Yini ingozi yomfutho wegazi ophansi ku-atherossteosis?

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Iningi labantu liqinisekile ukuthi umfutho wegazi ophakeme ungenye yezimpawu zokuqamba kwe-atherosulinosis, kodwa empeleni lokhu akunjalo. Njengoba izazi zesayensi yezenhliziyo zanamuhla ziphawula, umfutho wegazi ophakeme uyimbangela eyinhloko ye-atherosclerosis, hhayi umphumela wayo.

Iqiniso ngukuthi nomfutho wegazi ophakeme, kuvela imicrodamage ezindongeni zemithambo yegazi, bese igcwaliswa nge-cholesterol, enomthelela ekwakhekeni kwama-cholesterol plaques. Kepha ezigulini ezingenayo i-hypertension, i-atherossteosis ingavusa ukuhla komfutho wegazi futhi ibangele nomfutho wegazi ophakeme.

Kepha umfutho wegazi ophansi uhlobene kanjani ne-atherosulinosis, kungani ukuqhekeka kwemithambo yegazi kubangela ukuhanjiswa kwegazi, iyini ingozi yomfutho wegazi ophansi ku-atherosclerosis nokuthi ungayiphatha kanjani kahle? Le mibuzo iyathandeka ezigulini eziningi ezine-atherosulinosis enomfutho wegazi ophansi.

Kungani ukucindezela kuncipha nge-atherosclerosis

Wonke umuntu uyazi ukuthi umfutho wegazi ojwayelekile ungama-120/80 mm. Hg. Ubuciko. Noma kunjalo, hhayi noma yikuphi ukuphambuka kusuka kulesi sikhombisi kungabhekwa njenge-pathology. Ukukhuluma ngesimo esibuhlungu sesiguli nokuba khona kwe-hypotension kungenzeka kuphela lapho ingcindezi yehla ngaphansi kuka-100/60 mm. Hg. Ubuciko.

Ngaphezu kwalokho, ezigulini ezitholakala nge-atherossteosis, ukwehla okuphawulekile kwe-diastolic noma, ngendlela elula, ingcindezi ephansi iyaphawulwa. Lokhu kuyiqiniso ikakhulukazi ezigulini ezineminyaka engaphezu kwengama-40, lapho, ngaphandle kwe-atherosclerosis, izinguquko ezihlobene nobudala ohlelweni lwezinhliziyo nazo zibonakaliswa.

Lesi sici sichazwa iqiniso lokuthi nge-atherosclerosis emikhunjini emikhulu yomzimba, ikakhulukazi kwifomu le-aorta, cholesterol plaques, okuphazamisa ukuhamba kwegazi okujwayelekile. Ngaphezu kwalokho, imikhumbi ngokwayo ilahlekelwa yiwo ngobudala, iba ntekenteke futhi ibe mncane.

Ngenxa yalokhu, inani eliphelele lokujikeleza kwegazi emzimbeni womuntu liyancishiswa, okuyinto elimaza kakhulu ukunikezwa kwegazi emalungeni. Kepha umfutho wegazi ulinganiswa ngokuqondile emthanjeni we-brachial, ondla izicubu kanye nezinye izicubu zezandla ezinegazi.

Ngokwezinga elibi kakhulu, i-hypotension yenzeka ezigulini, ngaphezu kwe-atherosclerosis, nazo ezihlaselwa isifo sikashukela sohlobo 1 noma uhlobo 2. Kulokhu, i-angiopathy yesifo sikashukela ibuye ifakwe ekushintsheni kwe-atherosulinotic emikhunjini - i-pathical lesion of the wall vascular ngenxa kashukela wegazi ophakeme.

I-Angiopathy iyakwazi ukubhubhisa ngokuphelele kuqala imithambo emincane bese ilandelana, ngaleyo ndlela ikuphazamise ngokuphelele ukuhamba kwegazi emilenzeni. Lesi simo sivame ukuphela nge-necrosis yezicubu, ukuthuthukiswa kwe-necrosis enzima, kanye nokulahleka kwemilenze.

Akuyona ingozi kangako esigulini ukukhula okufanayo kwe-atherosulinosis kanye nokwehluleka kwenhliziyo, okungahle kube ngumphumela wokuhlaselwa yisifo senhliziyo, izinhliziyo zenhliziyo nokugula okungapheli kwe-pulmonary isifo.

Kulokhu, isiguli sizophinda sizwe ukwehla okuphawuleka kwengcindezi ye-diastolic.

Ingozi yengcindezi ephansi

Namuhla, kukhulunywa kakhulu ngomonakalo omkhulu empilweni ukuthi umfutho wegazi ophakeme ungadala ngaphandle kokunaka umfutho wegazi ophansi. Kodwa akuyona i-pathology eyingozi engabhebhethekisa ukukhula kwezinkinga ezinkulu.

Imiphumela emibi ikakhulukazi ingcindezi yegazi ephansi yohlelo lwezinzwa oluphakathi, ikakhulukazi ubuchopho. Iqiniso ngukuthi ngokuhlinzekelwa kwegazi okunganele, amangqamuzana obuchopho athola ukuntuleka komoya-mpilo kanye nezakhi zomzimba, okuphazamisa ukuxhumana kwe-neural futhi kuholele ekufeni kancane kwezicubu zobuchopho.

Njengoba i-pathology ikhombisa, ukulondolozwa isikhathi eside komfutho wegazi ophansi esigulini kuholela ekuguqukeni okungaphenduki ebuchosheni futhi kungadala ukwephulwa okuphelele kwayo yonke imisebenzi yesistimu yezinzwa ephakathi.

Ukwehla kokugeleza kwegazi okujwayelekile kuthinta umsebenzi hhayi wobuchopho kuphela, kepha nezinye izitho ezingaphakathi nezinhlelo zomuntu. Ngakho-ke ekucindezelekeni okuphansi kukhona ukuphazamiseka kwemisebenzi yendlela yesisu, isisu se-musculoskeletal, isimo esingokomzwelo, nezinhlelo zenhliziyo nezokuzala.

Ubungozi bokucindezela okuphansi kobuchopho:

  1. Ukucindezela nokuqhuma izinhlungu kugxiliwe ezingxenyeni ze-occipital nezangaphambili lekhanda. Qinisa ukukhathala, ukudla okusindayo kanye nokushintsha kwesimo sezulu;
  2. Isisi esihlala isikhathi eside. Ngokunyuka okubukhali, kube mnyama kumankules nesiyezi esinzima kuze kube yilapho sekulahlekelwe ukwazi;
  3. Ukugula kwe-Motion kwezokuhamba;
  4. Ukulimazeka kwememori, ukulahleka kokugxilisa ingqondo nokuphazamiseka;
  5. Ukwehla kwenqubo yokucabanga, wehlise izinga lobuhlakani;
  6. Ezimweni ezinzima kakhulu, ukuwohloka komqondo.

Imiphumela ye-hypotension emgodini wesisu nayo imbi. Iziguli ezine-hypotension zinobunzima obungapheli esiswini; ukushisa kwenhliziyo nokushaya; isicanucanu nokuhlanza; ukuntuleka kokudla, ukunambitheka okumunyu emlonyeni; ukuqhakaza kanye nokuqunjelwa njalo.

Ukulimala kwengcindezi encishisiwe yesistimu yenhliziyo:

  • Ubuhlungu esifundeni senhliziyo;
  • Ukufiphala komoya ngisho nangemuva kokusebenza kanzima, futhi kwesinye isikhathi kusesimweni esizolile;
  • Ukuqina kwamalunga, ngenxa yokuthi izingalo nemilenze kungabanda kakhulu;
  • I-palpitations yenhliziyo, ukuphazamiseka kwesigqi senhliziyo.

Ubungozi bokulahlekelwa kwengcindezi engapheli kohlelo lwe-musculoskeletal: Ubuhlungu obuhlangene; ubuhlungu bemisipha edlula ngesikhathi sokuzivocavoca (ukusebenza ngokomzimba kuthuthukisa ukuhamba kwegazi ezicutshini zemisipha); i-edema ikakhulukazi esifundeni semilenze.

Umphumela wokucindezela okuphansi esimweni sokuzwela sesiguli:

  1. Ukwanda kokukhathazeka, ukukhathazeka okungapheli;
  2. Ukuphazamiseka kokulala, inkathazo ukulala;
  3. I-apathy, ukwehla okuphawulekile kokusebenza;
  4. Ukuntula intshisekelo empilweni, ukungathandi ukwenza noma yini;
  5. Ukukhathala okungapheli, ukungaqapheli ngisho nangemva kokulala ngokugcwele;
  6. Ukweqisa ngokweqile ngemuva kokuvuka, kuyadingeka okungenani amahora ama-2 ukuze isiguli ekugcineni sivuke bese senza ibhizinisi labo. Inani eliphakeme lomsebenzi, njengomthetho, lenzeka emahoreni kusihlwa;
  7. Ukudangala kanye ne-neurosis;
  8. Ukungahambisani nemisindo ekhanyayo nokukhanya okukhanyayo.

Ukulimala kwe-hypotension ohlelweni lokuzala kusobala. Emadodeni, i-potency iya iba mbi futhi ekugcineni aqede ukungasebenzi ngocansi; nakwabesifazane - ukungahambisani nokuya esikhathini.

Ukwelashwa

Njengoba kungabonakala kulokhu okungenhla, umfutho wegazi ophansi awunakuba yingozi empilweni yabantu kune-hypertension. Ngaso leso sikhathi, uma umfutho wegazi ophakeme ungehliswa kusetshenziswa uhlu lonke lwemithi ehlukahlukene, khona-ke ayikho cishe imithi yokwengeza.

Umuthi wodwa we-hypotension amaphilisi we-caffeine, aziwa ukuthi ayingozi kakhulu ohlelweni lwezinhliziyo futhi anganconywa kubantu abane-vasher atherosclerosis. Ngesizathu esifanayo, nalesi sifo, akufanele uphuze inani elikhulu lekhofi, naphezu kwe-hypotension.

Kubalulekile ukuqonda ukuthi umfutho wegazi ophansi ku-atherosulinosis akusona isifo esihlukile, kepha umphumela wokuqhekeka kwemithambo yegazi nesifo senhliziyo (coronary senhliziyo). Ngakho-ke, ukuze ubhekane ne-hypotension, kubalulekile ukwenza yonke imizamo yokwelapha i-atherosclerosis ne-cholesterol ephansi yegazi.

Ungawandisa kanjani umfutho wegazi ngesikhathi sokuqina kwemithambo yegazi? Usizo:

  • Umsebenzi womzimba. Ukuhamba emoyeni omusha, ukugijima okukhanyayo, izivivinyo zasekuseni, ukubhukuda nokuhamba ngebhayisikile kuzoba wusizo ngokulinganayo ku-atherosclerosis kanye nomfutho wegazi ophansi. Ukuzivocavoca kuzosiza ekwehliseni ngempumelelo i-cholesterol yegazi, kuyilapho i-normalizing blood pressure, kwandisa ithoni ye-vascular, ngcono ukuhamba kwegazi nokuqinisa izicubu zenhliziyo. Kodwa-ke, kubalulekile ukuhlanganisa kahle imithwalo yezemidlalo nokuphumula okuhle ukuvimbela ukusebenza ngokweqile;
  • Massage Zonke izinhlobo zokugcoba, kufaka phakathi i-acupressure ne-reflexology, zilusizo olukhulu kwiziguli ezinomfutho wegazi ophansi ku-atherossteosis. Kuyasiza ukwandisa kakhulu ukugeleza kwegazi, yenze umsebenzi wezinhlelo zenhliziyo nezinzwa kube ngcono, uthuthukise imetabolism futhi uqinise izicubu zomzimba;
  • Hlukanisa ishawa. Ukusetshenziswa kweseshini esehlukile futhi kunezibuyekezo eziningi ezinhle ekwelapheni i-hypotension. Umphumela wokushintshashintsha kwamanzi apholile futhi afudumele emzimbeni kudala ukuncipha okubukhali nokwanda kwemithambo yegazi, okukuvumela ukuthi uqinise izindonga zemithambo, ukhuphule ukuqina futhi uthuthukise ukuhamba kwegazi emilenzeni. Noma kunjalo, kubalulekile ukuqaphela ukuthi umehluko wokushisa akufanele ube namandla kakhulu;
  • Ukulala okugcwele. Abantu abanomfutho wegazi ophansi badinga isikhathi esengeziwe sokulala ngokwanele futhi babuye babe namandla, ngakho-ke, ukulala kweziguli ezine-hypotension kufanele okungenani kube ngamahora ayi-9. Ngasikhathi sinye, kubalulekile ukuthi iziguli ezinomfutho wegazi ophansi zilale zingakashayi phakathi kwamabili, futhi okuhle kunakho konke ngo-23: 00;
  • Ukudla okufanele. Nge-atherossteosis eyinkimbinkimbi nge-hypotension, kubaluleke kakhulu ukulandela ukudla okuselapha okunokuqukethwe kwe-cholesterol ephansi. Isisekelo sokudla okunjalo okwelapha kufanele kube ukudla okunothe amavithamini, amaminerali, ama-antioxidants, i-fiber nezinye izinto ezibalulekile empilweni;
  • Amakhambi asetshenziswa ngamakhambi. Ukwenza ngcono ukusebenza kohlelo lwezinhliziyo kanye nokwandisa ithoni ye-vascular, tinctures yotshwala yamakhambi okwelapha anjenge-ginseng, eleutherococcus, i-pink radiola, i-echinacea ne-safflower levse kuzosiza. Kumele kuqashelwe ukuthi lokhu kuhlanganiswa kwemithi yemithi kufanele kuthathwe kuphela engxenyeni yokuqala yosuku, ukuze ungavumeli ukuqwasha.

Ukucindezela Okuvamile kwe-Atherossteosis

Iziguli eziningi zinentshisekelo kulo mbuzo, kungaba khona i-atherosulinosis enengcindezi evamile? Cha, lokhu akunakwenzeka, abafundi bezokwelapha abatshelwa ngakho ezinkulumweni zokuqala.

Ukuvalwa kwamangqamuzana ne-cholesterol plaques kuthinta kabi ukusebenza kohlelo lwezinhliziyo, oluthinta ngokushesha umfutho wegazi.

Kuyini hypotension echazwe kuvidiyo kule ndatshana.

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