Imiphumela emibi futhi engapheli yesifo sikashukela esibolile

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Isifo sikashukela esingakhokhwanga yisimo esiyingozi esingadala imiphumela emibi yezempilo. Imvamisa, le ndlela ye-pathology ibangelwa ukwephulwa kwendlela yokuphila, amaphutha ekudleni, noma ukuntuleka kwezidakamizwa ezidingekayo.

Ngakho-ke, kubaluleke kakhulu ukuxhumana nochwepheshe ngokushesha ngangokunokwenzeka futhi ulandele ngokusobala ukuqokwa kwakhe.

Umnyombo we-pathology

Abantu abaningi banesithakazelo kulo mbuzo: isifo sikashukela esibolile - siyini? Ngale ndlela yalesi sifo, umthamo ushukela awuncishiswa yimithi, obangela ubunzima obukhona.

Ukulawula ukusebenza ngempumelelo kwesifo sikashukela, ama-endocrinologists ahlukanisa izigaba eziningana ze-pathology:

  1. Isinxephezelo - zonke izinkomba zisondele kokujwayelekile. Umuntu akanalo ukoma, ukwanda kokuchama, noma ukoma emgodini womlomo.
  2. Ukukhokhelwa - ngalesi sikhathi izinhlaka zikashukela zanda kancane.
  3. Ukubuyiswa kwemali - kubonakaliswa ukwanda okungapheli koshukela wegazi. Kulesi sigaba, usongo lokuqhamuka nemiphumela eyingozi luphezulu. Zihlotshaniswa nokwelashwa okungafanele. Imvamisa, isigaba sokudonswa kwemali sibhekwa kuhlobo 1 sikashukela. Kodwa-ke, uhlobo lwesibili lwe-pathology luyingozi impela.

Izizathu

Ukuqonda ukuthi siyini isifo sikashukela esibolile, kufanele uhlaziye izimbangela zokuvela kwaso. Imvamisa, lesi simo kungenxa yokudla ngokweqile.

Uma isiguli sidla ama-carbohydrate amaningi, i-insulin nezinye izidakamizwa azikwazi ukugcina ivolumu evamile yeglucose.

Ngaphezu kwalokho, ukubola kwesifo sikashukela kuwumphumela wezinto ezinjalo:

  • Ukwelashwa okungalungile. Imbangela yezinkinga kungaba ukusetshenziswa kwemithamo emincane kakhulu yezidakamizwa. Futhi, i-pathology imvamisa iba umphumela wokuphazamiseka kokwelashwa kwezidakamizwa.
  • Ukusetshenziswa kwezithasiselo zokudla. Izinto ezinjalo zingasetshenziswa kuphela njengesengezo empahleni egxilile. Izithako kulezi zindlela zinomphumela oqondile kokuqukethwe ushukela. Ukuze uzinze ushukela, kukhonjiswa kuphela izidakamizwa.
  • Ukusetshenziswa kwemithi yasekhaya esikhundleni semithi.
  • Ama-pathologies ashubile wokutheleleka. Ukwehla kwesifo sikashukela kuvame ukuvela ngenxa yezifo ezithile eziphawuleka ngokuqina komzimba okuwohloka kanye nokulahleka kukamanzi.

Ucwaningo lokuxilonga

Ukuthola ushukela ongabaliwe, izindlela eziningi zokuxilonga ziyadingeka.

Kunemikhawulo eyisihluthulelo eminingana ekuvumela ukuthi uhlole isigaba sokunxeshezelwa

  • Inani loshukela emchameni;
  • IGlycated hemoglobin;
  • Inani le-glucose egazini lilinganiswa ngemuva kokudla nasesiswini esingenalutho.

Ngokubalulekile ukuhlolwa kwenani lama-triglycerides, ingcindezi, i-cholesterol. Futhi, odokotela bavame ukunquma inkomba yesisindo somzimba.

Isifo sikashukela esigabeni sokubola sibonakaliswa ngamapharamitha alandelayo:

  1. I-Glycated hemoglobin engaphezu kuka-7.5%;
  2. Inani loshukela emchameni lungaphezu kuka-0,5%;
  3. Umthamo we-glucose ngemuva kokudla ungaphezu kwe-10 mmol / l;
  4. I-cholesterol inkulu kune-6.5 mmol / L;
  5. Amazinga okushukela asheshayo adlula ku-7.8 mmol / L;
  6. Ukucindezela kuka-160/95 mm Hg. st.;
  7. Inkomba yesisindo somzimba iyakhuphuka - imvamisa ukuwohloka kwesifo sikashukela kuhambisana nokukhuluphala;
  8. Okuqukethwe kwe-triglyceride kukhulu kune-2.2 mmol / L.

Amapharamitha wokhiye angalawulwa ekhaya. Kulokhu, kufanelekile ukuba ne-glucometer. Le divayisi isiza ekuboneni inani loshukela egazini. Ukukhwabanisa kwenziwa ngesisu esingenalutho kanye namahora ambalwa ngemuva kokudla.

Imichilo yokuhlola, edayiswa kuyo yonke ikhemisi, isiza ekuboneni i-acetone noshukela kumchamo.

Imiphumela emibi

Uma isifo sikashukela singalashwa, ukubola kuzobangela imiphumela emibi. Ama-pathologies ashubile aqhubeka ngokushesha okukhulu.

Esimweni esinjalo, ukunakekelwa kwezokwelashwa ngokushesha kuyadingeka. Imiphumela eyingozi kakhulu ifaka:

  • I-Hyperglycemia;
  • I-Hypoglycemia;
  • Isifo sikashukela sikashukela.

Nge-hyperglycemia, kukhona ukwanda okusheshayo kwevolumu ushukela. Ngokuthuthuka kwayo, ubuthakathaka, ukoma, indlala kuyabonakala. Ukuze ubhekane nalokhu kwephulwa, udinga ukuthatha umjovo we-insulin ngokushesha.

I-Hypoglycemia - ukwehla okungazelelwe kwe-glucose yegazi. Lapho kuvela, ubuthakathaka nendlala enkulu kuzwakala. Uma ungamsizi umuntu, angawela enkingeni. Ukuze uzuze ushukela osheshayo, kufanele uphuze itiye noshukela noma udle uhlobo oluthile lobumnandi.

Isifo sikashukela esinesifo sikashukela singaba nezinhlobonhlobo eziningana. Amafomu weKetoacidotic, hyperosmolar, lactic acid ahlukaniswa. Ngamunye wabo ubonakala ngezimpawu ezithile kanye nomehluko wezinkomba zokwelashwa.

Esimweni esinjalo, isiguli kufanele silaliswe esibhedlela ngokushesha.

Imiphumela engapheli

Isifo sikashukela esiwohlokile sivame ukuveza imiphumela emibi. I-Pathology ithinta kabi isimo semithambo yegazi, umbono, isikhumba nezinye izitho.

Isikhumba nolwelwesi lwe-mucous

Kunemiphumela eminingana ethinta kabi i-dermis ne-mucous epithelium:

  1. I-dermopathy yesifo sikashukela - izinqubo ezingekho emthethweni esikhunjeni zivusa ukulahleka kwamanzi. Ngokubakhona okuqhubekayo kwalesi simo, i-dermatitis iyenzeka. Ebantwini, kuvela izindawo ezinemibala enamandla esikhunjeni. Ukwakheka kwezilonda zecophic emaphethelweni aphansi kungenzeka futhi.
  2. I-Xanthomatosis - leli gama libhekisela ekuhlukumezeni kwesikhumba, okuyizinsana ezibomvana. Zinobukhulu obuhlukahlukene futhi zihambisana nezinkinga ku-lipid metabolism. Ukwakheka okunjalo kutholakala endaweni yezinkalweni nezindalo zezinyawo.
  3. I-Lipoid necrobiosis - kwenzeka ezimweni ezinzima zesifo sikashukela. I-Pathology ihambisana nokwakhiwa kwamagungquza esikhunjeni, abizwa ngokuthi ama-papules. Njengoba inqubo iqhubeka, ama-dermis ezindaweni ezithintekile ayafa futhi abe yi-yellow hue. Kamuva, izilonda zokulimala ziyavela kulezi zindawo.
  4. I-Lipodystrophy - ngentuthuko yayo, ungqimba lwamafutha luyanyamalala esikhunjeni. Le nqubo ibonwa lapho kusetshenziswa amalungiselelo aphansi we-insulin. Kukhona ubungozi befomu le-hypertrophic, lapho, kunalokho, kukhiqizwa khona amafutha amaningi. Lokhu kungenxa yokulimala kwemetabolism yamafutha.
  5. Ukukhuluphala kwe-dysplastic - ngaphansi kwethonya le-insulin, ushukela udonswa izicubu ezinamafutha. Lokhu kudala ukuthi amadiphozithi avele endaweni engenhla yomzimba, kanti imilenze iba mncane.

Amathambo kanye nokuhlangana

Ukwehla kwesigaba sikashukela kubangela izilonda ezahlukahlukene zamalunga kanye nezicubu zamathambo. Imvamisa, i-pathologies enjalo ikhula:

  1. Isifo sikashukela sikashukela - sihambisana nokushintshwa kwamalunga nokuthuthukiswa kwezifo, okuhambisana nokwenziwa kwezilonda. Ngokuthuthukiswa kwale nqubo, kaningi kunezinkinga ezikhona ohlelweni lwezinzwa, lwe-metabolism, kanye nesakhiwo se-vascular. Imvamisa, i-pathology ithinta izinyawo, kepha kwesinye isikhathi izandla nazo ziyahlupheka.
  2. I-systemic osteoporosis - i-pathology yathuthuka kancane kancane, okuholela ekunciphiseni kwezicubu zamathambo. Ngenxa yalokhu, ama-fractures aba njalo kaningi. Ama-vertebrae kanye nokuhlangana kwe-hip kuvame ukuthinteka.

Izitho zokugaya

Isifo sikashukela esigabeni sokudonswa kwemali sivame ukwephula umsebenzi wokugaya ukudla. Okokuqala, umgodi womlomo uyahlupheka. Lokhu kuhambisana nokukhula kokulimala kwamazinyo we-carious, isifo se-periodontal, gingivitis.

Lapho i-pathology iqhubeka, kuvela ezinye izinkinga:

  • Isicanucanu
  • Ubuhlungu esiswini;
  • Ukuboniswa kwe-anorexia;
  • Ukopha.

I-Pathology idala izinkinga ngokufakwa kwezakhi zomzimba kanye nokulimazeka kwemoto yokusebenza. Lokhu kudala ukuqedwa kokuhlanganiswa kwama-enzyme ne-acid.

Ngokuvamile kuba nezinkinga ezinjalo:

  1. I-Enteropathy yesifo sikashukela - isibonakaliso esiyingqayizivele kuhudo. Ezimweni ezinzima, ukungahlali kahle kwefec kuyabonakala.
  2. I-hepatopathy yesifo sikashukela - ibonakala ngokuncipha kwezitolo ze-glycogen kanye nokwanda kwethonya le-lipid esibindini. Lokhu kuthukuthelisa hepatosis enamafutha. Lezi zinqubo zibangela ukuqina nokwanda kwesibindi. Isikhumba siphuzi.

Izinhlobo zemibono

Uhlobo olubunjiwe lukashukela luvame ukudala izinkinga zokubona.

Lokhu kufaka lokhu okulandelayo:

  • I-retinopathy yesifo sikashukela - ibonakala ngokwanda kwama-capillaries e-retina. Lapho izinto zangaphandle zinyamalala, kuvela ama-aneurysms nama-hemorrhages. Emazingeni okuqala, lokhu kucishe kungenzeki, kepha, njengoba lesi sifo sikhula, kunengozi yokwehla okukhulu kombono.
  • I-cataract yesifo sikashukela - ngokushintshashintsha kwezinga elibukhali kwevolumu ushukela, ilensi iba ngamafu. I-Pathology inentuthuko esheshayo, ngoba izindlela ezijwayelekile zokwelapha azisizi.

Uhlelo lwezinzwa

I-mellitus yesifo sikashukela engakhanswanga ivame ukudala ukuthuthukiswa kwe-neuropathy. Kulokhu kungalingani, izinguquko ze-pathological emikhunjini zinesici, esithinta ukusebenza kwemicu yezinzwa. Njengomphumela, imisebenzi ebucayi, ye-ezizimele kanye neyezimoto iyathinteka.

Ukuvimbela

Ukuvimbela ukukhula kwesifo sikashukela esibolile, udinga ukulandela lezi zincomo:

  • Namathela ngokuqinile ekudleni okunqunywe udokotela;
  • Thatha ngendlela ehlelekile umuthi ukuze wenze ushukela ajwayelekile
  • Njalo lawula umthamo weglucose;
  • Ziyahlolwa;
  • Nikeza ukusebenza komzimba okukhishwa.

Manje uyazi ukuthi ukubola kweshukela kuyini ukubola. Leli gama libhekisela esimweni esibucayi kakhulu esidala izinkinga eziyingozi. Ukugwema lezo zinkinga, Kubalulekile ukulandela ngokusobala ukuqokwa kwezokwelapha. Ekuwohlokeni okuncane kakhulu enhlalakahleni, kufanele uthinte uchwepheshe.

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