanya ukususwa kwezici eziholela ekwakhiweni kwesifo esingalapheki. Isibonelo, ku-mellitus yesifo sikashukela, kuyadingeka ukuthola isinxephezelo esizinzile sesifo, ukugcina izinga elifanele likashukela emzimbeni.
Ezimweni ezisezingeni eliphakeme, lapho ukwelashwa kwezidakamizwa kunganikeli umphumela owafunayo wokwelapha, phendukela ekungeneleleni kokuhlinzwa:
- I-Coronary artery idlula ngokuxhunyelelwa. Ngesikhathi sokuhlinzwa, udokotela udala ukuthi kusetshenziselwe ukugeleza kwegazi, edlula endaweni elimele.
- I-Balloon angioplasty. Kufakwa i-catheter ekhethekile ngaphakathi kwethambo labesifazane, ngemuva kwalokho ithuthukiselwe endaweni oyifunayo. Ngemuva kwalokho ibhaluni liyakhuphuka, okufaka isandla ekunwetshisweni komthambo we-coronary artery.
- Isikhuxu seCoronary. Ukuzikhohlisa kwezokwelapha kufaka ukwethulwa kwesiqu esinohlaka oluqinile ku-artery ethintekile.
Ungangezela ukwelashwa ngemithi ye-homeopathic. I-Homeopathy inikeza izinhlobo ezahlukahlukene zemikhiqizo ezisiza ukuqeda ama-cholesterol plaque. Izidakamizwa ezisebenza kahle kakhulu ze-atherossteosis zifaka iHolvacor, Cholesterolum, Pulsatilla.
Ukwelashwa kwe-homeopathic kwenziwa ngaphansi kokulawulwa kwe-homeopath okwazi ukuhlola ukusebenza ngempumelelo kwezindlela zokwelapha futhi, uma kunesidingo, kulungiswa uhlobo lokuwelapha.
Izinkinga kanye nokuvimbela okungenzeka
I-atherosclerosis yemikhumbi ye-coronary icasula ukubhujiswa kwemisipha yenhliziyo. Ngokwempilo, lokhu kubonakaliswa yisifo senhliziyo, i-angina pectoris, ukuphazamiseka kwesigqi senhliziyo. Izimpawu zokuhluleka kwenhliziyo zitholwa ngezikhathi ezithile.
Uma ama-cholesterol plaques eshaya imikhumbi eminingana ngasikhathi sinye, khona-ke lokhu kwandisa ingozi yokufa kwesifo sikashukela i-mellitus. Kukhona futhi amathuba aphezulu okufa ngenxa yokuqhuma kwe-plaque. Imvamisa kwenzeka kakhulu ngenkathi ebandayo ekuseni. I-Provocateur - ukucindezela ngokweqile noma ukuvivinya umzimba ngokweqile.
Lapho i-clot yegazi yakheka, ehlanganisa umthambo we-coronary, ingozi yokufa iphakeme. Izibalo ziyazi ukuthi ezimweni ezingama-60% isiguli asinaso isikhathi sokuyisa esibhedlela - uyafa. Ngomonakalo oyingxenye, i-angina pectoris iyenzeka. Imvamisa infarction yenzeka njalo; Izimpawu zayo zimi ngalendlela elandelayo:
- Ubuhlungu obukhali endaweni yesifuba - bubeza emuva;
- Yehlisa umfutho wegazi;
- Ukungazi kahle;
- Ukuphefumula.
Ngalezi zimpawu, ukunakekelwa kwezokwelashwa ngokushesha kuyadingeka. Enye inkinga yi-cardiossteosis. I-Pathology ibonakala ngokushintshwa kwamaseli ajwayelekile ngezicubu zesikhafu. Izicubu ezinjalo azibambi ekuqhekekeni kwenhliziyo, okuholela emthwalweni owandayo ku-myocardium.
Ukuvikela Isifo Sikashukela:
- Ukuqapha nsuku zonke ushukela wegazi, umfutho wegazi, i-cholesterol embi.
- I-Naturalization yesisindo somzimba ngokusebenzisa umsoco kanye nemidlalo.
- Ukudla okulinganiselayo, kucatshangelwa okuqukethwe yi-cholesterol ekudleni, inkomba ye-glycemic.
- Ukubambelela ngokuqinile kuzo zonke izincomo zikadokotela.
- Ukuzivocavoca okulinganiselayo (ukubhukuda, ukuhamba, ukugijima, i-aerobics).
- Ukwelashwa ngesikhathi kwezifo ezithathelwanayo.
- Izivivinyo zokuzivikela.
Ukufa kwabantu ngenxa yesifo senhliziyo sikashukela kuphezulu impela - empeleni, yimbangela eyinhloko yokufa kwabantu abaneminyaka engaphezu kwengama-50. Izinga lempilo ekulesi simo lincike ngokuphelele entandweni yesiguli: esifisweni sakhe sokuphila impilo ende futhi enempilo.
I-Hypertension kanye ne-atherosclerosis kuchazwe kuvidiyo kule ndatshana.
