Ukupakishwa kwezimpahla namuhla kukhumbuza kakhulu inkontileka eyenziwe ngobuqili: kufanele ufunde ngokucophelela ngokucophelela okubhaliwe emhlane kwifonti encane kunazo zonke. Musa ukujaha ukuthenga umkhiqizo lapho ubona izinhlamvu ezinkulu "zinoshukela" kwilebula; kungenzeka impela ukuthi uqukethe nezinye izithako, izinzuzo zakhona ezibuzwa nanamuhla.
Akuyona imfihlo ukuthi ushukela alimazi amazinyo kuphela, kepha nemithambo yegazi, isibindi sihlupheka kakhulu kulo. Kodwa-ke, ekwakhiweni kwezifo ezahlukahlukene, indima ebalulekile ayidlalwanga kuphela ngoshukela oludliwayo, kodwa futhi nezinhlobonhlobo zalo. Kusuka kuluphi uhlobo loshukela esiludlayo, kuya ngokuthi ubungozi obungakanani bezifo ze-metabolic kanye nokuvela kwezinkinga zenhliziyo nemithambo yegazi kukhuphuka.
Le ndatshana izogxila kwi-fructose: amaswidi ngale monosaccharide, omasquerades njengomkhiqizo onempilo, awanconywa namuhla ngabanesifo sikashukela ezigulini zabo. Khumbula ukuthi i-fructose ayinikezi umuzwa wokuqaqamba futhi ibhebhethekisa ukumelana ne-insulin, futhi icacisa imiphumela yezifundo zakamuva.
Iziphetho ezenziwe yiqembu lososayensi eliholwa nguMartha Alegret wase-University of Barcelona libonisa ukuthi ukudla i-fructose kuthinta kabi isimo se-metabolism kanye nohlelo lokujikeleza kwegazi. Kuliqiniso, amagundane wokuhlola abambe iqhaza ekuhlolweni kwawo.
Abaphenyi baseSpain benze ucwaningo kwabesifazane, njengoba besabela ngokushesha kwabesilisa ekushintsheni futhi bakhombisa izinguquko ze-metabolic. Izifundo zokuhlolwa ngomsila zahlukaniswa zaba ngamaqembu amabili: izinyanga ezimbili zanikwa ukudla okuqinile okuqinile, kepha elinye iqembu lanikezwa i-glucose kanti elinye kwakuyi-fructose. Futhi-ke siqhathanise imiphumela, isisindo sokulinganisa, inani lama-triglycerides egazini futhi sihlola isimo semikhumbi.
NgokukaProfessor Alegrett, inani lama-triglycerides eplasma yegazi landa kakhulu kulezo zilwane ezaziphekwe i-fructose. Lo mphumela awunakuchazwa ngokuhlanganiswa okukhethekile kwamafutha e-hepatic, ngoba zombili i-glucose ne-fructose zivusa ukwakheka kwamafutha esibindini.
Kumagundane ekudleni kwe-fructose, izinga le-enzyme enkulu elibhekele ukushiswa kwamafutha, i-CPT1A, lehlile. Lokhu kungakhombisa ukuthi i-fructose inganciphisa inqubo yokushiswa kwamafutha futhi ikhuphule ukuphuma kwe-triglycerides egazini.
Ososayensi babuye baqhathanisa izimpendulo ezahlukahlukene zezinkomba ezibonisa isifo se-vascular. Ukuze senze lokhu, safunda ukusabela kwe-aorta ezintweni ezenza ukuthi imikhumbi ingene futhi ikhule. Ezinyamazana okudla kwazo okubandakanya i-fructose, amandla we-aorta wokuphumula ayemenyezelwe (kuqhathaniswa neqembu lokulawula).
Kumagundane anikezwe i-fructose, bekukhona nezimpawu zokushintsha kwesibindi (ezifundweni zakuqala, ososayensi bebevele bebhalile iqiniso lokuthi izimpawu ze-hepatosis enamafutha ziyimpawu hhayi eyabesifazane kuphela, kepha neyabesilisa). Ngaphezu kwalokho, lezi zifundo zikhombise ukwanda okukhulu kwesisindo.
Abaphenyi baseSpain baphethe ukuthi i-fructose ibambezela inqubo yokushiswa kwamafutha futhi inyusa ukuhlanganiswa kwamafutha esibindini, okungaholela ekwandeni kasayizi wamadepho wamafutha kulesi sitho ne-hepatosis enamafutha. Lesi sifo ekuqaleni asizenzi sizizwe, ngoba sinjenge-asymptomatic, kepha, ekugcineni, singadala inqubo yokuvuvukala esibindini futhi sibangele ukuqala kwezifo ezinzima.
