Isifo sikashukela i-mellitus yiyona endocrinopathy evame kakhulu kubantu besintu emhlabeni. Umkhuba wokusa kokusa ukwanda kwe-glucose ekuseni, imvamisa kusuka ku-4 - 6, kepha kwesinye isikhathi ihlala kuze kube ngu-9 ekuseni. Isimanga sathola igama laso ngenxa yokuhlangana kwesikhathi lapho ushukela wawukhuphuka kusukela ekuntweleni kokusa.
Kungani kukhona into enjalo
Uma sikhuluma ngokulawulwa komzimba kwama-hormone ngokomzimba, khona-ke ukwanda kwe-monosaccharide egazini ekuseni kuyinto evamile. Lokhu kungenxa yokukhishwa kwansuku zonke kwama-glucocorticoids, ukukhishwa okukhulu okwenziwa ekuseni. Lezi zindawo zokugcina zinempahla yokuvuselela ukwakheka kwe-glucose esibindini, esivele singene egazini.
Kumuntu ophile kahle, ukukhishwa kwe-glucose kunxephezelwa yi-insulin, ekhishwa yi-pancreas ngenani elifanele. Ku-mellitus yesifo sikashukela, kuya ngohlobo, i-insulin ayikhiqizwa ngenani elidingeka umzimba, noma ama-receptors asezicubu ayamelana nalo. Umphumela uba yi-hyperglycemia.
![](http://img.diabetesentity.com/img/diab-2020/7898/fenomen-utrennej-zari-u-diabetikov.jpg)
Kubaluleke kakhulu ukunquma izinga likashukela kaningana phakathi nosuku ukuze kutholakale into yokusa yasekuseni ngesikhathi.
Kuyini ingozi yento
Futhi, ukuthuthukiswa kwezimo ezibucayi ngenxa yokushintshashintsha okubukhali kushukela wegazi akunakwa. Izimo ezinjalo zifaka i-coma: hypoglycemic, hyperglycemic, ne-hyperosmolar. Lezi zingqinamba zanda ngejubane lombani - ukusuka emaminithini ambalwa kuya emahoreni ambalwa. Akunakwenzeka ukubikezela ukuqala kwabo ngokumelene nesizinda sezimpawu ezikhona manje.
Ithebula "Izinkinga ezinzima zesifo sikashukela"
Izinkinga | Izizathu | Iqembu lengozi | Izimpawu |
I-Hypoglycemia | Amazinga we-glucose angaphansi kuka-2,5 mmol / L avela ku:
| Iziguli ezinesifo sikashukela sanoma yiluphi uhlobo nobudala ziyavezwa. | Ukulahlekelwa ukwazi, ukukhuphuka kokujuluka, imvuthuluka, ukuphefumula okungashoni. Ngenkathi ugcina ukwazi - umuzwa wendlala. |
I-Hyperglycemia | Ukunyuka kweshukela egazini okungaphezu kuka-15 mmol / l ngenxa:
| Ama-diabetes noma yiluphi uhlobo nobudala, athambekele ekucindezelekeni. | Isikhumba esomile, ukuqina, ukuncipha kwezwi lemisipha, ukoma ongacimekiyo, ukuchama kaningi, ukuphefumula okujulile okunomsindo, ukuhogela iphunga le-acetone emlonyeni. |
I-Hyperosmolar coma | Ushukela ophakeme namazinga e-sodium. Imvamisa phakathi kokuphelelwa ngamanzi emzimbeni. | Iziguli zeminyaka eyi-senile, zivame kakhulu isifo sikashukela sohlobo 2. | Ukoma okungenakuqedwa, ukuchama njalo. |
I-ketoacidosis | Ikhula kungakapheli izinsuku ezimbalwa ngenxa yokuqongelelwa kwemikhiqizo ye-metabolic yamafutha nama-carbohydrate. | Thayipha iziguli zikashukela ezi-1 | Ukulahlekelwa ukwazi, i-acetone ephuma emlonyeni, ukuvimba kwezitho ezibalulekile. |
Ungathola kanjani uma unenkinga
Ukuba khona kwalesi sifo kuqinisekiswa ngokwanda kwenkomba ye-glucose kushukela likashukela ekuseni, uma unikezwa ukuthi ebusuku inkomba yayijwayelekile. Kulokhu, izilinganiso kufanele zithathwe phakathi nobusuku. Kusukela phakathi kwamabili, bese kuqhubeka kusukela emahoreni amathathu kuye kwele-7 ekuseni ngehora. Uma ubona ukwanda okubushelelezi kushukela ekuseni, khona-ke empeleni umkhuba wokusa kokusa.
Ukuxilongwa kufanele kwehlukaniswe neSomoji syndrome, okubuye kuboniswe ngokwanda kokukhishwa kwe-glucose ekuseni. Kepha lapha isizathu silele kokungaphezulu kwe-insulin ekhishwe ebusuku. Ukweqisa umuthi kuholela esimweni se-hypoglycemia, lapho umzimba ufaka khona imisebenzi yokuvikela futhi ufihle ama-hormone aphambanayo. Lokhu kokugcina kusiza ushukela ukuba ungene egazini - futhi umphumela we-hyperglycemia.
Ngakho-ke, i-asubuhi yesifo sofuba iyaziveza kungakhathalekile ukuthi ithamo le-insulin liphathwe nini ebusuku, kanti uSomoji ulungile ngenxa yokwedlula umuthi.
![](http://img.diabetesentity.com/img/diab-2020/7898/fenomen-utrennej-zari-u-diabetikov-2.jpg)
Uma isiguli sinenkinga yokusa kokusa, zonke izinkinga zesifo sikashukela ziqhubeka ngokushesha okukhulu.
Ungabhekana kanjani nenkinga
Ushukela wegazi ophakeme kumele njalo ulwiwe. Futhi nge-dawn syndrome, ama-endocrinologists ancoma okulandelayo:
- Dlulisa umjovo wobusuku we-insulin emahoreni angama-1-3 kamuva kunokwejwayelekile. Umphumela wemithamo emide yesidakamizwa izowa ekuseni.
- Uma ungabekezeleli isikhathi sokulawulwa ebusuku somuthi, ungenza umthamo we-insulin wesikhathi esifushane emahoreni "ngaphambi kokusa", ngo-4: 4-4.30 ekuseni. Lapho-ke uzophunyuka ekukhuphukeni. Kepha kulokhu, kudinga ukukhethwa okukhethekile komthamo womuthi, ngoba ngisho nangokweqisa okuncane, ungadala i-hypoglycemia, okungeyona ingozi kangako emzimbeni wesifo sikashukela.
- Indlela enengqondo kunazo zonke, kepha ebiza kakhulu ukufaka iphampu ye-insulin. Iqapha izinga likashukela nsuku zonke, futhi wena, ngokwazi okudlayo nomsebenzi owenziwa nsuku zonke, nquma izinga le-insulin nesikhathi eliza ngaphansi kwesikhumba.
Hlakulela umkhuba wokubheka njalo ushukela wegazi lakho. Vakashela udokotela wakho bese ubheka futhi ulungise ukwelashwa kwakho njengoba kudingeka. Le yindlela ongayigwema ngayo imiphumela emibi.