I-glucose eneminyaka engu-18: inani elamukelekayo

I-Glycemia ingukuhlushwa kashukela emzimbeni womuntu. Okuqukethwe kwayo kuncike emsebenzini wezinqubo ze-metabolic, inani lokudla elichithwayo, ukusebenza kwezitho zangaphakathi nezinhlelo.

I-Monosaccharide (ushukela) “uphethiloli” osiza uhlelo lwangaphakathi lokusebenza. Umuntu uthola le ngxenye kuphela ekudleni, ayikho eminye imithombo. Ngokushoda, ubuchopho buqala ukuhlupheka.

Imvamisa yoshukela wegazi lapho uneminyaka eyi-19 iyefana neyabantu abadala. Akufanele ibe ngaphansi kwamayunithi angama-3.5, kepha hhayi ngaphezu kwama-5,5 amayunithi. Uma ukuhlolwa okuningana kukhombisa ukuphambuka, kudingeka ukwelashwa okufanele.

Izimo ze-Hyperglycemic kanye ne-hypoglycemic ziyingozi empilweni yabantu nokuphila. Ukwehluleka okuhlukahlukene kwenzeka ngaphakathi komzimba, okuvusa izifo ezingamahlalakhona, kwehlise kakhulu izinga lempilo.

Isimo sokuhlushwa ushukela ngineminyaka engu-19 ubudala

Ukuze uqonde ngokuphelele uma ngabe kukhula ama-pathologies amakhulu, udinga ukwazi ukuthi yini ushukela ojwayelekile emantombazaneni nakubabafana. Umkhawulo ovumelekile ugcinwa yi-insulin ye-hormone. Le nto yenziwa kusetshenziswa ama-pancreas.

Lapho i-hormone incane noma izicubu zingaboni "lokhu" kwengxenye, ukwanda kwesikhombi kwenzeka, okuholela ezinkingeni ezahlukahlukene. Eminyakeni engu-19 ubudala, imbangela yimikhuba emibi yokudla.

Emhlabeni wanamuhla, cishe yonke imikhiqizo yokudla iqukethe amakhemikhali, ama-preservatives, ama-flavour, njll, athinta kabi umzimba. Isimo siyanda ngokubhema, izimo ezicindezelayo.

Ukukhuluphala kungenye indlela yokukhula. Ukudla okungenamsoco eminyakeni eyi-18 kuya kwe-19 kuholela ekukhuluphiseni, ngokulandelana, kukhona ukuncipha kokuzwela kwezicubu kwe-insulin egazini. Ngokolwazi olunikezwe yi-World Health Organisation, amanani ajwayelekile alandelayo:

• Iminyaka yengane isukela ezinsukwini ezimbili kuya enyangeni eyodwa - amanani atholakalayo asukela ku-2.8 kuya ku-4,4 mmol / l.
• Kusuka enyangeni eyodwa kuye kufinyelela eminyakeni eyi-14, okujwayelekile kumelelwa ngokuhlukahluka kusuka kumayunithi angama-3.3 kuya kwangama-5.5.
• Ukusuka eminyakeni eyi-14 kuye kweyi-19, nakulabo abadala, amanani athembekile - amayunithi angama-3.5-5,5.

Lapho ushukela eneminyaka eyishumi nesishiyagalolunye, isibonelo, amayunithi ayi-6.0, khona-ke lesi yisimo se-hyperglycemic. Uma ngabe ukwehla kuya kumayunithi angama-3,2 noma kungaphansi, lesi yisimo se-hypoglycemic. Kungakhathalekile ubudala, lezi zimo ezimbili zibeka engcupheni impilo; ukulungiswa kwezokwelapha kuyadingeka. Ukungakunaki lokhu kuholela ekuhlukumezeni okuhlukahlukene, kufaka phakathi okungenakuphikwa.

Hlukanisa amanani egazi le-capillary (uketshezi lwegciwane lithathwe emunweni wesiguli) negazi le-venous (elithathwe emthanjeni). Ngokuvamile, imiphumela ye-venous imvamisa i-12%. Kuqhathaniswa nokuhlolwa kwegazi kusuka ngomunwe ngaphambi kokudla.

Ngaphezu kwalokho, uma ukuhlaziywa kokuqala kukhombisa ukuphambuka, isibonelo, amayunithi angama-3.0, khona-ke ukukhuluma nge-hypoglycemia akufanele. Ukuqinisekisa umphumela, isifundo esiphindaphindwayo siyimpoqo.

Uma intombazane eneminyaka engu-19 ikhulelwe, khona-ke kuye okwejwayelekile ushukela kufinyelela kumayunithi ayi-6.3. Ngaphezulu kwalesi paramende, ukubhekwa kwezokwelapha okuqhubekayo, ucwaningo olwengeziwe luyadingeka.

Ukubonakaliswa kwemitholampilo kweglucose ephezulu

Isifo sikashukela yisifo esingelapheki esihambisana nokuthathwa kukashukela emzimbeni. Unyaka nonyaka kutholakala iziguli zeminyaka ehlukile. Imvamisa, abafana namantombazane amancane banquma uhlobo lokuqala lokugula.

Eminyakeni yobudala, ezimweni eziningi, kutholakala isifo sohlobo 2. I-Pathology ingaqhubekela phambili iminyaka, futhi kaningi lapho isithola, isiguli sivele sinezinkinga ezahlukahlukene ngemithambo yegazi, umsebenzi wesistimu yezinzwa ephakathi, njll.

Ukwanda kweglue glucose kunganqunywa kusetshenziswa i-glucometer ekhaya. Leli thuluzi elikhethekile lizonikeza umphumela ofanele ngemizuzu. Kepha ukubonakaliswa kwemitholampilo kuyasiza nokusola lesi sifo:

1. Ukwehla amandla njalo, ukukhathala ngenxa yokuntuleka komzimba.
2. Ukwanda kwesifiso sokudla, kuyilapho kukhona ukwehla kwesisindo somzimba.
3. Umlomo owomile, uhlala womile. Ukuphuza amanzi akususi uphawu.
4. Ukuya njalo endlini encane, ukwabiwa komchamo okuningi.
5. Izinduna, izilonda, amathumba, njll. Ziyavela esikhunjeni. Lezi zilonda zikhathazeka isikhathi eside, zingaphili.
6. Iluma egunjini.
7. Isimo somzimba esinciphile, ukwehla kokusebenza.
8. Imikhuhlane ejwayelekile nezifo zokuphefumula, ukusabela komzimba, njll.

Lezi zimpawu zingakhombisa ukukhula kwesifo sikashukela. Kufanele kukhunjulwe ukuthi azibonwa zonke ndawonye; isiguli singase sibe nezimpawu ezi-2 kuya kwezi-4 zomtholampilo okuxoxwe ngazo ngenhla.

Engcupheni yiziguli ezinomlando wokungasebenzi kahle kwesibindi nezinso, ukukhuluphala ngokweqile, kanye nokukhuluphala ngokweqile. Okunye okwenza ukukhula kwalesi sifo ukuqunjelwa kwefa. Uma abazali benesifo sikashukela sohlobo 1, umuntu kufanele ayinake kakhulu impilo yabo, ngezikhathi ezithile anikele ngegazi ushukela.

Ngesikhathi sokukhulelwa, kubaluleke kakhulu ukuthola imbangela eholela esimweni se-hyperglycemic, ngoba kunokusongela okuphindwe kabili - kumama nengane. Imvamisa lapho eneminyaka yobudala engama-19, kwehle ukwehla kweglucose. Uma ungabuyisi ibhalansi ngesikhathi, lokhu kuholela ekukhathaleni nasezilinganisweni ezilandelayo zomzimba.

I-pathogenesis yoshukela ophansi kungenxa yekhefu elide phakathi kokudla, ukuzikhandla okukhulu ngokomzimba, ukuzila ukudla, njll.

Ucwaningo Sikashukela

Ekutholakalweni kwesifo sikashukela, isifundo esisodwa soketshezi lwezinto eziphilayo esivela emunweni asanele. Kuyadingeka ukwenza ucwaningo oluningana ukuze uqambe isithombe esiphelele.

Udokotela wakho angancoma ukuzimisela kokubekezelela i-monosaccharide. Umongo omfishane: bathatha igazi emunweni, bese banikeza isiguli umthwalo ngesimo se-glucose (chaza emanzini, udinga ukuphuza), ngemuva kwesikhashana kwenziwa elinye isampula legazi.

Ukuhlolwa kwemiphumela ngemuva kokulayisha ushukela:

• Uma kungekho zinkinga zempilo, khona-ke kufika kumayunithi angama-7.8.
• Isifo sikashukela (lesi akusona isifo sikashukela okwamanje, kodwa lapho sikhona kwezinto ezisikayo, isifo esingamahlalakhona siyakheka) - ukuguquguquka kwamayunithi angama-7.8-11.1.
• I-Pathology - amayunithi angaphezu kuka-11.1.

Ngemuva kwalokho kuyadingeka ukuthola ukuthi kusebenza kwe-carbohydrate metabolism emzimbeni. Ukuze wenze lokhu, udinga ukubala izici ezimbili. Owokuqala inani le-hyperglycemic, libonisa isilinganiso seglucose esiswini esingenalutho nangemva kokuzivocavoca. Inani layo ngokwesilinganiso akufanele lidlule amayunithi ayi-1.7. Inkomba yesibili yisibalo se-hypoglycemic, hhayi ephakeme kunamayunithi angama-1,3. Kunqunywa ushukela ngemuva kokulayisha emiphumeleni ngaphambi kokudla.

Lapho kukhona imiphumela engabazayo, ukuhlaziya kwe-hemoglobin ye-glycated kunganconywa njengokuhlaziywa okwengeziwe. Izinzuzo zayo ukuthi umuntu anganikela ngegazi ngemuva kokudla, kusihlwa noma ekuseni, okungukuthi, nganoma yisiphi isikhathi esikahle. Imiphumela ayinciki emithini eyenziwe, izingcindezi, izifo ezingamahlalakhona, umlando.

Okuqukethwe hemoglobin glycated:

I-Glycated hemoglobin iyisifundo esiphumelela kunazo zonke kukho konke ukunikezwa kwezokwelapha kwanamuhla. Noma kunjalo, inokungathandeki okuthile. Okokuqala, yile izindleko. Uma kunezinkinga nge-gland yegilo, kungahle kube nomphumela omuhle ongamanga. Nge-hemoglobin ephansi, kukhona ubungozi bomphumela ohlanekezelwe.

Ushukela wegazi ojwayelekile uyisihluthulelo somsebenzi ophelele wazo zonke izitho nezinhlelo. Uma kwenzeka kuphambuka, kuyadingeka ukuthi useshe izimbangela futhi uziqede.

Izinga likashukela wegazi lichazwe kuvidiyo kule ndatshana.