Umcimbi wokuvuka kokusa kushukela sikashukela

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Isifo sikashukela i-mellitus yiyona endocrinopathy evame kakhulu kubantu besintu emhlabeni. Umkhuba wokusa kokusa ukwanda kwe-glucose ekuseni, imvamisa kusuka ku-4 - 6, kepha kwesinye isikhathi ihlala kuze kube ngu-9 ekuseni. Isimanga sathola igama laso ngenxa yokuhlangana kwesikhathi lapho ushukela wawukhuphuka kusukela ekuntweleni kokusa.

Kungani kukhona into enjalo

Uma sikhuluma ngokulawulwa komzimba kwama-hormone ngokomzimba, khona-ke ukwanda kwe-monosaccharide egazini ekuseni kuyinto evamile. Lokhu kungenxa yokukhishwa kwansuku zonke kwama-glucocorticoids, ukukhishwa okukhulu okwenziwa ekuseni. Lezi zindawo zokugcina zinempahla yokuvuselela ukwakheka kwe-glucose esibindini, esivele singene egazini.

Kumuntu ophile kahle, ukukhishwa kwe-glucose kunxephezelwa yi-insulin, ekhishwa yi-pancreas ngenani elifanele. Ku-mellitus yesifo sikashukela, kuya ngohlobo, i-insulin ayikhiqizwa ngenani elidingeka umzimba, noma ama-receptors asezicubu ayamelana nalo. Umphumela uba yi-hyperglycemia.


Kubaluleke kakhulu ukunquma izinga likashukela kaningana phakathi nosuku ukuze kutholakale into yokusa yasekuseni ngesikhathi.

Kuyini ingozi yento

Izinguquko ezisheshayo kushukela wegazi zigcwele ukukhula okusheshisa kwezinkinga. Bonke abantu abanesifo sikashukela banobungozi bemiphumela emibi. Lokhu kufaka phakathi: i-retinopathy yesifo sikashukela, i-nephropathy, i-neuropathy, i-angiopathy, unyawo lwesifo sikashukela.

Futhi, ukuthuthukiswa kwezimo ezibucayi ngenxa yokushintshashintsha okubukhali kushukela wegazi akunakwa. Izimo ezinjalo zifaka i-coma: hypoglycemic, hyperglycemic, ne-hyperosmolar. Lezi zingqinamba zanda ngejubane lombani - ukusuka emaminithini ambalwa kuya emahoreni ambalwa. Akunakwenzeka ukubikezela ukuqala kwabo ngokumelene nesizinda sezimpawu ezikhona manje.

Ithebula "Izinkinga ezinzima zesifo sikashukela"

IzinkingaIzizathuIqembu lengoziIzimpawu
I-HypoglycemiaAmazinga we-glucose angaphansi kuka-2,5 mmol / L avela ku:
  • ukwethulwa komthamo omkhulu we-insulin;
  • ukudla okunganele ngemuva kokusebenzisa i-insulin;
  • ukusebenza ngokweqile komzimba.
Iziguli ezinesifo sikashukela sanoma yiluphi uhlobo nobudala ziyavezwa.Ukulahlekelwa ukwazi, ukukhuphuka kokujuluka, imvuthuluka, ukuphefumula okungashoni. Ngenkathi ugcina ukwazi - umuzwa wendlala.
I-HyperglycemiaUkunyuka kweshukela egazini okungaphezu kuka-15 mmol / l ngenxa:
  • ukuntuleka kwe-insulin;
  • ukungahambisani nokudla;
  • isifo sikashukela esingaziwa.
Ama-diabetes noma yiluphi uhlobo nobudala, athambekele ekucindezelekeni.Isikhumba esomile, ukuqina, ukuncipha kwezwi lemisipha, ukoma ongacimekiyo, ukuchama kaningi, ukuphefumula okujulile okunomsindo, ukuhogela iphunga le-acetone emlonyeni.
I-Hyperosmolar comaUshukela ophakeme namazinga e-sodium. Imvamisa phakathi kokuphelelwa ngamanzi emzimbeni.Iziguli zeminyaka eyi-senile, zivame kakhulu isifo sikashukela sohlobo 2.Ukoma okungenakuqedwa, ukuchama njalo.
I-ketoacidosisIkhula kungakapheli izinsuku ezimbalwa ngenxa yokuqongelelwa kwemikhiqizo ye-metabolic yamafutha nama-carbohydrate.Thayipha iziguli zikashukela ezi-1Ukulahlekelwa ukwazi, i-acetone ephuma emlonyeni, ukuvimba kwezitho ezibalulekile.

Ungathola kanjani uma unenkinga

Ukuba khona kwalesi sifo kuqinisekiswa ngokwanda kwenkomba ye-glucose kushukela likashukela ekuseni, uma unikezwa ukuthi ebusuku inkomba yayijwayelekile. Kulokhu, izilinganiso kufanele zithathwe phakathi nobusuku. Kusukela phakathi kwamabili, bese kuqhubeka kusukela emahoreni amathathu kuye kwele-7 ekuseni ngehora. Uma ubona ukwanda okubushelelezi kushukela ekuseni, khona-ke empeleni umkhuba wokusa kokusa.

Ukuxilongwa kufanele kwehlukaniswe neSomoji syndrome, okubuye kuboniswe ngokwanda kokukhishwa kwe-glucose ekuseni. Kepha lapha isizathu silele kokungaphezulu kwe-insulin ekhishwe ebusuku. Ukweqisa umuthi kuholela esimweni se-hypoglycemia, lapho umzimba ufaka khona imisebenzi yokuvikela futhi ufihle ama-hormone aphambanayo. Lokhu kokugcina kusiza ushukela ukuba ungene egazini - futhi umphumela we-hyperglycemia.

Ngakho-ke, i-asubuhi yesifo sofuba iyaziveza kungakhathalekile ukuthi ithamo le-insulin liphathwe nini ebusuku, kanti uSomoji ulungile ngenxa yokwedlula umuthi.


Uma isiguli sinenkinga yokusa kokusa, zonke izinkinga zesifo sikashukela ziqhubeka ngokushesha okukhulu.

Ungabhekana kanjani nenkinga

Ushukela wegazi ophakeme kumele njalo ulwiwe. Futhi nge-dawn syndrome, ama-endocrinologists ancoma okulandelayo:

  1. Dlulisa umjovo wobusuku we-insulin emahoreni angama-1-3 kamuva kunokwejwayelekile. Umphumela wemithamo emide yesidakamizwa izowa ekuseni.
  2. Uma ungabekezeleli isikhathi sokulawulwa ebusuku somuthi, ungenza umthamo we-insulin wesikhathi esifushane emahoreni "ngaphambi kokusa", ngo-4: 4-4.30 ekuseni. Lapho-ke uzophunyuka ekukhuphukeni. Kepha kulokhu, kudinga ukukhethwa okukhethekile komthamo womuthi, ngoba ngisho nangokweqisa okuncane, ungadala i-hypoglycemia, okungeyona ingozi kangako emzimbeni wesifo sikashukela.
  3. Indlela enengqondo kunazo zonke, kepha ebiza kakhulu ukufaka iphampu ye-insulin. Iqapha izinga likashukela nsuku zonke, futhi wena, ngokwazi okudlayo nomsebenzi owenziwa nsuku zonke, nquma izinga le-insulin nesikhathi eliza ngaphansi kwesikhumba.

Hlakulela umkhuba wokubheka njalo ushukela wegazi lakho. Vakashela udokotela wakho bese ubheka futhi ulungise ukwelashwa kwakho njengoba kudingeka. Le yindlela ongayigwema ngayo imiphumela emibi.

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