I-atherossteosis ibhekwa njengenye yezindlela ezivame kakhulu zenhliziyo. Udinga nje ukucabanga ngalezi zibalo: imiphumela ebulalayo evela ezifweni ezihambisana nohlelo lokujikeleza eRussia Federation ingama-800,9 kubantu abayizinkulungwane eziyikhulu, lapho eJapan - 187,4, naseFrance - 182.8.
Ukwanda kwenani leziguli ezine-atherossteosis ikakhulu kuhlotshaniswa nendlela yokuphila engasebenzi kanye nokungondleki. Ukuxilongwa kusenesikhathi kwe-pathology kusiza ukuvikela imiphumela emibi - ukuthuthukiswa kokuhluleka kwenhliziyo, i-hypoxia, ischemia, njll. Ukwelashwa kufaka izakhi eziningana: ukuphuza umuthi, ukwelashwa kokudla, futhi ezimweni ezimbi kakhulu, ngisho nokuhlinzwa.
Kuyini isifo sokuqina kwemizwa?
Abaningi bazwile ngalesi sifo, kodwa lapho babuzwa ukuthi yini i-atherosclerosis, hhayi wonke umuntu onganikeza impendulo efanele. Lesi sifo yisilonda semikhumbi yohlobo lwe-musical-elastic and nokunwebeka ngama-atheromatous plaque, okuyindlela yokuqongelela i-cholesterol nezinye izingxenyana zezakhi ezikhethekile zamaprotheni - lipoprotein. I-Pathology iba ngenxa yokulimala kwe-lipid ne-protein metabolism.
Kunezinhlobo eziningi zama-lipoprotein aphatha i-cholesterol emthanjeni wegazi futhi awudlulisele kuzo zonke izakhiwo zeselula: i-high density (HDL), i-low density (LDL) kanye ne-density ephansi kakhulu (VLDL). Ukuvela kwama-atherosclerotic plaque kuhlotshaniswa ngqo ne-predominance ye-LDL ne-VLDL emzimbeni, okwaveza umqondo we-cholesterol "embi". Lezi zingxube azincibilikisi uketshezi, ngakho-ke, inani lazo ngokweqile egazini liholela ekubhekeni kokudilizwa kuqala ngesimo sezindawo ezinamafutha, bese kuba nama-plagi atheromatous.
I-HDL ("enhle" cholesterol) isebenza ngendlela ehluke ngokuphelele: incibilika kahle egazini lomuntu, ngakho-ke izinga labo eliphakeme libonisa impilo enhle. Ukuba khona kwenqwaba yama-HDL kuvimbela ukuthuthukiswa kwamathafa we-atherosulinotic nokukhula, okuholela ekulahlekelweni kokuqina komkhumbi, ukuwohloka nokuqhekeka.
Namuhla akukho kuvumelana ngemvelaphi yalesi sifo. Ezokwelapha ziyazi imibono eminingi:
- ukuboniswa kwama-lipoproteins ezindongeni zezindawo;
- ukwephulwa komsebenzi wokuvikela we-endothelium (ungqimba lwangaphakathi lwamangqamuzana) nabameli bawo;
- ukuvela kwesisekelo sendlala yama-musulin abushelelezi;
- ukungasebenzi kahle kohlelo lwe-antioxidant;
- ukungasebenzi kwe-leukocytes nama-macrophage, ukungena kwawo odongeni lwe-vascular;
- ukulimala kwe-endothelial nge-cytomegalovirus, i-herpes, njll;
- ubukhona besici esiyindlalifa odongeni lwesitsha;
- ukulimala kwezindonga ze-artery nge-chlamydia;
- izinguquko ze-hormonal ezihambisana nobudala.
Muva nje, izigameko ze-atherossteosis sezidlulile isibalo samacala okulimala, izifo ezithathelwanayo kanye neoplasms enobungozi.
Imvamisa, kutholakala ukuthi uneminyaka engama-45-50, kanti inani leziguli zesilisa liphindwe izikhathi ezingama-3-4 kunowesifazane.
Izici ezifaka isandla ekuthuthukiseni lesi sifo
Kunezici eziningi ezandisa amathuba okuthuthukisa isifo sokuqina kwemithambo yegazi.
Kuze kube manje, iwebhusayithi ye-European Cardiology Society idaliwe, lapho ungabala khona ubungozi be-pathology ye-inhliziyo.
Lezi zimo ezilandelayo kanye nezifo zidlala indima enkulu ekubekweni kwamathafa atheromatous.
Ukubhema. Ama-resins ne-nicotine, ayingxenye yokwakhiwa, athinta kabi izindonga ze-vascular. Ukubhema isikhathi eside kukhulisa amathuba okuba nesifo senhliziyo, i-hypertension ne-hyperlipidemia.
Hyperlipoproteinemia. Ukwanda kwama-lipids egazi nama-lipoprotein kuyinto evame ukwenzeka. Kuyadingeka ukukhalisa i-alamu lapho ukugcotshwa kwe-cholesterol ephelele kukhushulwe ngaphezu kwe-5 mmol / L ne-LDL ingaphezu kwe-3 mmol / L.
Umfutho wegazi ophezulu we-Arterial. Ngomfutho wegazi okhuphuka njalo (ngaphezu kwe-140/90 mm Hg), ukuqina kwemithambo kwehla futhi amathuba okuncipha kwama-atheromatous plaques ayanda.
Isifo sikashukela mellitus. Lesi sifo sibonisa ngokwanda kwe-glucose ngenxa yokukhiqizwa okunganele noma ukunqamuka ngokuphelele kokukhiqizwa kwe-insulin. Inkambo ende ye-pathology ithinta kabi isimo semithambo yegazi nokuphela kwezinzwa, ngakho-ke isifo sokuqina kwezitho zofuzo kwesinye isikhathi sivela ngokuvela ngemuva kwesifo sikashukela.
Ukuntuleka kokuzivocavoca. Nsuku zonke, umuntu udinga ukuthatha uhambo emoyeni ohlanzekile nokuvivinya umzimba. Umsebenzi ophansi uholela ekuphazamisekeni kwe-metabolic, okuholela ekukhuleni isisindo, isifo sikashukela kanye ne-atherosulinosis.
Ukukhuluphala nemikhuba emibi yokudla. Ngokukhuluphala ngokweqile, kukhona ukwanda kwe-lumen yemithambo yegazi nokukhuphuka komfutho wegazi. Umthwalo onjalo ku-vascular system ufaka ama-pathologies ahlukahlukene. Ukuba khona ekudleni kwenani elikhulu lamafutha ezilwane kunomthelela ekuhlulekeni kwe-lipid metabolism.
Iminyaka nobulili. Lapho umuntu eba umdala, kuphakama amathuba okuba nesifo sokuqina kwemithambo yegazi. Ukuqala kokuvezwa kwamathafa atheromatous kwenzeka eneminyaka engama-45-50 yeminyaka. Ngaphezu kwalokho, emadodeni lokhu kutholwa kwenziwa kaningi kaningi, futhi lesi sifo uqobo sikhula eminyakeni eyi-10 ngaphambi kwengxenyana efanelekile.
Isakhi sofuzo. I-atherossteosis ijwayele ukutholakala phambi kwezihlobo ezine-pathology efanayo. Kufakazelwe ngokwesayensi ukuthi abantu abanokuthambekela kokulimaza i-lipid metabolism bane-atherosulinosis yokuqala (ngaphansi kweminyaka engama-50).
Ezinye izimbangela yi-hypothyroidism, postmenopause, hyperfibrinogenemia (amazinga aphezulu e-fibrinogen egazini), i-homocysteinemia (amazinga aphezulu e-homocysteine egazini) kanye ne-homocysteinuria (ukuba khona kwe-Homocysteine emchameni).
Indlela yokuthuthukiswa kwe-atherosulinosis
Esezinhlelweni zokufunda lesi sifo, kwasungulwa ukuthi kunezigaba zokuthuthuka kwalesi sifo ezahluka ngezibonakaliso ze-pathophysiological.
Ukuqhubeka kwalesi sifo kwenzeka ngezigaba ezintathu - ukunqwabelana kwamabala e-lipid, ukuqina kwe-lipid nokuthuthukiswa kwezinkinga.
Kwephulwa kwe-lipid metabolism, kunokushintshwa kwesilinganiso se-cholesterol, phospholipids, amaprotheni nokwakheka ngokweqile kwe-beta-lipoproteins.
Kunesidingo sokuhlolwa okuningiliziwe kwe-pathogenesis ye-atherosulinosis:
- Ukuqongelelwa kwezindawo ze-lipid. Kulesi sigaba, umuntu akaziboni izimpawu, engazi ngalesi sifo. Kodwa-ke, inqubo ye-pathological isivele isiqalile: kunokushintshwa okuguquguqukayo ezindongeni zemithambo yegazi, ekubukeni okufana nemichilo ephuzi nobude bayo bonke bomthambo. Ngaphezu kwalokho, izingxenye ezithile zegazi ziyathinteka. Ukuqhubekela phambili kwe-pathology kusheshiswa izifo ezihlangene.
- Ukuqongelela i-Lipid. Isakhiwo sezicubu esingaphansi kwemichilo ye-lipid siqala ukufuthelana, okuwukuphendula komzimba kumuntu ongacabangi. Ngokuhamba kwesikhathi, ukugxila kokuvuvukala kuholela ekubumbeni kongqimba lwamafutha nokukhula kwezicubu. Ngakho-ke, ukunqwabelana kwamafutha kuqala ukubethela nokukhuphuka ngaphezu kodonga lomkhumbi.
- Ukuthuthukiswa kwezinkinga. Isimo sokuqhubeka kwezifo sinokukhetha ezimbili - ukuqhuma kwe-cholesterol plaque noma ukwakheka kwezihibe zegazi. Lapho ukuqhuma kwe-plaque atheromatous, ukwakheka kwama-deposits amasha noma ukukhululwa kwegazi elikhulu kungenzeka. Lapho ukuqhekeka kwegazi kwakha, ubunzima obuyingozi kakhulu ukuvimbela imithambo emikhulu, okuholele ekushayweni, i-necrosis yesakhiwo sezicubu kanye ne-gangrene.
Akunakwenzeka ukubikezela ukuthi lesi sifo sikhula ngokushesha kangakanani: kungaqhubeka ngokulingana noma ngokushesha okukhulu. Indlela yokuthuthukiswa kwalesi sifo idlula isuka ezinyangeni eziningana iye eminyakeni eminingana.
Lokhu kuthonywa yizici ngazinye zezinqubo ze-metabolic, ukuba khona kokulawulwa kofuzo nezinye izici.
Izinhlobo kanye nezimpawu ze-atherosulinosis
Ukuhlukaniswa okuvame kakhulu kwalesi sifo ngokuya ngendawo ethintekile yemithambo enama-atheromatous deposits.
Izinhlobo ze-pathology zingazibonakalisa, yize ukulimala okuhleliwe kwimithambo yemithambo yegazi kuvame ukubonwa.
Ngaphezu kwalokho, izimpawu ze-atherosclerosis ziyehluka ngokuya ngohlobo lwazo.
Kuyadingeka ukuhlukanisa izinhlobo ezilandelayo zesifo:
- Isifo sokuqina kwemithambo yegazi. Ngomonakalo wemithambo yegazi yenhliziyo, imiphumela engenzeka efana nesifo senhliziyo, isifo senhliziyo ne-angina pectoris. Izikhalazo zesiguli zihambisana nobuhlungu benhliziyo obuqhamukela ekhwebeni noma kulo lonke ubude bengalo, kufiphalaza emunweni, emizweni ebuhlungu lapho uphefumula, kuphefumula, ukufushane komoya, ukuhlanza noma isicanucanu, ubuhlungu emuva, ukuzwa ukubanda, ukujuluka nokujuluka, ubuthakathaka emilenzeni.
- I-Atherosulinosis yemikhumbi ye-brachiocentral (BCC). Liziveza ikakhulu isiyezi lapho liguqula indawo, i-migraine, ukushona kwemilenze nezingalo, ukufenda, umbono ofiphele, "izimpukane noma amachashazi" phambi kwamehlo.
- Ukwehlukanisa i-atherosulinosis yemikhawulo engezansi nengaphezulu. Isibonakaliso sokuqala sokugula umuzwa wokuphola kanye "namagogo" emilenzeni nasezingalweni. Isici esithile futhi siyisikhumba esiphaphathekile. Esikhathini esisekupheleni kwesikhathi, kubuhlungu emilenzeni, ukushayeka kwezinwele emaphethelweni, izilonda eziyinqwaba, ukuvuvukala, ubomvu beminwe, esimweni esibi kakhulu, ukuthuthukiswa kwe-necrosis kuyabonakala.
- Ifomu le-aortic. I-atherosclerotic lesion ye-artery enkulu kunazo zonke ithinta ukusebenza kwazo zonke izinhlelo ze-organ. Kwezinye iziguli, usawoti we-calcium uqongelela isizinda sefomu le-aortic le-atherosclerosis. Ngokuhamba kwesikhathi, inqubo ye-pathological iholela ku-sclerosis of cusps ye-valve kanye nokunciphisa indandatho ye-fibrous valve. Isibonakaliso esiyingozi kakhulu salesi sifo i-stenosis ye-aortic orifice.
- I-Cerebral atherosulinosis yemikhumbi yokuqwasha. Ngama-atherosclerosis asabalalisa, i-cephalgia iyakhula, i.e. ukuphathwa ikhanda ngaphandle kwendawo ngqo yemvelo yokuqhuma, ukuqwasha, i-tinnitus, izinkinga nge-spatial oriental, ukuxhumana, ubuthi, ukuguqulwa kobuntu, ukukhuluma okungahambi kahle, ukuphefumula kanye nokudla. Esigabeni sokugcina, kufikelwa yi-senile, dementia, kanye nokwehla kobuhlakani.
Ngokwehlukana, ibuye ihlukanise uhlobo olunjalo lwesifo njenge-atherosclerosis of the renal artery. Ukuvela kwama-cholesterol plaques kufaka izinhlungu ngesikhathi sokuchama, ukuhlanganiswa kwegazi kuyabonakala kumchamo. Isiguli sikhononda kaningi isicanucanu nokuhlanza, izinhlungu esiswini kanye naphansi emuva.
Esifundweni eselabhorethri, iqoqo le-potassium esegazini liyabonakala.
Ubudlelwano be-atherosclerosis nesifo sikashukela
I-cholesterol ibandakanyeka ekuhlanganiseni kwamajusi wokugaya kanye nama-hormone e-pancreatic. Le khompiyutha akuyona imbangela yesifo sikashukela, kepha ithinta inkambo yayo.
Ngamazinga kashukela akhuphukile, amathuba okukhula kwe-atherosclerosis akhuphuka kakhulu. Ngaphezu kwalokho, i-atherossteosis yandisa ushukela wesifo sikashukela. Isifo sikashukela, kungakhathalekile ubulili nobudala, sihlushwa izilonda ze-atheromatous vascular ngokulinganayo.
I-atherossteosis ingaqhamuka ngemuva kwesifo sikashukela esincike ku-insulin futhi esinga-insulin. Ngokuqukethwe kweglucose ephezulu, umzimba uyaphazamiseka, okuholela ekwehlulekeni kwe-lipid metabolism nokugaya. Izindonga ze-vascular ziba buthaka futhi zitholakale ngokungena kwe-cholesterol "embi" ukungena egazini.
Uma i-atherossteosis ihambisana nesifo sikashukela sohlobo lokuqala noma lwesibili, isiguli singathola lezi zinkinga ezilandelayo:
- i-pathology ye-inhliziyo ikhula esemncane, naphezu kweqiniso lokuthi ihlala itholakala eminyakeni engaphezu kwengu-45-50;
- ukuvaleka kwemithambo kwenzeka, iba ntekenteke kakhulu futhi inciphile, maqondana nalokhu, amathuba okushaywa unhlangothi nokukhula kwe-aneurysms kuyanda.
I-Atherossteosis, njengomthetho, iba ngokuhlelekile, okuyingozi kobuchopho, izilenge, inhliziyo nemikhumbi emikhulu.
Izinkinga zokwelashwa okungasebenzi
Ukungasebenzi kwesineke noma ukwelashwa okungasebenzi kuholela ezinkingeni ezahlukahlukene. Njengoba isithombe somtholampilo sihlala singacacisiwe ezigabeni zokuqala zokukhula kwalesi sifo, ukuxilongwa ngesikhathi kufeza indima ebalulekile.
I-atherossteosis yesistimu yemithambo yegazi futhi iphazamisa ukushintshwa kwemithambo phezu kwesikhathi ifaka ukwehluleka kokuqina kwemithambo yethambo noma okubi kakhulu. Inqubo ye-pathological yenzeka ngokumelene nesizinda se-stenosis esilinganisayo se-lumen yemikhumbi.
Ukuntuleka okungapheli kokuhlinzekelwa kwegazi ezithweni ezilandela ukubangela i-hypoxia, ischemia, atrophy kanye ne-dystrophy, ukuvela kwesifo sokuqina okuyisisekelo, kanye nokusakazeka kwezicubu ezithintekayo.
Njengomphumela wenkambo ende yokwentuleka kwemithambo ye-acute, ukuvaleka kwemithambo nge-thrombus noma i-embolus kwenzeka - izinhlayiya ze-plaque eqhumayo. Lesi simo sikhonjiswa yizimpawu zokuhlaselwa yisifo senhliziyo kanye ne-ischemia esibuhlungu.
Umphumela oyingozi kakhulu enkambisweni ye-atherosclerosis ukuqhekeka kwe-aneurysm yomkhumbi. Kwezinye izimo, lokhu kuholela ekufeni.
Ukuze uvikele imiphumela emibi, kuyadingeka ukulandela indlela odla ngayo, uphuze umuthi onqunyelwe bese ulandela yonke imiyalo kadokotela.
Imigomo yokuhlonza lesi sifo
Ukuxilongwa kwalesi sifo kufaka izindlela eziningi zelebhu kanye nezinsizakalo.
Okokuqala, isiguli siza kudokotela ayohlolwa, eqoqa idatha ye-anamnesis.
Uchwepheshe onolwazi udonsela ukunaka kokungabikho kwezinwele emaphethelweni, ipuleti lezipikili elonakalisiwe, ukwehla kwesisindo sesiguli, ukukhuphuka kwengcindezi yegazi, ukuba khona kokububula kwenhliziyo, ukuqina kokuqina, ukukhuluphala ngokweqile kwezindlala ze-sebaceous nezithukuthuku, ukuvuvukala ngokungabikho kwe-pathologies ye-renal.
Uma udokotela esola i-atherosclerosis esigulini, umyalela ukuba ahlolwe futhi afundise:
- Amasampula egazi avela emthanjeni ukuthola inani eliphelele le-cholesterol ne-atherogenicity.
- I-Aortography yezimpawu ezicacisayo zefomu le-aortic ye-atherosclerosis. Lesi sifo sikhonjiswa ngokubalwa, ukubakhona kwama-aneurysms, uphawu, ubude nezandiso ze-artery ku-sternum noma i-peritoneum.
- I-Coronography, esiza ukubona isimo semithambo yenhliziyo nokuba khona kwama-cholesterol deposits kuwo.
- I-Angiography - ucwaningo lokugeleza kwegazi leminye imithambo yegazi kusetshenziswa ukwethulwa kwe-medium ephakathi ne-radiography.
- I-UZDG yemithambo ye-renal isiza ukunquma ukuphulwa okusebenzayo kwesitho nokukhula kwe-atherosclerosis kuyo.
- Ukuvuselelwa kabusha kwamaphethelo aphansi kunquma ukuhamba kwegazi ukuphazamiseka emilenzeni, kanye nokuba khona kwama-cholesterol plaques nokukhula.
I-Ultrasound yezitho ngazinye nayo iyasetshenziswa, kuyasiza ukuhlola ijubane lokuhamba kwegazi emithanjeni yemithambo yegazi. Le ndlela inquma ngokunembile ukuphambuka okuncane kakhulu kanye nezinga lokuntuleka kwegazi.
Ukwelashwa nokwelashwa
Ngokwezibalo nokubuyekezwa, kumaphesenti angama-80 wamacala, ukuphuza umuthi kwanele ukuqeda izimpawu ze-atherosclerosis futhi kulawule ukuthuthuka kwayo okuqhubekayo.
Ngokuphikisana nesizinda somuthi wokulashwa kwezidakamizwa, okudingeka ukugcinwa kokudla okhethekile kanye nokuzivocavoca isiguli.
Lapho wenza ukwelashwa, kufanele kulandelwe indlela ehlanganisiwe yokwenziwa kwezokwelapha.
Izidakamizwa ezisebenzayo zokwelapha i-atherossteosis yilezi:
- Statins (Atorvastatin, Rosuvastatin) - izidakamizwa ezinciphisa ukusebenza kwesibindi ekukhiqizeni i-cholesterol. Leli qembu lezidakamizwa linqunywa kaningi.
- Ama-fibrate (Atromide, Tricor) angamayeza isenzo sakhe okuhloswe ngaso ukubhujiswa kwama-triglycerides.
- Ama-sequestrants we-LCD (Colestyramine, Kolesevelam) - izidakamizwa ezivimba ukwakheka kwama-bile acid ngesibindi. Ngenxa yalokhu, isibindi sisebenzisa i-cholesterol eningi ukwenza inqubo yokugaya ibe ejwayelekile.
- I-Nicotinic acid kanye nokutholakala kwayo kuyizidakamizwa ezokwehlisa i-cholesterol, futhi zibe nomphumela we-antispasmodic and vasodilating.
Ngokuphikisana nesizinda sokwelashwa okuyinhloko, kusetshenziswe neminye imishanguzo - ama-antiplatelet agents, amavithamini, ama-angioprotectors, izidakamizwa, umuthi wokuthuthukisa ukondleka nokujikeleza kwegazi, izidakamizwa ze-antispasmodics kanye ne-vasodilator. Ngaphambi kokuthatha imishanguzo, kufanele uthintane nodokotela futhi ufunde incazelo yayo
Ezimweni ezisezingeni eliphakeme, ukwelashwa nokwelashwa kudla ngokungasebenzi. Ukuze uvikele izinkinga ezinkulu, ukuhlinzwa kuyenziwa:
- ukuhlinzwa ngokweqile - “ukuqiniswa” komkhumbi othintwe yi-atherosulinosis kumthambo onempilo nokwakheka komugqa omusha wegazi.
- i-vastery prosthetics - ukubuyiselwa okuphelele komkhumbi nokubuyiselwa kokuhlinzekwa kwegazi.
Uma kunesidingo, i-angioplasty iyasetshenziswa - ukuhlanza nokukhulisa umkhumbi ngokwazisa i-catheter ngokusebenzisa umthambo womuntu wesifazane.
Ukudla ukwelashwa kwe-atherossteosis
Ngokungeziwe ekwelashweni kwezidakamizwa, indlela esidla ngayo i-atherosclerosis idlala indima ebalulekile.
Ngaphandle kokuthi izitho zangaphakathi zikhiqiza ama-80% e-cholesterol, ama-20% asele angena emzimbeni ngokudla.
Umgomo oyisisekelo wokwelashwa kokudla ekwelapheni i-atherosclerosis ukunciphisa inani le-cholesterol evela ngaphandle.
Ngale njongo, kuyadingeka ukushiya imikhiqizo enjalo:
- inyama enamafutha namanoni ezilwane - ingulube, idada, ihansi, isinqe, njll;
- i-offal - isibindi, ubuchopho;
- izinhlobo zezinhlanzi ezinamafutha - i-mackerel, i-carp yesiliva, i-herring, i-halibut, njll;
- ukhukhamba, ukhukhamba, obhemayo futhi othosiwe;
- imikhiqizo yobisi enamafutha;
- amasoseji nososeji;
- izikhupha zamaqanda;
- iziphuzo ezimnandi ze-carbonated, ikofi eqinile netiye;
- imikhiqizo yokubhaka kafulawa we-premium;
- amaswidi - ushokoledi, amaswidi, amakhukhi, njll.
Asikho isidingo sokucasuka ukuthi kufanele ushiye imikhiqizo eminingi oyaziyo. Noma ngabe welashwa i-atherossteosis, ungadla izitsha ezihlwabusayo futhi ezinempilo. Ziyabanjiswa, zibilisiwe noma zithunyelwe kuhhavini. Awukwazi ukwengeza usawoti omningi (isilinganiso sansuku zonke - amagremu ama-5), ungathathelwa indawo nge-pepper ebomvu noma emnyama nezinye izinongo. Ukudla kuvumela ukungena kwemikhiqizo elandelayo:
- inyama ebusayo - inyama yomvundla, inkukhu, njll;
- imikhiqizo yobisi enamafutha amancane;
- izinhlobo zezinhlanzi ezinamafutha amancane - i-hake, i-pike perch, i-bream, i-carp, njll;
- imikhiqizo yokubhaka enamafutha;
- izithelo ezintsha, amajikijolo, amakhambi nemifino;
- itiye eliluhlaza elibuthakathaka, iziphuzo zemvelo.
Ukuhambisana nokudla nokuzivocavoca umzimba kuyindlela enhle yokuvimbela isifo sokuqina kwemithambo yegazi nezinye izifo zenhliziyo, okufanele zinamathelwe kwababili abagulayo nabanempilo.
Yini i-atherossteosis ezotshela uchwepheshe kuvidiyo ekulesi sihloko.
