I-Atherossteosis yisifo sezwe lanamuhla. Kuvela ngenxa yezinto eziningi. Amacala amaningi abangelwa ama-pathologies atholakele ngesimo sezinga elikhuphukile le-lipoprotein ephansi.
Ngokubanga kwezizathu, azikwazi ukuthinta imithambo yenhliziyo kanye nenhliziyo kuphela, kepha nazo zonke ezinye izitho zomzimba. Ngenxa yezinqubo ezithile, izindonga zemikhumbi zigcwaliswa ngamachashaza e-atherosulinotic, okwenza ukungondleki kwezitho kungenzeki noma kube nzima. Ngaphezu kwalokho, lokhu kugqokwa hhayi nge-atherossteosis kuphela, kodwa futhi nesifo senhliziyo esiku-coronary, stroke, isifo senhliziyo. Konke kuncike endaweni yomonakalo wemithambo yegazi.
Enye yezinhlobonhlobo ze-atherosulinosis isebenzisa kabi i-atherosulinosis. Lolu uhlobo lwesifo oluyingozi, oluhambisana nokulimala kwezicubu zomzimba emzimbeni wonke wenhliziyo.
Ngenxa yalokhu, ukwephulwa kokwethembeka kwama-valve, lapho-ke umsebenzi wenhliziyo uphazamiseka. Lesi sifo emabangeni okuqala asizivezi, ngakho-ke, ukuxilongwa emazingeni okuqala akuvamile kakhulu.
Leli qiniso lenza ukwelashwa kube nzima kakhulu, ngoba amacala anganakwa ahambisana nezinkinga eziningi ezithinta cishe zonke izinhlelo zomzimba. Ukuze uqonde ukuthi ungayelapha kanjani i-atherosclerosis nokuthi iyini, kufanele uqonde izindlela zesenzo sayo.
Lesi simo sidinga ukuxilongwa kusenesikhathi nokwelashwa okwanele. Izinga lokucindezelwa, ikhwalithi yempilo ngokuzayo, kanye nocwaningo lwesifo kungenzeka.
Ngokutholakala kwanoma yisiphi isifo, imbangela iyadingeka futhi i-atherosclerosis nayo iyodwa.
Kunezizathu eziningi eziba nomthelela ekubukekeni nasekuqhubekeni kwesifo.
Kumuntu, lesi sifo siqala singanakwa ngokuphelele, futhi ngasikhathi sinye, izinqubo eziningi ezingenakulungiswa ziqala emzimbeni.
Lesi sifo senzeka ngenxa:
- Umfutho wegazi ophakeme
- I-Rheumatism
- Umonakalo we-Ischemic kuma-cardiomyocyte.
- Ukuvuvukala kwe-myocardial.
- I-Cardiac arrhythmias.
- I-Dystrophic noma i-hypertrophic phenomena ku-myocardium.
- Isifo sikashukela mellitus.
- Isisindo esiningi.
- Ukuhlinzwa enhliziyweni, ubuchopho.
- Ukulimala emsipha wenhliziyo.
- Ukusebenzisa kabi utshwala.
- Ukubhema.
- Ukuzelapha.
- Ukucindezela okuvama ukuholela ekwehlukaneni ngokomqondo nangokomzwelo njengomphumela.
- Ukuguga.
- Izinqubo ezilandelanayo ezandisa inani lezinsimbi ezisindayo emzimbeni.
- Ukungondleki.
- Isakhi sofuzo.
- Ukuntuleka kokuzivocavoca umzimba.
I-atherossteosis enjalo, ezimeni eziningi, ivela ngokumelene nesizinda se-coronary atherosclerosis esikhona. Unguhlobo oluyingozi kakhulu lwalesi sifo.
Ngalesi sifo, i-necrotic foci yasakazeka ngemithambo yegazi, ekhulisa isimo sesiguli nsuku zonke.
Ngokuvamile kwenzeka ukuthi nge-atherossteosis enjalo i-aneurysm iyakhiwa, ithuthukiswa ngaphansi kwethonya lezinto zangaphakathi nezangaphandle. Uma iqhuma, isiguli siyafa.
Ukuvela kwe-atherosclerosis encane kuhlobene nokuvuvukala kwe-myocardial, okucasulayo ukuvela kwesifo senhliziyo.
Ngaphezu kwalokho, lesi sifo sinezigaba ezintathu zokuthuthuka:
- ukuphazamiseka kokujikeleza emzimbeni;
- ukuvela kwe-ischemia;
- ukufa kwamangqamuzana emisipha, ukubuyiselwa kwawo ngezicubu zesilonda.
Izigaba ezimbili zokuqala zicishe zingabonakali kubantu, impilo icishe ingashintshi. Esigabeni sokugcina, ungazizwa ushintsho olukhulu ngesimo. Iningi labantu alinciphisi ukubaluleka okuningi kulokhu, kepha ekubonakalisweni okuncane udinga ukuya kuchwepheshe.
Kufanele kukhunjulwe ukuthi ukutholwa kwesifo kusenesikhathi kusiza ukwenza ngcono isimo futhi kuvikele nemiphumela engenakuphikwa.
I-atherossteosis enjalo iboniswa ukubonakaliswa kancane kancane kwezimpawu.
Izigaba zokuqala azivezi nganoma iyiphi indlela, noma kunjalo, kwezinye iziguli, i-arrhythmia kanye ne-infuction ephazamisekile kwaphawulwa.
Uma i-myocardium ithinteka kakhulu, isiguli sizwa:
- Ukuphefumula njalo. Isibonakaliso esinjalo singabukwa ngomonakalo we-ventricle yenhliziyo yangakwesobunxele. Okokuqala, le nto ingabonakala ngokuzikhandla ngokomzimba okunamandla, bese kuba njalo cishe njalo, ngokunyakaza okuncane. Lokhu kuvame ukubonwa ikakhulukazi ngokuqhekeka kwe-myocardium.
- Hudula inhliziyo ebuhlungu.
- Ukukhwehlela. Ukukhwehlela okunjalo kubizwa ngokuthi yi-cardiac. Iyabonakala ngesikhathi sokuzikhandla ngokomzimba, futhi ngesilonda esijulile cishe njalo. Imbangela yalokhu kuvezwa kungukulimala kwezicubu zamaphaphu. Isifuba somoya somoya singenza nalokhu kukhwehlela. Kulesi simo, ukuphuma komzimba kuvela - i-frothy sputum futhi iyathela.
- Ukwanda kobuthakathaka, ukusebenza okwehlisiwe.
- Ubuhlungu kwi-hypochondrium efanele. Kungenxa yalolu hlobo lwe-atherosulinosis lolu phawu lubonakala kakhulu kulo. Kuvela maqondana nokuqina kwesiyingi sokuqala sokujikeleza kwegazi. Ubuhlungu bungahlanganiswa nokuvuvukala kwemikhawulo engezansi, ama-ascites.
- Ukulahlekelwa ukwazi okwenzeka ngenxa yokuqalwa kwesifo se-arrhasmic.
- Ukuvuvukala kwemilenze. Ikakhulu izibonakalisa kusihlwa, lapho ekuseni yonke into iwela endaweni kanye nokuvuvukala kunyamalale ngokuphelele. Esigabeni sokuqala, ukuvuvukala kwamaqakala kungabonakala, kepha lapho-ke kungabonakala ezinkalweni.
- Izimpawu ze-inhliziyo inhliziyo. Kuba ngumphumela wokungasebenzi kahle kwenhliziyo.
- Isikhumba esiluhlaza okwesibhakabhaka. Ukuthuthukiswa kwe-cyanosis kucasulwa ngomonakalo ojulile we-myocardial, kuvela ikakhulukazi kunxantathu we-nasolabial.
- Ukuguqulwa kwezipikili, ukwanda kokulahlekelwa izinwele, ngenxa yokuphazamiseka kokujikeleza kwegazi.
- Hyperpigmentation kwesikhumba.
- Ubuhlungu entanyeni.
Izinga lokubonakaliswa kwe-atherosclerosis lincike ebangeni lokulimala kwesifo se-artery cortery kanye nokunikezwa kwegazi.
Ezimweni ezisezingeni eliphakeme kakhulu, izimpawu zingakhula ngasikhathi sinye.
Uma umuntu ebuka ukuvela okungenani kwezimpawu ezi-3, kufanele uthintane nodokotela onguchwepheshe ngokushesha.
Uma lesi sifo siqala ukwenzeka, izimpawu ze-atherosulinosis zingavela ngokuhambisana nezimpawu zokuhlaselwa yinhliziyo, ischemia, kanye nokwehluleka.
Ukuxilongwa ngesikhathi kutholakala kungasindisi impilo kuphela, kepha futhi nokuphila kwesiguli.
Yilolu hlobo lwesifo oludinga ukuphenywa ngeqoqo lezindlela.
Ukuze uthole ukuxilongwa okunembile, kufanele uhlole isimo sesiguli kusuka kuzo zonke izinhlangothi.
Ubunzima bezindlela zokuxilonga kufaka phakathi:
- Ukuhlolwa kwesiguli, ukuqoqwa kwezikhalazo nama-anamnesis, udokotela ubuza ukuthi yiziphi izimpawu ezibonwa esigulini, ukuthi wayegula kanjani ngaphambili, ukuthi iyiphi indlela yokuphila eqondiswa ngumuntu;
- ukuhlolwa kwegazi kokuhlaziywa kwe-biochemical, ukuhlaziywa kuzokhombisa izinga le-cholesterol, ukuhlola isimo sempilo sesiguli futhi kukhombise ubukhona noma ukungabikho kwezifo ezingamahlalakhona;
- I-chocardiogram izokhombisa ubukhona noma ukungabikho kwe-arrhythmia, izinguquko kumakhono wokusebenza we-myocardium, futhi kukhombise izinga lokushaya kwenhliziyo
- I-BCC ultrasound ihlola izinga lokusebenza kwenhliziyo, ukuba khona kwezilonda ezihlasela izicubu zenhliziyo;
- I-MRI izonquma indawo lapho kuthuthukiswa khona i-pathology.
Ngemuva kokuxilongwa, ukwelashwa okufika ngesikhathi kwe-pathology kubalulekile. Lokhu kuzonciphisa ukukhula kwesifo futhi kwehlise namathuba okuba nezinkinga.
Amanye amaphilisi we-cholesterol nemithamo yawo kufanele anikezwe udokotela kuphela. Uma usebenzisa ukwelashwa ngokwakho akuvamile ukuthi kwenzeke into enhle, isikhathi esiningi lesi sifo siqhubeka ngokushesha.
Ukuze kuqedwe ukulimala kwe-myocardial, izidakamizwa kumele zisetshenziswe. Ukwelapha kubandakanya ukwamukelwa:
- Nitrate, okungaholela ku-systemic venodilation. Izidakamizwa zinciphisa ukucabanga ku-myocardium futhi zisuse isidingo saso se-oxygen. Uma zithathwa njalo, ukuhamba kwegazi kuzothuthuka ngokuhamba kwesikhathi.
- Anaprilina. Kuthuthukisa isimo sempilo ngaphansi kwethonya lomsebenzi womzimba. Kepha, banemiphumela emibi eminingi ngesimo sokuncipha kwamandla okushaya kwenhliziyo okukhuphukile, ukukhathala okwandisiwe, ukukhishwa okuqhubekayo.
- Abamelene neCalcium. Ngaphansi kwesenzo sabo, umfutho wegazi kanye nezifo zenhliziyo ziyancipha, isidingo samaseli ezinhliziyo e-oxygen siyancipha. Kepha, bayakwazi ukuphazamisa ukuqhuba kahle.
Ngokuhambisana nalokhu, udinga ukuthatha izidakamizwa ezokwehlisa i-cholesterol. Lokhu kubaluleke kakhulu uma isiguli sibhekene nesimo sokwehla kokulimala. Zonke izidakamizwa zibekwa ngesisekelo sedatha yomuntu ngamunye, ngokwesikimu esichazwe udokotela.
Ukuzilawula kanye nokuphatha izidakamizwa kungaholela ezinkingeni ezahlukahlukene.
Okuqaphelekile kakhulu ukuthi kudliwa yisiguli esine-disuse atherossteosis.
Kusetshenziswa ngokuhlanganiswa nezinye izindlela zokwelapha.
Ngaphandle kokudla, umphumela wokwelashwa uzoba mncane, ngoba zonke izitho zincike ekunakekelweni nsuku zonke.
Yikho okungavusa izifo uma kungenayo into ewusizo.
Ungadla kanjani nge-cholesterol ephezulu? Ukudla izakhamzimba kufaka:
- Ukuqedwa kokudla okunamafutha, okubhemayo, okuthosiwe ekudleni, kwekhofi netiyeni nakho kufanele kushiywe;
- nciphisa ukusetshenziswa kwamafutha ezilwane;
- kunciphisa ukusetshenziswa kwamamanzi okweqile;
- ukuncishiswa kwenani kasawoti osetshenzisiwe;
- ukufaka inyama esikhundleni semikhiqizo yezinhlanzi;
- ukufakwa ekudleni kwenani elikhulu lemifino nezithelo.
Futhi, lokhu kudla kunikela ngokudla okuvela emgomweni, umgomo oyinhloko wawo ukudla okuncane, kepha kaningi. Le ndlela inciphisa umthwalo ohlelweni lokugaya futhi ithuthukise inqubo yokugaya ukudla.
Ukudla okunempilo kanye nokwelashwa ngezidakamizwa kuzosiza ukunciphisa inkambo yesifo, kwehlise ijubane kancane futhi kususe amathuba ezinkinga ngendlela yokuhlaselwa yisifo senhliziyo, unhlangothi. Ukuze ukugweme, udinga ukuthi uhlolwe njalo, udle ngendlela efanele futhi ungasebenzisi kabi imikhuba emibi. Kuzosiza futhi ekwelashweni nasekuvimbeleni isifo semidlalo.
Imininingwane nge-atherosclerosis ichazwe kuvidiyo kule ngxenye.
