Ingozi ushukela wegazi amayunithi angama-24 - ukuthi wenzeni?

Amazinga kashukela egazi ajwayelekile aqala ku-3.3 kuya ku-5.5 mmol / L. Ukwanda kwalezi zinombolo kukhombisa i-prediabetes, isimo somkhawulo esisongela ukukhula ngokushesha kwesifo sikashukela. Amanani angenhla kwe-8-11 mmol / L ajwayele ukutholwa ushukela.

Futhi uma isifo sikashukela sinoshukela wegazi osondele kumayunithi angama-24, lezi yizinkomba eziyingozi kakhulu. Amanani anjalo atholakala kubantu abavame ukwazi ngesifo sabo. Umbuzo onengqondo uwukuthi, uma umuntu elashwa, ingakhuphuka kanjani i-glucose kumanani anjalo? Kukhona incazelo yakho konke.

Kungani abanesifo Sikashukela Bangangezela Ushukela

Kunezizathu eziningi zokunyuka okubukhali kweglucose yegazi. Kepha kaningi, izikhathi eziyingozi ezinjalo ziba umphumela wokungondleki, ukucindezela okukhulu, ukungasebenzi ngokomzimba nezifo ezithathelanayo (ngezifo ezithile, ukusebenza kwezitho zangaphakathi kukhubazeka kakhulu).

Zonke lezi zinto zingakhuphula ushukela ngisho nasezigulini ezinempilo. Ku-diabetes, amaconsi kashukela ajwayelekile, uma engabizwa ngokuthi angancani. Kepha uma ushukela ugxuma ngempela, udinga ukuqonda - ukuthi yini le ndaba, yiziphi izinto eziholele kulokhu.

Isifo sikashukela siyisigaba seziguli esidinga ngokusobala ukuqonda uhlobo lokugula kwazo. Isifo sikashukela sesiphenduka indlela yokuphila, ama-nuances ahambisana nalesi sifo angazikhumbuza nsuku zonke.

Kubaluleke kakhulu ukuthi umuntu angaziswanga ngokugula kwakhe kuphela, kepha futhi ngaphandle kokuthuthumela, abhekane nezinkomba eziphakeme ku-glucometer (ithuluzi lokulinganisa lokuhlushwa kweglucose yegazi). Kumele aqonde ukuthi yimaphi amanani amukelekile, okuyingozi, futhi abucayi.

Ingozi enkulu ngamanani aphezulu kashukela

Ukwanda kwamanani eglucose kubonakaliswa ukwanda kwesimo okubonakalayo, futhi lesi simo singafinyelela enkingeni - isigaxa se-ketoacidotic asibekelwa eceleni (ezigulini ezithembele ku-insulin - i-hypermolar coma).

Lapho ubungozi be-ketoacidotic coma bukhuphuka:

• Ngokunyuka kwamayunithi eshukela 16 +;
• Uma kutholakala i-acetone emchameni;
• Lapho ususa i-glucose ngomchamo ngaphezu kwe-50 g / l.

Okokuqala, umzimba ulawula ukwanda okuphawulekayo uqobo ngokwako. Umsebenzi wokunxenxa uyasebenza. Kepha ngemuva kwesikhashana, izimpawu ze-hyperglycemia ziyaziwa. Uma umuntu engasizwa ngesikhathi, ushukela ngeke wehliswe, kuzovela izinkinga.

Ukuthi ikomaacidotic coma isondela kushiwo:

1. Ubuhlungu besisu besekhaya obungacacile;
2. I-odorone ye-acetone emgodleni womlomo;
3. Isikhumba esomile;
4. Ukuthamba kwezibuko zamehlo;
5. Ukuphefumula okujulile;
6. Ukuphazamiseka kwengqondo kwe-dyspeptic.

Kungani amafutha esikhundleni se-glucose eqala ukuchitheka

Lapho umzimba ungakwazi ukusebenzisa ushukela njengomthombo wamandla, inqubo yokubuyiselwa kwemali isebenza, umzimba uthatha amafutha angenayo. Ngenkathi yokuguqulwa kwabo, kukhululwa ama-ketones. Lokhu akulutho ngaphandle kobuthi obunqwabelana kahle emzimbeni futhi buwushevu. Futhi ukudakwa okunjalo kubangelwe ukuthi kudale ukhubazeka kwesifo sikashukela.

Yini ekwazi ukubikezela inhlekelele ye-ketoacidosis:

• Izimpawu zokuqala zohlobo 1 sikashukela;
• Ukwelashwa okungalungile kohlobo lweshukela 1;
• Ukwehluleka ukuhambisana nokudla okunqunyiwe;
• Ama-pathologies athelelekayo ezithweni zezitho zofuzo;
• Isifo senhliziyo;
• Izifo zokuphefumula;
• Izinqubo zokuhlinzwa;
• Zonke izinhlobo zokulimala okungathi sína;
• Ukuphazamiseka kwe-Endocrine;
• Isikhathi sokuzala ingane.

I-ketoacidosis yesifo sikashukela ingabonakala ngeziqu ezintathu. Ngokwezinga eliphansi lenqubo ye-pathological, ukuchama kuba njalo, isiguli sizizwa sinezicanucanu, kubonakale izimpawu zokuqala zokudakwa. Kunephunga le-acetone emoyeni ogula okhathele.

Ngokwesilinganiso se-ketoacidosis esimaphakathi, isimo sesiguli siya ngokuya siba sibi. Izimpawu ze-inhliziyo ze-inhliziyo ebusweni, ukucindezela kuyancipha, i-tachycardia iqala. Isiguli siphazanyiswa ukuphazamiseka kwe-dyspeptic kanye nokungajabuli ku-epigastrium.

Izinga elinamandla le-ketoacidosis liyingozi ikakhulukazi - isimo siyadamba, abafundi abaniki ukusabela kokukhanya, nephunga elibi le-acetone. Izimpawu ezisobala zokuphelelwa ngamandla emzimbeni ziyatholakala.

Itholakala kanjani i-ketoacidosis?

Ngezimpawu zokuqala zesimo esinjalo, isiguli kudingeka sigijime emtholampilo, noma okungcono, shayela i-ambulensi.

Odokotela bazohlaziya izimpawu, bahlole umlando wezokwelapha, ama-pathologies ahlobene. Ngokuqinisekile bazokwenza uhlolo lwegazi oluphuthumayo ushukela, kanye ne-urinalysis ukuthola imizimba ye-ketone, kanye ne-acetone. Ukuhlolwa kwegazi kanye nomchamo, kanye nokunikela ngegazi kwe-biochemistry, kuzodingeka.

Ukwelashwa kuqala ngokuqinile ngemuva kokuxilongwa okuyikho. Imvamisa, i-pathology enjalo idinga ukwelashwa ezimeni zokuphinda uvuseleleke.

Izinketho zokwelapha zifaka:

• Ukwelashwa kwe-insulin. I-insulin inikezwa ushukela wegazi ophansi. Ukulawulwa kweshukela okulesi simo akususiwe.
• Izenzo zokuphelelwa amanzi emzimbeni. Uketshezi olulahlekile kufanele luphinde luphinde lwenziwe. I-saline iphathwe ngaphakathi kwethambo.
  Ukulungiswa kokwehluleka kwe-elektrogtiki.
• Ukwelashwa ngama-antibiotic. Lesi silinganiso siyadingeka ukuvikela ukuqhubeka kwezinkinga ezithathelwanayo.
• Ama-Anticoagulants.

Khula kanjani i-hyperosmolar coma?

Lesi simo siyaqhubeka kungakapheli amasonto amabili. Izinga le-glucose lingakhuphukela emazingeni okusongela - amayunithi angama-50, kuyilapho ushukela egxiliwe umchamo.

Izimpawu ze-hypersmolar coma ezingenayo:

1. Ukomela nobuthakathaka obukhulu;
2. Isikhumba esomile nolwelwesi lwe-mucous;
3. Ukunwaya amashiya;
4. Ukuntuleka kwephunga le-acetone;
5. Ukuphefumula okungaphakathi, okuvame kakhulu futhi okunobungako bokuphefumula.

Ubuhlungu besisu abutholakali ku-hypersmolar coma, kanye nokuphazamiseka komqondo. Kodwa ubungozi bokuthola ukuhluleka kakhulu kwe-renal kuphezulu, ngakho-ke usizo kufanele lube lusheshe.

Nge-hypoglycemia, umuntu angaba nomuzwa ongajwayelekile wendlala, kwenzeka i-euphoria, isimilo singase sibe yinqaba, ukuhlangana kuzophazamiseka, kuqale ukuvela isifo, isiguli sikhononda ngokuba mnyama emehlweni.

Ingabe kukhona amaphutha ekwelashweni kwe-insulin?

Kumuntu onesifo sikashukela, ukugxuma okunjalo kushukela njengoba amayunithi angama-24 kunamaphutha amaningi ekwelashweni. Okusho ukuthi, uma umuntu elashwa, khona-ke akufanele kube khona amanani aphakeme kangako. Isiphetho silula: ukwelashwa kunikeze uhlobo oluthile lokungasebenzi kahle, futhi lokhu kungenzeka kakhulu.

5 amaphutha ejwayelekile wokwelashwa kwe-insulin:

1. Hhayi lolo hlobo lwezidakamizwa. Namuhla, kusetshenziswa izinhlobo ezi-4 eziyinhloko ze-insulin - lezi zibizwa ngokuthi ama-insulin amafushane futhi abambelela ngokufushane, zinikezwa ngaphambi kokudla futhi zisebenzela ukulungisa izinga likashukela elikhuphukile. Lokho okubizwa ngokuthi ama-insulin asebenza phakathi nendawo kanye nokufaka insulin isikhathi eside kuzogcina amanani ajwayelekile eglucose yegazi lapho ama-insulin amafushane “engahambelani. Futhi uma usebenzisa amasu we "insulin emfushane nede" umuntu angadideka ohlelweni. Kodwa kaningi, i-insulin ekulesi simo namanje ayikhuphukeli emazingeni okusongela, ngokuvamile ushukela, kunalokho, kwehla kakhulu.
2. Shintsha ipeni lesirinji le-insulin. Namuhla, amapeni wesirinji ayakwazi ukusebenzisa noma ukusebenziseka kabusha. Lezi zokugcina zithandwa yiziguli eziningi. Kepha uma uthatha ipeni lomunye umuntu, yebo upotoza inaliti yakho kukho, kunengozi yokubamba ukutheleleka kothile.
3. Yeqa umjovo noma insulin enganele yokugula. Isibonelo, umuntu unomkhuhlane wesikhathi noma umkhuhlane. Futhi ngesizathu esithile, abanye abanesifo sikashukela bakholelwa ukuthi isimo esinjalo nemijovo ayihambelani. Kepha lokhu kuyiphutha elikhulu! Ngesikhathi sokugula, izinga le-glucose lingakhuphuka ngisho nangokwengeziwe, ngoba isenzo sama-hormone we-contra-hormonal sizoba nomthelela kulokhu.
4. I-insulin ayigcinwanga kahle. Amasheya womuthi kufanele agcinwe kahle. Kufanele babe esiqandisini, izinga lokushisa elidingekayo lingama-2-6 degrees. Ukulungiselela okuvulekile kuvunyelwe ukugcinwa kuze kufike izinsuku ezingama-28 kwidatha yokushisa yasekamelweni, kepha kude kuphela nokushisa nemisebe ye-ultraviolet. Awukwazi ukuphatha isidakamizwa esineqhwa, ngoba umuthi onempilo yeshelufu ephelelwe isikhathi awuphathwanga, nomuthi ogcinwe ngokungeyikho.
5. Ukusebenza ngokomzimba esiqongweni se-insulin. Amakilasi noma ukuhamba emgwaqweni, okwenzeka ngenhla kwesinyathelo se-insulin, nakho kungaholela ekushintsheni okukhulu kwamanani. Kuliqiniso, imvamisa namanje kusenzelwa i-hypoglycemia.

Kepha udinga ukuhlola zonke izinguqulo ezingenzeka. UShukela wagxuma kakhulu - xhumana nodokotela, futhi ungaqageli ukuthi yini okungahambanga kahle. Qikelela ngesikhathi sokukhuphuka kwezifo ezingamahlalakhona, ngaleso sikhathi ubungozi bokugxuma eglucose kukhulu ikakhulukazi.

Eqa ushukela ne-pancreatic atrophy

I-pancreatic atrophy yisimo esibonakala ngokuncipha kosayizi besitho, kanye nokucekelwa phansi kwesakhiwo se-gland nokungasebenzi kwayo. Kaningi, ushintsho olunjalo lwenzeka ngomzimba onesifo sikashukela esiqhubekayo.

Izimpawu ezijwayelekile zalesi sifo kubuhlungu kuyi-hypochondrium yangakwesobunxele, ukuqhuma njalo ngemuva kokudla, ukudumba kolimi (kuba okusajingijolo noma okubomvu), ukuphrinta kwamazinyo noqwembe olubonakalayo olimini. Ngokushesha lapho kuvela izimpawu ezinjalo, udinga ukuya kudokotela we-gastroenterologist. Udokotela uzonikeza izifundo ezifundisayo ukunquma i-pathology.

Ushukela wegazi amayunithi angama-24 - okumele ukwenze

Ushukela onjalo ophakeme uvame ukunqunywa kuma-diabetes, futhi umuntu ubona lezi zinombolo emamitha lapho ethatha isilinganiso. Ngokuvamile, amanani aqakathekileko atholakala ekuhlolweni kwegazi okujwayelekile, kepha izinkomba ezinjalo endaweni yesibhedlela (ngokwesibonelo, ngemuva kokuhlinzwa) zivame kakhulu.

Uma isiguli ekhaya sithola ushukela omningi kumamaki anjalo, udinga ukuxhumana nodokotela. Isinqumo esilungile kunazo zonke ukubiza i-ambulensi.

Qiniseka ukuthi uchazela ochwepheshe ukuthi yini edale ukuthi kugxume ushukela, okwandulelayo ukuwohloka kwenhlala-kahle. Khumbula ukuthi udlile, ingabe wayifaka i-insulin kahle nangesikhathi (uma unesifo sikashukela esincike ku-insulin). Imininingwane enjalo enembile izohambisana nezenzo zabodokotela.

Kuyiqiniso, akunakwenzeka ukulindela ukuthi "konke kuzodlula ngokwakho". Amayunithi angama-24 izinga likashukela eliphakeme kakhulu, ngakho-ke ukungasebenzi akunakuthethelelwa. Ukubukwa kwesiteshi kanye nokuhlolwa okugcwele kukhonjisiwe. Uma amanani anjalo eglucose etholakala kwelinye lamalungu omndeni, izihlobo kufanele zazi ukuthi lowo muntu udinga ukwelashwa.

Ividiyo - imithetho yokwelashwa kwe-insulin.