Imininingwane

Iziguli ezingaphezu kwesigidi ezingama-415 ezinesifo sikashukela emhlabeni, ezingaphezu kwezigidi ezine ezweni laseRussia, kanye nabanesifo sikashukela okungenani abangama-35,000 esifundeni sase-Astrakhan - lezi yizibalo ezidumazayo zezigameko zesifo sikashukela, esanda minyaka yonke. Kwenziwa ini esifundeni ukuvikela nokwelashwa kwalesi sifo, yiziphi imicimbi ezenziwayo kanye nokuthi hlobo luni lwezinzuzo abanesifo sikashukela?

I-Arteriossteosis ingukuqina, ukuqina nokulahleka kwe-elasticity ngezindonga zemikhumbi ye-arterial yesistimu yokujikeleza kwegazi. Lokhu kudalwa kokukhula kwengqondo kwenzeka ngenxa yokwakheka kwama-cholesterol ekubunjweni okungaphakathi kwezindonga zemithambo yegazi. Ngenxa yalokhu, kunokuvinjelwa kancane kancane kokugeleza kwegazi kwizitho nezicubu zangaphakathi.

Cabanga ngombuzo ofanele ofanele - ngabe i-cholesterol fat, noma cha? Ukuyiqonda, kufanele icaciswe ukuthi le nto iqukethe ukwakheka kwe-plasma yegazi, ngendlela yezakhiwo eziyinkimbinkimbi ezinamaprotheni ezokuhambisa. Ubuningi bekhompiyutha bukhiqizwa ngumzimba ngokwawo usebenzisa amaseli wesibindi.

Ukuxilongwa kwe-hypertension kwesinye isikhathi kungenziwa ngephutha, isiguli sithatha ukwelashwa isikhathi eside, kepha akulethi umphumela. Iziguli zilahlekelwa ukholo ekwenzeni intuthuko yazo, futhi kancane kancane ziba nezinkinga eziningi eziyingozi. Cishe i-15% yamacala okwehla kwengcindezi yegazi ahambisana ne-hypertension yezimpawu ezibangelwa yi-pathologies yezitho zangaphakathi ezithintekayo kulawulo lokucindezela.

I-Atherossteosis iyindlela yokuziphatha ebucayi, enomphumela omubi ohlelweni lonke lwemikhumbi yabantu. Ngokukhula kakhulu kwalesi sifo, maningi amathuba okufa noma okhubazekile. Enye yezindlela eziyingozi zalesi sifo i-multifocal atherosulinosis, ngokuthuthukiswa kwayo lapho kunqotshwa khona hhayi kweqembu elilodwa lemikhumbi, kodwa eminingana.

Isifo sokuqina kwenhliziyo siyindlela yokuthinteka lapho kuthinteka khona imithambo yemithambo yegazi. Lokhu kuholela ekungasebenzi kahle ekunikezelweni kwegazi kuyi-myocardium. I-atherossteosis yimbangela evame kakhulu yokufa. Imvamisa, lesi sifo sihlaselwa isifo sikashukela i-mellitus, njenge-complication ye-hyperglycemia engapheli. Ukwelashwa kwalesi sifo kufanele kube okufika ngesikhathi, okuphelele futhi kube isikhathi eside.

I-cholesterol iyingxenye ebalulekile yomzimba womuntu nezilwane. Umuthi ubandakanyeka ezinqubweni eziningi zokuphila, uqukethe ulwelwesi lwamaseli, ukhuthaza ukukhiqizwa kwama-hormone ocansi kanye nokufakwa kwamavithamini athile. I-cholesterol ephezulu ivame ukutholakala nesifo sikashukela sohlobo 2.

I-Arterial hypertension ivame ukubizwa ngokuthi umbulali othule, ngoba lesi sifo sihlala isikhathi eside ngaphandle kwezimpawu. I-Pathology ibonakaliswa izinga eliphakeme lomfutho wegazi lapho i-systolic ingaphezulu kwe-140 mm Hg. Ubuciko., Diastolic ngaphezu kwe-90 mm RT. Ubuciko. Ngokwezibalo, umfutho wegazi ophakeme uthinta amadoda aze afike eminyakeni engu-45 futhi abesifazane emva kokuya esikhathini.

I-Atherossteosis ingukuphazamiseka kwengqondo okuhambisana nokwakhiwa kwama-cholesterol deposits ebusweni bangaphakathi bezindonga zemikhumbi ye-arterial system yojikelezo lwegazi. Ehlelweni lokuqhubeka, ukwanda kwezicubu ezixhunyiwe kanye nokwakhiwa kwezigcawu ze-atherosulinotic kwenzeka. Njengomphumela wenqubo ye-pathological, i-lumen yemikhumbi iyachichima, okuholela ekunikezelweni kwegazi okungalungisiwe kwezicubu nezitho zomzimba.

Esakhiweni sobumba kukhona isibalo esikhulu semithambo yegazi. Umsebenzi wabo ukuqinisekisa ukuthi isitho sigcwaliswa ngegazi futhi silethwe esimweni sokukhula phakathi kokuvuka kocansi. Uma kwenzeka ukwephulwa ohlelweni lokujikeleza lwesitho sangasese sowesilisa, kuyehla ukwehla kwe-potency.

Izifo zesistimu yenhliziyo, esekelwe kwizibalo zomhlaba, zihlala endaweni yokuqala ngokuya ngokufa. Lolu hlu lwezifo nama-pathologies luhlanganisa ukuhlaselwa yisifo senhliziyo, imivimbo, i-occlusion ye-arterial, i-gangrene, ischemia necrosis. Imvamisa, bonke banesizathu esisodwa, esifihliwe ezingeni elandayo lama-lipids egazi.

I-atherossteosis iyaqhubeka nokuba ngesinye sezifo zokucwaninga eziphambili. Inani elikhulu lezifo ezinokubonakaliswa okuhluke ngokuphelele kwemitholampilo kuhlotshaniswa ne-atherosclerosis, futhi phakathi kwabo infarction myocardial; isifo sohlangothi; ama-aneurysms wesisu; ischemia ephansi lomlenze. Zinquma ikakhulukazi ukungasebenzi komzimba kanye nokufa.

I-cholesterol yinto engamanzi engabonakali engxenyeni yeseli yomzimba womuntu, enendima ebonakalayo empilweni ephelele. Iyancibilika emafutheni kanye nama-soltiv organic. Iningi likhiqizwa izitho zomuntu ngokwazo, futhi ngamaphesenti angama-20 kuphela angena emzimbeni ngemikhiqizo edliwayo.

I-Atherossteosis yisifo esithinta imithambo yezinhlobo ezinwebeka futhi izicubu, sibaphuca izimfanelo zabo zemvelo zokwenza umsebenzi othola ukwethuka kanye nokuqina kwegazi. Kulokhu, i-fat-protein detritus inqwabelana odongeni lomkhumbi, namafomu e-plaque. I-plaque ephumela lapho iyakhula ngokushesha futhi ikhule, ikhuphuka ngokweqile kwegazi kuze kube yilapho ivimba ngokuphelele.

Iningi labantu liqinisekile ukuthi umfutho wegazi ophakeme ungenye yezimpawu zokuqamba kwe-atherosulinosis, kodwa empeleni lokhu akunjalo. Njengoba izazi zesayensi yezenhliziyo zanamuhla ziphawula, umfutho wegazi ophakeme uyimbangela eyinhloko ye-atherosclerosis, hhayi umphumela wayo. Iqiniso ngukuthi nomfutho wegazi ophakeme, kuvela imicrodamage ezindongeni zemithambo yegazi, bese igcwaliswa nge-cholesterol, enomthelela ekwakhekeni kwama-cholesterol plaques.

Kubantu asebekhulile nabadala, kunengozi enkulu yokuba nesifo senhliziyo. I-pathology enjalo iyingozi ekuthuthukisweni kwe-myocardial infarction, okugcina kube imbangela yezinguquko ezingenakulungiswa. Eminye yemiphumela yokuhlaselwa yi-atherosclerotic post-infarction cardiossteosis. Le yinkimbinkimbi kakhulu yesifo senhliziyo esijwayelekile, okuvame ukuthi ngemuva kokuhlupheka kwenhliziyo ihlasele ekufeni komuntu.

Izifo eziningi ziwumphumela wokungondleki kanye nemikhuba emibi. Ngenxa yalokhu, izinto eziwusizo empeleni azingeni emzimbeni, ngenxa yalokho iba sengozini futhi amasistimu ayo awakwazi ukuphendula izifo. Ngakho-ke, i-atherossteosis namavithamini axhumekile, ngoba ukuhlinzeka ngomzimba ngamavithamini namaminerali awusizo, umphumela wawo wehla kancane.

I-Atherosclerosis kanye ne-pathology eyibangayo ngabaholi phakathi kwezifo ezibulalayo. Lesi sifo sibonakaliswa ngokufakwa kwe-cholesterol ezindongeni zemithambo yegazi, okugcina kuba yicwecwe le-atherosclerotic. Le nto ayilapheki. Ngokuhamba kwesikhathi, izindawo eziqinile ziqinile ngenxa yokungakwazi kwe-cholesterol ukuncibilika emanzini.

I-Atherossteosis yisifo esingelapheki eside senhliziyo nemikhumbi emikhulu, ebonakala ngokulimala kwezindonga zemithambo nokufakwa kwezixuku ze-atheromatous kuso ngokuvalwa okwengeziwe kwe-lumen nokukhula kwezinkinga ezivela ebuchosheni, enhliziyweni, izinso, emikhawulweni ephansi. Lesi sifo uqobo senzeka ikakhulukazi kwabaluphele, yize manje kufakwa i-cholesterol encane ezindongeni zemithambo yegazi ngisho nasezinganeni nasebasha.

Ukusebenza kahle kwengqondo isihluthulelo sempilo yomzimba wonke. Nguwo lo mzimba ohlinzeka futhi olawula ukusebenza okwanele kwazo zonke ezinye izitho nezinhlelo. Emhlabeni wonke, izifo ezivame kakhulu zobuchopho zingama-vascular, futhi phakathi kwazo isikhundla esiphambili kungokwalapho isifo sezitho zangasese.