Naphezu kweqiniso lokuthi ukuxilongwa kwesifo sikashukela kuyathuthuka minyaka yonke, kuvela izindlela ezintsha nezidakamizwa zokunxephezela i-hyperglycemia, ukuxhaphaka kwalesi sifo kudale ukuthi iqiniso lokuthi isifo sikashukela singesesithathu ohlwini lwezifo eziyingozi kakhulu ngemuva kwenhliziyo ne-oncological.
Isifo sikashukela yisifo esibulalayo ekwakhiweni kwezinkinga. Imvamisa, babulawa yisifo sikashukela ngokukhula kokuhluleka kwezinso kanye nokuphazamiseka kokujikeleza kwezifo enhliziyweni nasebuchosheni.
Ngokwanda kwezinkomba zokwelashwa kwe-insulin nokutholakala kwalesi sidakamizwa kubantu, kanye nokwethulwa kwe-insulin eyenziwe ngomuntu emisebenzini yezokwelapha, ukufa okuvela kushukela ngenxa yokuthuthuka kukwehlile, kepha lokhu kungenzeka ngokuqapha okwanele kwamazinga kashukela nokunganakwa kwezincomo zikadokotela. .
Isifo senhliziyo njengengozi yokufa kwesifo sikashukela
Ushintsho lwethambo emikhunjini ezigulini ezinolwazi olude lwalesi sifo zitholakala cishe emaphesheni ayikhulu. Isizathu salokhu ukukhula kokuqala kwezinqubo ze-atherosulinotic esemncane ezinesifo sikashukela sohlobo 1 kanye nenkambo yesifo sikashukela sohlobo 2 esinzima.
I-atherossteosis kusifo sikashukela ihlelekile ngokwemvelo futhi ngokufanayo ithinta abesifazane nabesilisa. Izimbangela zokufa kwesifo sikashukela i-mellitus ezihambisana ne-atherossteosis infarction myocardial, ischemia noma i-cerebral hemorrhage, i-gangrene yemikhawulo engezansi.
Ukufakelwa kwemisipha ye-cardiac ezigulini ezinesifo sikashukela kwenzeka kaningi izikhathi ezingama-3-5 kunakulabo bonke abantu. Umtholampilo wakhe, njengokomthetho, unophawu oluphansi, ngaphandle kwesifo esijwayelekile sobuhlungu, okuholela ekuxilongweni sekwephuzile futhi siyimbangela evamile yokufa kwesifo sikashukela.
Inkambo yokuhlaselwa yisifo senhliziyo sikashukela inezici ezinjalo:
- Isilonda esikhulu.
- Ngokuvamile kungena kulo lonke udonga lwe-myocardium.
- Kuvela kabusha.
- Amafomu amakhulu ngokuqagela okungathandeki.
- Isikhathi sokululama eside.
- Umphumela obuthakathaka wokwelashwa kwendabuko.
Ukufa okuphezulu kwesifo sikashukela, kuhlangene ne-infyoction ye-myocardial, kubangelwa izinkinga ezifana nokushiswa kwe-cardiogenic, ukuboshwa okungazelelwe kwenhliziyo, ukuthuthukiswa kwe-aneurysm, i-pulmonary edema, ne-arrhythmia.
Ngaphezu kwe-myocardial infarction, iziguli ezinesifo sikashukela zivame ukuba nezimpawu zokuhluleka kwenhliziyo, i-coronary thrombosis, kanye nezinga eliphakeme le-hypertension ye-arterial. Bona, njengomthetho, baholela ezinkingeni eziyinkimbinkimbi, ezihlanganisiwe ezikhulisa inqubo yokuvuselelwa kwezifo zenhliziyo.
Ukuchaza izizathu zokuthi kungani isilonda esiyingozi semithambo singaba khona ngohlobo lwesibili lwe-mellitus yesifo sikashukela, kunezici eziningi ezibizwa ngokuthi: umphumela onobuthi we-hyperglycemia, i-cholesterol ekhuphukayo yegazi, ukwanda kokuqina, i-insulin ephezulu.
Ebusweni bemikhuba emibi ngendlela yokubhema, ukusetshenziswa kabi kotshwala, ukusebenza okuncane komzimba kanye nokusetshenziswa kwamafutha amaningi, ingozi yokufa ngaphambi kwesikhathi kushukela ikhuphuka.
Ingozi ye-nephropathy kushukela
Ukulimala kwesifo sikashukela okuqondile okuqondile kubizwa ngokuthi i-nephropathy. Kwenzeka ngenxa yokufakwa esikhundleni kwezicubu ezisebenzayo ngezicubu ezixhunywayo, ngokuncipha kancane kancane kokusebenza kwezinso kuze kube sekuthuthukisweni kokwehluleka kwezinso.
Lesi yimbangela evame kakhulu yokufa kuhlobo lwe-1 mellitus yesifo sikashukela, nasenkambeni ende yesifo sohlobo 2. Le nqubo ye-pathological ingahle ingabonakali ezigabeni zokuqala, okuholela ekutholeni isikhathi sekwephuzile, lapho ukulimala kwezinso okungalapheki kubangela ukwehla kokuhlunga kwe-glomerular futhi kuboniswe ngezimpawu ze-uremia.
Ukuze kutholakale i-nephropathy, zonke iziguli ezinesifo sikashukela ziboniswa ukuhlolwa komchamo kokuqukethwe kwamaprotheni, ukuzimisela kwesilinganiso sokuhlunga, kanye nokuhlolwa kwe-urea ne-creatinine. Ukulahleka njalo kwamaprotheni kumchamo kusho ukuthi iningi lama-glomeruli liyafa ezinso, futhi kusebenza kwawo ukuqeda ubuthi.
Nge-nephropathy yesifo sikashukela esiteji sokwehluleka kweziinso okungapheli, kuvela izimpawu ezilandelayo:
- I-Edematous syndrome iyakhula.
- Ukwanda kwengcindezi yegazi kuyaqhubeka.
- Izinga lenhliziyo liyanda.
- I-anemia itholakala egazini.
- Iziguli zikhononda ngobuthakathaka obukhulu, isicanucanu, ikhanda elibi nesikhumba esinyayo.
- Kunezimpawu zokuqongelela uketshezi emaphashini.
- Ukuphefumula kuyenzeka.
Ukuqhubeka kokuhluleka kwe-renal kudinga ukudluliselwa kweziguli ku-hemodialysis, ngaphandle kokufakelwa kwezinso, ukufa kusuka kwisifo sikashukela i-mellitus kwenzeka ngenxa yobuthi bomzimba ngemikhiqizo ye-metabolic, ukutheleleka, ukuhluleka kwenhliziyo.
Esigabeni esibulalayo se-nephropathy, kuvela ukoma we-uremic, okusho ukuthi umuntu uzofa kungekudala.
Isifo sikashukela se-polyneuropathy
Ukusuka kulokho, nokuthi abantu bafa kanjani, ngokuthuthukiswa komonakalo ohlelweni lwezinzwa njengohlobo lwe-diopic neuropathy, kuya ngesimo sayo. Enye yezindlela zokubonakaliswa kwalesi sifo ngokuthi isifo sikashukela sikashukela.
Ngenxa yokuphazamiseka kokujikeleza kwegazi nokufakwa emaphethelweni aphansi, i-ischemia yesisu esibuhlungu iba khona, okuholela ekwakhekeni kwe-gangrene ngesidingo sokunqunywa okuphuthumayo. Izilonda ze-peptic, ezingapholi isikhathi eside, zingadidaniswa nokutheleleka.
Ezimweni ezimbi kakhulu, ngokumelene nesizinda sokugomeka okwehlisiwe, iziguli zakha i-osteomyelitis bese ukutheleleka kungena emgodini wegazi - i-sepsis ejwayelekile.
Njengoba i-mellitus yesifo sikashukela ivame ukuphumela ekuphikisweni ngisho nakwezidakamizwa ezinamandla ze-antibacterial, ukufa kwabantu kusuka kushukela okunale nkinga kuyanda.
I-Hypoglycemic coma
Ngingabulawa yisifo sikashukela ngenxa yeglucose ephansi legazi? Lesi simo sivame ngokwelashwa kwe-insulin yohlobo 1 sikashukela. Ukwehla ushukela wegazi kwenzeka ngethamo eliphezulu le-insulin, ukungondleki, umsebenzi wesibindi ongasebenzi kahle, ukwehluleka kwezinso.
Imvamisa kakhulu i-hypoglycemia yenzeka lapho kuphuzwa utshwala, ikakhulukazi esiswini esingenalutho, kungacasulwa ukukhulelwa, ukuzalwa kwengane ngesifo sikashukela kanye nokuzivocavoca umzimba okuphezulu. Izimo zeComatose zivame ukuba yinkinga yokuphathwa kwe-insulin ngesikhathi sokungenelela kokuhlinzwa noma ukwelashwa kwe-ketoacidosis.
Njengoba unesifo sikashukela, ungafa ekwehliseni ushukela okubukhali, njengoba ikhehla likhula ngokushesha, kwesinye isikhathi kungakapheli nemizuzu eyi-10-10 ukulahleka kwengqondo nokuboshwa kokuphefumula. Kulokhu, kunezimpawu ezinjalo zomonakalo ezikhungweni ezibalulekile zobuchopho:
- Akukho okukhanyayo.
- Ithoni yemisipha iyancipha.
- Isigqi senhliziyo siphukile.
- Umfutho wegazi wehla kakhulu.
I-Hyperosmolar coma
Imbangela yokufa kohlobo 2 sikashukela i-mellitus kungaba ukukhula kwesimo se-hyperosmolar, okuwukubonakaliswa kokuwohloka okukhulu kwezinqubo ze-metabolic. I-Hyperglycemia ingafinyelela ku-35 -50 mmol / l, kukhona ukudonswa komzimba emzimbeni, ukwanda kokuqukethwe kwezingxube ze-sodium ne-nitrogen egazini.
Ukuthi bayafa yini isifo sikashukela ezimweni ezinjalo kuya ngokuthi ukuxilongwa kwenziwa kahle yini. Umtholampilo we-hyperosmolar coma kungaba ukubonakaliswa kokuqala kwesifo sikashukela, futhi isifundo saso sifana nezimpawu zengozi e-acute ye-cerebrovascular: paralysis, cramp of the low decrease, ukubanjwa ngamandla kwe-epileptoid, ukunyakaza kwamehlo okungahleliwe.
Esimweni se-hyperosmolar, akukho phunga le-acetone elisuka emlonyeni, ngoba alibonakali nge-ketoacidosis yesifo sikashukela, ukuphefumula kukaKusmaul akukho. Ukuphefumula okuphawulelwe ukuphefumula, ukuzindla, ukwehla kwengcindezi yegazi, i-edema yangaphakathi ehambisana ne-vein thrombosis ejulile iyaphawulwa.
Uma ukwelashwa kwe-infusion kungaqalwa ngokushesha, khona-ke iziguli zingabulawa yizimbangela ezinjalo:
- Umthamo wegazi onganele.
- I-pancreatic necrosis.
- Ukwehluleka kwezinso.
- I-Thrombosis, i-pulmonary embolism.
- I-Cerebral edema.
- Ingozi ye-cerebrovascular ajali.
Ividiyo ekulesi sihloko izachaza izimbangela zokufa kwesifo sikashukela.
