Izimpawu zesifo sikashukela emadodeni nakwingozi yayo

Masisho khona manjalo ukuthi ngenkathi isifo sikashukela singelapheki. Umgomo wokwelapha ukuvumelanisa ushukela wegazi, ngamanye amazwi, isinxephezelo sikashukela. Uma owesilisa egcina i-glucose ejwayelekile isikhathi eside, izinkinga azikhuli, futhi abanye baphinde bacindezeleke. Ukuphila kahle noshukela onxephezelwe kulungile, azikho izimpawu zesifo, isikhathi sokuphila sifana nakwabesilisa abaphilile. Ngenxa yemvelo yesifo sikashukela, ukuvimbela kubalulekile. Isifo sikashukela singagwenywa ngosizo lwezinyathelo ezilula ezifakana nomqondo wendlela yokuphila enempilo.

Izimpawu kanye nezimpawu zesifo sikashukela esingamukeli

Izigameko zesifo sikashukela ziyanda unyaka nonyaka. Emncane, amadoda amaningi ayagula kunabesifazane; lapho eseneminyaka engama-55 ubudala, inani labanesifo sikashukela sobabili ubulili liyafana. Ngokwezibalo, sonke isiguli sesithathu asazi ukuthi sesikhule isifo sikashukela, asizizwa zimpawu futhi siyaqhubeka nokuphila indlela esijwayelekile, sandisa imiphumela yalesi sifo.

Ekhuluma ngesifo sikashukela emadodeni ngemuva kweminyaka engama-30, imvamisa asho isifo esivame kakhulu (ngaphezu kwe-90% yamacala) isifo se-2. Imvamisa eminyakeni yokuqala ngemuva kokuqala kokuphazamiseka kwe-metabolic, lolu hlobo lwesifo sikashukela alunazo izimpawu ezishiwoyo, lungatholakala kuphela ngosizo lokuhlolwa.

Isifo sikashukela nokucindezela kuzoba yinto yesikhathi esedlule

• Ukwehlela ushukela -95%
• Ukuqedwa kwe-vein thrombosis - 70%
• Ukuqedwa kokushaya kwenhliziyo okuqinile -90%
• Ukuqeda umfutho wegazi ophakeme - 92%
• Ukwanda kwamandla ngesikhathi sosuku, ngcono ukulala ebusuku -97%

Siqala kanjani isifo sikashukela:

Lezi zimpawu zivame kakhulu ngasikhathi sinye, imvamisa amadoda aqaphela eyodwa noma ezimbili zazo. Kwenzeka ukuthi izimpawu zoshukela omningi zibonakale kuphela emavikini ambalwa esigabeni sokuqala sikashukela, ngemuva kwalokho umzimba uthola ukujwayela izimo ezintsha, futhi yonke imizwa engathandeki inyamalale.

Izimpawu zokuphela kwesifo sikashukela emadodeni zenzeka ngemuva kweminyaka eminingana yokuphila noshukela ophezulu:

1. Ukuphulukiswa okuncane kwamanxeba amancane, ukubukeka kwama-scuffs ezinyaweni, esifundeni se-inguinal noma se-axillary.
2. Ukwanda kwevolumu yesisu, ukwakheka kwe-"lifebuoy" enamafutha edlulela ezinhlangothini futhi ingxenye kuya emuva.
3. Iluma emadodeni ethweni le-glans, azungeze i-anus, ngaphakathi kwe-urethra.
4. Ukhunta emlonyeni, kwizinzipho, emathangeni esikhumba, ezizalweni zangasese. Izimpawu zesifo sikashukela emadodeni yi-balanitis noma i-balanoposthitis. Izimpawu zabo ukubaluka, ibala elimhlophe, ubomvu bekhanda lobumba kanye / noma ijwabu. Ukuvuvukala kubangela ubuhlungu ngesikhathi sokulala naphakathi kokuchama.
5. Isikhumba esomile, ikakhulukazi ezinyaweni, amathole nezandla. Kamuva, ukuqunjelwa kwesikhumba kuqala, bese kuvela amabala abomvu kumasayithi wokucwilisa.
6. Ukudonsa emilenzeni, iminwe. Isifo sikashukela sibuye sikhonjiswe ngesimo sokuphazamiseka ekuzwelweni kwezindawo ezithile zesikhumba, ukugoba ebusuku amathole.
7. Ukuncipha kokuzwela kwephenti ye-glans, izinkinga nge-erection.
8. Umbono ofiphele. Izimpawu zomthelela omubi wesifo sikashukela e-retina - owesilisa ubona inkungu, izindawo ezimpunga ezimpunga zingavela phambi kwamehlo akhe.
9. I-Black acanthosis ingenye yezimpawu ezingavamile zesifo sikashukela. Kubonakala ngesimo sezindawo ezimnyama zembala emphethweni wesikhumba, imvamisa ezindongeni noma ezinsolweni.

Amanye amadoda akaziboni izimpawu kuze kube ukuthi ushukela ukhuphuka aze afike ezinombolweni eziyingozi - 14 mmol / L nangaphezulu. Nge-hyperglycemia ephezulu kangako, umzimba awukwazi ukulawula i-metabolism, futhi ukubola kwayo kwenzeka. Izidumbu zeKetone ziqala ukungena egazini, okuholela ekutheni kube nephunga le-acetone lomchamo, umoya ophelile, kwesinye isikhathi isikhumba. Uma ungasinaki lesi sibonakaliso, lesi simo singaba sikhulu ezinsukwini ezimbalwa sibe ikhehla lesifo sikashukela.

Uhlobo lwe-1 carbohydrate ukuphazamiseka luboniswa inkambo esheshayo. Izimpawu zesifo sikashukela kubantu besilisa ziyafana nakuhlobo 2 lwesifo, kodwa zivela endabeni yamahora. Ushukela ukhula ngokushesha, ezinsukwini ezimbalwa isiguli singawela enkingeni. Emadodeni emva kweminyaka engama-40, ukudalwa kwesifo sikashukela sohlobo 1 kuyisifo. Uma izimpawu zikashukela ophezulu ziphakama ngokuzumayo futhi zanda ngokushesha, kufanelekile ukuthi uhlole uhlobo oluphakathi lwesifo phakathi kohlobo 1 nohlobo 2 - isifo sikashukela.

Izimbangela eziphambili zesifo sikashukela emadodeni

I-glucose ingumhlinzeki wamandla ophambili womzimba wethu, inqwaba yawo siyithola ekudleni. Ngemuva kokugaya ukudla futhi wephule noshukela oyinkimbinkimbi, ushukela ungena egazini. Ukuze iqhubekele phambili emangqamuzaneni, umzimba udinga i-insulin. Le hormone yenziwa ama-pancreas ukuphendula ukwanda koshukela wegazi.

Esigabeni sokuqala sesifo sikashukela emadodeni, ukumelana ne-insulin kukhula kancane kancane. Imbangela yalesi simo wukudla ngokweqile ushukela egazini kusuka ekudleni okune-carb ephezulu. Abathengi abakhulu be-glucose yimisipha. Ezingeni eliphansi lomsebenzi, izindleko zincane, okusho ukuthi amaseli ngazo zonke izindlela angavimbela ukungena koshukela okungadingekile kuwo. Bangakwenza lokhu ngendlela eyodwa: ngokunganaki i-insulin, okungukuthi, ngokuthuthukisa ukumelana ne-insulin.

Izizathu ezandisa amathuba ohlobo 2 emadodeni:

1. Ukudla okunempilo.
2. Ukukhuluphala ngokweqile Lapho umuntu ekhuluphele ngokweqile, kuphakama ukumelana kwakhe ne-insulin.
3. Ukusebenzelana kwasekhaya kwamafutha omzimba. Ukukhuluphala okuyingozi kakhulu okujwayelekile kwabesilisa yisisu. Izimpawu zalo: amanoni aqongelela phakathi kwezitho, kwakhiwa isisu esikhulu esinzima. Isibonakaliso sengozi enkulu yesifo sikashukela ivolumu yesisu esingaphezu kwama-94 cm.
4. Ukungasebenzi, ukuntuleka kwesisindo semisipha. I-glucose engaphansi ichithwa empilweni, kulapho ihlala khona egazini.
5. Izici zokuzizwa. Ukuqagela kokulimala kwe-carbohydrate metabolism kungabikezelwa ngokufunda umlando wezihlobo eziseduze. Lesi sizathu asinqumi. Indoda esebenzayo emamatheka ayinabo neze ubungozi besifo sikashukela, noma ngabe bobabili abazali bayo benalesi sifo.
6. Iminyaka. Ngemuva kweminyaka engama-50, isifo sikashukela emadodeni sivela kaningi ngamaphesenti angama-30 kunasemncane. Odokotela basikisela ukuthi lokhu kungenxa yokuthola isisindo nempilo engejwayelekile. Bheka i-athikili - amazinga kashukela egazini emadodeni ngemuva kweminyaka engama-50.

Izindlela zokuxilonga

Izimpawu ezihlukaniswa kahle zesifo sikashukela emadodeni amaningi zivame ukuvela kuphela nge-glucose ephezulu yegazi. Lokhu kuvame ukwenzeka eminyakeni engama-5-7 ngemuva kokuqala kwesifo. Njengomthetho, ukuhlolwa ngalesi sikhathi sale sifo kwembula izinkinga ezingaphezu kwesisodwa.

Ungasibona kanjani isifo sikashukela kusenesikhathi:

1. Ukuhlaziywa okulula kunazo zonke i-glucose yegazi; amasampula enziwa kusuka kumthambo kuphela esiswini esingenalutho. Kumuntu onempilo, umphumela kufanele ungabi ngaphezu kwe-5.9 mmol / l, unesifo sikashukela - ngaphezulu 7. Uma lokho kuhlaziywa kwenziwa kusetshenziswa imitha yeglucose yasekhaya, ushukela ongaphezulu kuka-6.1 uphawu lokuxwayisa. Ama-Glucometer anokunemba okuphansi, ngakho-ke, ukuhlaziywa kwelebhu kuyadingeka ukuqinisekisa lesi sifo.
2. Ukuhlolwa kokubekezelela kwe-glucose kuzosiza ukukhomba ukwanda kokungamelana kwe-insulin esigabeni se-prediabetes. Igazi lidonswa kabili ngaphakathi kwamahora ama-2, phakathi kwamasampula isiguli esisebenzisa ushukela. Umphumela wesilinganiso sesibili ngaphezulu kwe-7.8 ulinganiselwa njenge-prediabetes, ngaphezulu kwe-11.1 - njengesifo sikashukela.

Eminyakeni yobudala ephakathi nangaphakathi kuyadingeka ukuthatha izivivinyo njalo eminyakeni emi-3, kwabesilisa abanamathuba aphezulu wesifo sikashukela - minyaka yonke.

Ukwelashwa kwesifo sikashukela ezigabeni ezihlukene

Uhlelo lokubuyiswa kwesifo sikashukela alula. Uma lesi sifo sitholakele emabangeni okuqala, ukulahlekelwa ushukela, kwanele ukunciphisa umzimba ngokweqile, ukukhuphula umsebenzi (kusuka emaminithini we-150 wokuqeqeshwa okunamandla ngesonto), ukwakha izicubu zomzimba, kanye nokudla okuphansi kwe-carb. Ukunciphisa ukumelana ne-insulin, i-metformin (Siofor et al.) Imvama ukunqunywa. Amaphutha wokudla okunempilo angalungiswa kancane kusetshenziswa i-acarbose (Glucobay).

Uma isifo sikashukela sitholwa endodeni ngemuva kweminyaka eminingana, khona-ke ukudla, ezemidlalo kanye ne-metformin ukukhokhela lesi sifo kungahle kunganele. Ngalesi sikhathi, ukukhiqizwa kwe-insulin kwehla, ngakho-ke iziqobosho ze-pancreatic zibekelwa ngokwengeziwe: i-glibenclamide yendabuko (Maninil), glimepiride (Amaril), vildagliptin yanamuhla (Galvus), sitagliptin (Januvia) nabanye.

Uma unamathela ekudleni okuphansi kwe-carb futhi uthatha amalungiselelo athambile we-pancreatic, i-insulin synthesis kushukela inganwetshwa amashumi eminyaka. Lapho ushukela wegazi uvame ukwedlula okujwayelekile, izinkinga ezisebenza ngokuzikhandla zizothuthuka, ngokushesha ama-pancreas azoncipha futhi ayeke ukukhiqiza i-insulin.

Lapho nje i-insulin esemuntwini inganele i-carbohydrate metabolism ejwayelekile, kuzofanele ifakwe umjovo. Ekuqaleni, imijovo engu-1-2 ngosuku yanele. Uma ukwakheka kwe-insulin sekuyekile ngokuphelele, uhlelo lokwelashwa kwe-insulin luba ngokufanayo ngohlobo lwesifo sikashukela sohlobo 1: kusuka kumijovo emi-4 ngosuku.

Izinkinga nemiphumela

Inani elincane labanesifo sikashukela (ngaphansi kuka-5%), ubunzima buba nokwelashwa okufika ngesikhathi. Cishe i-15% yamadoda inenhlanhla. Banokuthuthuka okuthe chu kancane ngisho nokulawulwa kancane koshukela. Ebuningini obukhulu, ubukhulu bezinkinga zesifo sikashukela ngokuqondile buya ngezinga lesinxephezelo salo.

Isifo sikashukela siyingozi kubo bonke, ngaphandle kokukhetha, izicubu zomzimba wendoda. Imbangela ye-pathologies izinqubo ze-glycation, noma ushukela, eziqina kaningi uma ushukela wegazi enyuswa. Okokuqala, izikejana ezihlangana okokuqala noshukela ziyahlupheka. Izindonga zabo zilahlekelwa yithoni, zibe zintekenteke, ziyeke ukufeza imisebenzi yazo. Ama-capillaries amancane ayachithwa, izingosi zezicubu zihlala ngaphandle kokuhlinzekwa kwegazi. Imiphumela ye-hyperglycemia ibonakala ikakhulukazi kwizitho ezithinta ukusebenza kwazo ngokungenakwenzeka ngaphandle kwenethiwekhi ebanzi yemikhumbi: izinso ne-retina. Ukwehlulwa kwemikhumbi emikhulu kuholela ekuphikelelekeni kokuqina komfutho wegazi, izikhathi eziningana kwandisa inani lokuhlaselwa yinhliziyo nemivimbo emadodeni.

I-Glycation ye-hemoglobin enoshukela omningi ingafinyelela ku-20%, ngenxa yalokho amaseli abhekana nendlala yomoya ophela njalo. Ukuhlushwa isifo sikashukela nezinzwa. Ama-Neuropathies okokusebenza kwasendaweni okuhlukile ayimbangela yobuhlungu obungalapheki ezingalweni nasezilimeni zezinyawo. Isifo sikashukela sithinta amandla abantu besilisa. Ukunikezwa kwegazi emithanjeni ye-cavernous kuba ngokunganele, imizwa yezitho zangasese iyancipha ngenxa ye-neuropathy. Ukulimala ezinhlizweni kuholela eqinisweni lokuthi ama-sphincters ayeka ukusebenza ngokujwayelekile, i-ejaculation ejikelezayo iyakhula: isidoda asiphumi ngaphandle, kepha singena ebhlorini, okwenza ingqondo yemvelo yengane ingenzeki.

Ukuvimbela

Ukukhishwa kwezici ezandisa amathuba wesifo sikashukela kuyizinyathelo eziphambili zokuvimbela kwaso. Ukwehla kwesisindo kokujwayelekile, ukuncishiswa okubukhali kwama-carbohydrate, ukususwa ngokuphelele koshukela okulula (ushukela, i-sucrose) ekudleni, ukuqeqeshwa njalo kungaqinisekisa ukuvimbela ushukela. Noma esigabeni se-prediabetes, lapho ushukela usuvele wedlulele ngalokujwayelekile, lezi zindlela zokuvimbela zisebenza ku-75% wamadoda.