Namuhla, kunabantu abacishe babe yizigidi ezingama-366 abanesifo sikashukela emhlabeni wonke. Ngokusho kweRegistry Yombuso waseRussia ekuqaleni konyaka we-2012, zingaphezu kwezi-3.5 iziguli ezazinalesi sifo esibuhlungu ezazibhalisiwe ezweni. Ngaphezu kwama-80% wabo asevele enezinkinga zesifo sikashukela.
Uma uthemba izibalo, khona-ke ama-80% eziguli afa ngezifo zesimo senhliziyo. Izimbangela eziphambili zokufa kwabanesifo sikashukela:
- isifo sohlangothi;
- infarction myocardial;
- gangren.
Ukufa akuveli kulesi sifo uqobo, kodwa kusuka ezinkingeni zako
Ngalezo zinsuku lapho i-insulin ingekho, izingane ezivela kwisifo sikashukela zafa ngemuva kweminyaka engu-2-3 yokugula. Namuhla, lapho umuthi ufakelwe ama-insulin anamuhla, ungaphila ngokugcwele ngesifo sikashukela kuze kube ukuguga. Kepha kunezimo ezithile zalokhu.
Odokotela bahlala bezama ukucacisela iziguli zabo ukuthi azife ngqo ngenxa yesifo sikashukela. Izimbangela zokufa kweziguli yizinkinga ezifakwa yilesi sifo. Abantu abangu-3,800,000 abanesifo sikashukela bafa minyaka yonke emhlabeni. Lesi sibalo esesabekayo ngempela.
Iziguli ezinolwazi kahle ezimweni eziningi zivame ukuphuza imithi ukuvikela isifo sikashukela noma ukwelapha esivele satholakala. Uma inqubo isivele isiqalile, khona-ke ukuyiyeka kunzima kakhulu. Imithi iletha impumuzo isikhashana, kepha ukululama okugcwele akwenzeki.
Ungaba kanjani? Ngabe ayikho indlela yokuphuma nokufa kuzofika kungekudala? Kuyavela ukuthi yonke into ayesabeki kangako futhi ungaphila nesifo sikashukela. Kunabantu abangaqondi ukuthi izinkinga ezihlasela kakhulu zeshukela sikashukela wegazi ophezulu. Yile nto enomphumela onobuthi emzimbeni, uma ingaphandle kokujwayelekile.
Kungakho nje izidakamizwa ezisanda kufakwa zidlala indima enkulu ekuvinjelweni kwezinkinga, okokuqala ukulondolozwa kwansuku zonke kwe-glucose egazini ezingeni elifanele.
Kubalulekile! Izinto zokwelapha zisebenza kahle lapho amazinga kashukela egazini evamile. Uma ngabe le nkomba ihlala i-overestimated, ukuvikela kanye nokwelashwa kungasebenzi. Empini yokulwa nesifo sikashukela, inhloso eyinhloko ukubuyisela ushukela glucose kube okujwayelekile.
Ushukela omningi ulimaza izindonga zemithambo yegazi nama-capillaries. Lokhu kusebenza ohlelweni lonke lokunikezela ngegazi. Yomibili imithambo ye-cerebral and coronary is ethintekile, imikhawulo engezansi (unyawo sikashukela) iyathinteka.
I-Atherossteosis (i-atherosulinotic plaque) iba yimikhumbi ethintekile, okuholele ekuqhekekeni kwe-lumen ye-vascular. Umphumela we-pathology enjalo yile:
- ukushaya kwenhliziyo;
- isifo sohlangothi;
- ukunqunywa kwengalo.
Ingozi yesifo senhliziyo nohlobo 2 sikashukela ikhuphuka ngezikhathi ezingama-2-3. Shono lezi zifo zisendaweni yokuqala ohlwini lokufa okuphezulu kweziguli. Kepha kunezinye izizathu ezinkulu ongabulawa kuzo.
Ucwaningo oluthokozisayo luyaziwa olufakazele ubudlelwano obuqondile phakathi kwemvamisa yokulawulwa kwe-glycemic kanye nezinga le-glucose egazini legazi ezigulini ezinesifo sikashukela sohlobo 1.
Kuyavela ukuthi uma ulinganisa izinga le-hemoglobin ye-glycated izikhathi 8-10 ngosuku, ingagcinwa ebangeni elihle.
Ngeshwa, ayikho idatha enjalo yeziguli ezinesifo sikashukela sohlobo 2, kodwa akunakwenzeka ukuthi ukukala okuqhubekayo kungasenza lesi simo sibe sibi, kungenzeka kakhulu, kusazothuthuka.
Ezinye izimbangela zokufa kusuka ku-Type 1 sikashukela
Impela abantu abaningi bayazi ukuthi izinkinga zesifo sikashukela ziqinile futhi zingamahlalakhona. Okuxoxwe ngenhla kuphathelene nezinkinga ezingamahlalakhona. Manje sizogxila ezinkingeni ezinkulu kakhulu. Kukhona imibuso emibili enjalo:
- I-Hypoglycemia nekhoa kungumphumela kashukela omncane wegazi.
- I-Hyperglycemia ne-coma - ushukela uphezulu kakhulu.
Kukhona ne-hyperosmolar coma, etholakala ikakhulukazi kwiziguli esezikhulile, kepha namuhla lesi simo sinqabile kakhulu. Kodwa-ke, kuholela nasekufeni kwesiguli.
Ungawela enkomeni ye-hypoglycemic ngemuva kokuphuza utshwala, futhi amacala anjalo ajwayelekile. Ngakho-ke, i-alcohol ingumkhiqizo oyingozi kakhulu wesifo sikashukela futhi kuyadingeka ukwenqaba ukubuphuza, ikakhulukazi ngoba ungaphila ngokuphelele ngaphandle kwawo.
Ukudakwa, umuntu akakwazi ukuhlola kahle isimo futhi aqaphele izimpawu zokuqala ze-hypoglycemia. Labo abaseduze bangavele bacabange ukuthi umuntu uphuze kakhulu futhi angenzi lutho. Ngenxa yalokhu, ungalahlekelwa ukwazi futhi uwe ekhefini le-hypoglycemic.
Kulesi simo, umuntu angachitha ubusuku bonke, futhi ngalesi sikhathi izinguquko zizokwenzeka ebuchosheni ezingabuyiswa. Sikhuluma nge-edema yamakhemikhali, esezimweni eziningi igcina ngokufa.
Noma odokotela bekwazi ukususa isiguli ekhefini, akunasiqiniseko sokuthi amakhono akhe engqondo nawezimoto azobuyela kumuntu. Ungaphenduka "imifino" ephila kuphela ekhombisa.
I-ketoacidosis
Ukwanda okungapheli kwamazinga kashukela okuqhubeka isikhathi eside kungaholela ekuqongeleleni kobuchopho kanye nezinye izingxenye zomzimba wemikhiqizo yamafutha oxidation - ama-acetones nemizimba ye-ketone. Lesi simo saziwa kwezokwelapha ngokuthi yi-ketoacidosis yesifo sikashukela.
I-Ketoacidosis iyingozi kakhulu, ama-ketones anobuthi kakhulu ebuchosheni bomuntu. Namuhla, odokotela bafunde ukubhekana ngempumelelo nalokhu kubonakaliswa. Usebenzisa izindlela ezikhona zokuzithiba, ungavimbela lesi simo ngokuzimela.
Ukuvimbela i-ketoacidosis kuqukethe ukulinganisa njalo amazinga kashukela egazini futhi uhlole umchamo ngezikhathi ezithile nge-acetone usebenzisa imichilo yokuhlola. Umuntu ngamunye kufanele azenzele iziphetho ezifanele. Ngemuva kwakho konke, isifo sikashukela kulula ukusivimba kunokulwa nezinkinga zempilo yami yonke.
