"Slow killer", unesifo sikashukela: iyiphi ingozi yesifo futhi yimiphi imiphumela engaholela kuyo?

Onolwazi be-endocrinologists bathi zonke iziguli ezinesifo sikashukela zingahlukaniswa izigaba ezimbili.

Ukuwa kwangaphambilini ukwethuka kanye ne-hypochondria, bahlala belalela bona uqobo, bebheka izimpawu ezintsha.

Ngengozi bathatha umuthi, owelulekwa ngumakhelwane emnyango, baphendukela kwabelaphi, abalaphi bamakhambi futhi bacishe baba ngama-shamans. Ngokwemvelo, indlela enjalo ayikwazi ukuholela kunoma yini enhle. Eyesibili ayinaki ngokuphelele inkinga, inethemba lokuthi "mhlawumbe."

Leli yiqembu leziguli eliyingozi kakhulu. Uma ungakunaki ukwelashwa, izinkinga ngeke zithathe isikhathi eside. Futhi, ngeshwa, isigaba esincane kunazo zonke, sesithathu. Lababantu bahlola lesi simo ngokwanele futhi basize udokotela, afeze ngokugcwele ukuqokwa kwakhe.

Lo mbhalo unikezelwe ezigulini ezivela emaqenjini amabili okuqala. Umuntu kudingeka aqonde ukuthi isifo sikashukela asisisho isigwebo; umuntu kufanele akhethe ukwelashwa okwenele. Okwesibili ngeke kulimaze ukwazi ukuthi yiziphi izinkinga ezifakwe yilesi sifo. Amaqhinga e-Ostrich awakaze asize muntu. Ngasikhathi sinye, ukuqaliswa kokuqokwa kochwepheshe kuzogwema izinkinga futhi kuthuthukise kakhulu impilo.

Akusikho nje ukuthi kunegama elithi “umuntu onempilo enempilo”. Kuyadingeka ukulwela ngokunembile ukunxephezela lesi sifo. Uma ushukela ugcinwa ezingeni elijwayelekile, izinkinga ngeke zikhule. Noma ngabe zinjalo, khona-ke ngesifo esinxephezelwe, ukubuyisa kungatholakala. Njengoba isinxephezelo silungiswa, isimo siya ngokuya siba ngcono.

Ushukela ophezulu wegazi uthinta kanjani umzimba?

Njengomthetho, izimbangela zenkinga yesifo sikashukela zihlangana nomunye, ngakho-ke, kuleso naleso simo, akunakwenzeka ukuthi sibeke noma yisiphi isici esidabukisayo:

1. ukuphakama kwe-glucose. Uma phakathi nosuku kukhuphuka njalo (njengenketho, ukushintshashintsha okuncane kungenzeka), khona-ke izinkinga zingavinjelwa. Isikhathi esithile, umzimba uzosebenzisa izindlela zemvelo zokuncephezela, khona-ke azinqunyelwe. Uma ukhomba ukukhula kwezinkinga ngesikhathi, ungazihlehlisa. Kepha ingozi ukuthi ama-pathologies akhula kubantu abahlukene ngejubane elingalingani. Ngakho-ke, kubalulekile ukulandela isheduli yezivivinyo zokuvimbela. Le yindlela yangempela yokugcina impilo nokusebenza;
2. izinguquko ezisheshayo nezibalulekile kushukela wegazi. Ushintsho olukhulu kakhulu kushukela ngesikhathi sasemini ngu-5 mmol / l, ezinganeni - 7 mmol / l. Ijubane lokunciphisa ushukela nalo libalulekile (inani eliphakeme liyi-4 mmol / l). Uma ushukela wehla ngokushesha kakhulu, khona-ke izimpawu ze-hypoglycemia zingavela ngokufundwa okujwayelekile kwamamitha kashukela;
3. izici ngazinye. Kuyenzeka ukuthi izinkinga ziba khona ngisho nangoshukela onxephezelwe. Ngenhlanhla, lokhu kwenzeka kaningi. Isizathu yikuba sengozini komuntu ngamunye kwesitho, njenge "sixhumanisi esibuthakathaka ". Ngokwezibalo, ama-5% abantu aba nezinkinga ngisho noma enyuka ushukela omfushane futhi olinganiselayo.

Uma kungenakwenzeka ukuthonya isizathu sokugcina nganoma iyiphi indlela, khona-ke okokuqala nokwesibili kungenzeka. Okuyingozi ikakhulukazi amazinga aphezulu kashukela.

Bazifunela abanesifo sikashukela "abanolwazi" lweminyaka eminingana. Ubuqili nobungozi babo ukuthi abonakali kakhulu, kepha kancane kancane. Noma ngabe welashwa ngendlela eyiyo, akunaso isiqiniseko esiphelele sokuthi imiphumela esephuzile ingagwenywa.

Umthelela enhliziyweni nemithambo yegazi

I-Angiopathy - Lesi isilonda se-vascular, futhi sikhula ngokushesha kakhulu kunezinye izinkinga.

Okokuqala, ukwakheka kwezicubu zomzimba kuyaphazamiseka, amathuba okuba i-atherosclerosis ayanda, futhi nengozi ye-thrombosis iyanda.

Ukwephula kobuningi bemithambo yegazi, ushintsho esakhiweni sezindonga zabo kuholela eqinisweni lokuthi ukulethwa kwegazi kwizitho nokunikezwa kwezakhi zomzimba kuya ngokuya kukhula kancane. Ingozi yesifo senhliziyo nemivimbo, ekhathaza abanesifo sikashukela ngokwangempela, ikhula kakhulu.

Ubungozi ohlelweni lwezinzwa lomuntu

Ukusatshiswa kwemivimbo sekushiwo. Ngaphezu kwalokho, ukuphazamiseka kwesistimu yezinzwa ezinjenge-polyneuropathy kuvame ukutholakala.

Ngaphansi kwaleli gama kuncipha kokuzwela kwezingalo kubuhlungu nokushisa.

Isimo sikhonjiswa ngokuxineka nokukhubazeka ngesimo "samagilavu" kanye "nesitoko". Imizwa engathandeki iyaqina ebusuku. Ubuthakathaka emaphethelweni bajoyina le nto. Kwesinye isikhathi isiguli sihlushwa yizinhlungu ezihlala zihlangana.

Yini esongela izitho zombono?

Inkinga evamile yombono yi-retinopathy. Lesi yisilonda ngemuva kwesifo sikashukela se-ocular retina.

Ngokuqhubeka kwesifo, ukuphuma kwaso kwenzeka, ukuphuma kwesibindi esikhwameni. Umbono uya ngokuya ukhula kancane kancane, ngisho nobumpumputhe obugcwele kungenzeka.

Uma "okuhlangenwe nakho" kwesifo sikashukela sekuyiminyaka engama-20, khona-ke ingozi yokuthola i-retinopathy cishe ikhulu. Imvamisa ngokumelana nesizinda se-hyperglycemia, i-cataract nayo iyakhula - ukugcwala kwelensi yeso.

Ukuthuthukiswa kwezifo zesikhumba, amathambo nemilenze

Isifo sikashukela - Enye yezinkinga ezingemnandi kakhulu, ezivame kakhulu futhi eziyingozi zesifo sikashukela.

Endaweni yezinyawo, kwakhiwa izilonda ezingaphili kahle, izilonda, kuze kufike ezindaweni zezicubu ezifile. Imbangela yezilonda eziyizidudla ukwephula ukunikezwa kwegazi esikhunjeni. Bapholisa kanzima. Unembe ngamunye walesi sango uyisango elivulekile lokutheleleka.

Ngakho-ke, abanesifo sikashukela badinga ukubheka ngokucophelela inhlanzeko yonyawo, bagweme ukugxila kwezipikili, futhi bakhethe izicathulo ezifanele. Ikhulukazi kulabo abanesifo sikashukela, kukhululwa amasokisi anganakubamba gum emlenzeni.

Umphumela omubi ezinso nasezibindini

Umsebenzi wezinso ongasebenzi kahle ekugcineni uholela ekuthuthukiseni ukwehluleka kwezinso. Lesi sifo (isifo sikashukela) sikhula kancane, ngaphandle kokukhombisa izimpawu ezigabeni zokuqala.

Kunezigaba ezinje zesifo sikashukela:

• hyperfiltration - kuboniswa yi-renal hypertrophy, ukwanda kosayizi wabo;
• i-microalbuminuria. Isibonakaliso esibi ukubonakala komchamo wamaprotheni we-albhamuin;
• i-macroalbuminuria - ukukhuphuka kwenani lamaprotheni adalulwa emchazweni, e-hypertension;
• ukuthuthukiswa kokuhluleka kwezinso.

Ngokuqondene nokulimala kwesibindi, ngokumelene nesizinda se-hyperglycemia, kuvela i-metabolic syndrome, ephelezelwa i-steatohepatitis, i-steatosis, ukungasebenzi kahle kwama-metabolism. Umongo walezi zimo ukunqwabelana kwamafutha esibindini futhi, ngenxa yalokho, ukwenziwa kabusha kwamaseli awo.

Isifo sikashukela sikashukela

Isifo sikashukela esinesifo sikashukela sibonisa kulolo hlobo 1 sikashukela sohlobo 2. Kunezinhlobonhlobo zako:

• ketoacidosis - Lokhu kunqwabelana egazini lezidumbu ze-ketone - imikhiqizo ye-metabolism yamafutha. Kwenzeka ezigulini ezinesifo sikashukela sohlobo 1. Izizathu zokulimala, amaphutha ekudleni, ukungenelela kokuhlinzwa. I-ketoacidosis ibonakaliswa ukulahlekelwa ukwazi, ukwephulwa okubukhali komsebenzi wezitho ezibalulekile;
• i-hyperosmolar coma. Le nkinga ijwayelekile kwiziguli ezinesifo sikashukela sohlobo 2, kakhulu kubantu asebekhulile. Lesi simo sibonakaliswa ukwanda kokugxila kwegazi kwe-glucose ne-sodium, ukomisa. Ukuboniswa: umuzwa wokoma ngokweqile, ukukhuphuka komchamo okwandayo;
• hypoglycemia - Ukwehla okubukhali (kwesinye isikhathi kuye kwizinombolo eziphansi kakhulu) ekugxambeni kwe-glucose egazini. Lesi simo sibonakaliswa yilokhu: ukwethuka, ukujuluka ngokweqile, ukuphazamiseka kokuziphatha (njengokudakwa kotshwala). Uma kungekho zinyathelo ezisebenzayo zokusiza, ukulahlekelwa ukwazi, ukweyisa, kuze kufike ku-hypoglycemic coma ,joyina. I-Hypoglycemia ingaholela kokuthi: ukuphuza utshwala, ukuzikhandla okukhulu ngokomzimba, ukuthatha (futhi ikakhulukazi i-overdose!) Kwezidakamizwa ezithile;
• i-lactocidotic coma - ukunqwabelana kwe-lactic acid egazini. Kwenzeka kubantu abanesifo sikashukela asebekhulile abahlushwa isifo sikashukela, izinso, inhliziyo nesibindi. Izibonakalisa njengokwehla komfutho wegazi, ukuntuleka komchamo, ukwazi ukungasebenzi kahle.

Iningi lalezi zimo lithuthuka cishe ngokushesha okukhulu. Okuhlukile yi-hyperosmolar coma, ethuthuka kancane kancane, kuze kufike amasonto ambalwa. Ingozi enkulu ukuthi ukukhomba lezi zingqinamba akulula. Kunokugula okuningi okufana ngakho kunzima ukucabanga ngezimpawu ezisongela.

Iziphi izinkinga ezingakuholela lapho ukhulelwe?

Amazinga kashukela aphezulu ayingozi kowesifazane okhulelwe nakibelethayo, ngakho-ke, kudingeka ukuthi kuhlolwe igazi ngezikhathi ezithile.

I-glucose ephezulu kakhulu evunyelwe lapho ukhulelwe ngu-5.1 mmol / L:

• 5.1-7.1 mmol / L - isifo sikashukela sokukhulelwa;
• ngaphezu kwe-7.1 mmol / l - kuboniswa isifo sikashukela, okungukuthi, esisodwa esizohlala ngemuva kokuzalwa kwengane.

Izingozi ezivame kakhulu kwabesifazane abakhulelwe abanesifo sikashukela.

• ukuvuvukala kwezinso;
• preeclampia;
• izinkinga zokuzalwa.

Ubungozi bokukhipha isisu ngokuzimela luyanda. Ngokwezibalo, le yingxenye yesithathu yazo zonke izinto zokukhulelwa ezinesifo sikashukela. Isizathu ukuguga kwangaphambi kwesikhathi kwe-placenta, okuholela ekubulambeni i-oksijini endle.

Isifo sikashukela siyingozi kanjani enganeni?

Izinkinga ezinkulu zesifo sikashukela ezinganeni nasentsheni ziyefana nakwabantu abadala: hyper-, hypoglycemic and hyperosmolar coma, ketoacidosis.

Ngokuqondene nezinkinga ezingapheli, lokhu kufaka:

• uhlelo lwezinzwa - i-neuropathy, i-encephalopathy;
• izinso - i-nephropathy;
• amehlo - retinopathy.

Ukwehla kokungazeleleki kuholela ezifweni ezithathelwanayo. Kunezimo ezejwayelekile zesifo sofuba.

Isifo sikashukela nomdlavuza: kukhona ukuxhumana?

Odokotela banamuhla bayathemba ukuthi ingozi yokuba neoplasms enesifo sikashukela iphezulu kunakuqala kubantu abanempilo enhle. Ngokwezibalo, abantu abanesifo sikashukela bavame ukuba nomdlavuza wendlala, isimila samanyikwe, izinso, amathumbu kanye nesinye.

Isifo sikashukela esihambisana ne-anemia

Imbangela ye-anemia (ukwehla kokuxineka kwe-hemoglobin) kungukuphazamiseka komsebenzi wezinso futhi, ngenxa yalokho, ukuncipha kokuqashelwa kwe-erythropoietin ye-hormone.

Amangqamuzana abomvu egazi ajwayelekile futhi ane-anemia yokuntuleka kwe-iron

Ukuntuleka kwale hormone kuholela ekuwohlokeni emsebenzini womongo wamathambo abomvu, owenza umsebenzi we-hematopoietic. Izimbangela ezingeziwe ze-anemia kwisifo sikashukela ukulahleka okukhulu kwamaprotheni okukhishwe kumchamo nokushoda kwensimbi.

Kanye nokwelashwa nge-vitamin complexes, i-erythropoietin inikezwa abanesifo sikashukela, okuholela ekujuleni kwesithombe segazi.

Izici zokwelashwa kwamakhaza kushukela: amaphilisi namakhambi

Iziguli ezinesifo sikashukela zithambekele kumakhaza ngenxa yokwehla kokungaziguli. Udinga ukuqaphela kakhulu ngokuthatha umuthi.

Inketho enhle ukuhlanganisa ukuphathwa kwamathebulethi nodokotela wakho. Kuyasiza ukwehlisa umkhuhlane (impumulo egelezayo, ukukhwehlela, ikhanda) i-linden itiye noma amanzi ashisayo ngokufakwa kwe-ginger.

Kuzokwenzekani uma ungasitholi isifo futhi uselapha?

Ngaphezu kwezinkinga zesifo sikashukela "esisheshayo", kunezifo eziningi ezenzeka ndawonye. Ukunganaki ukwelashwa kungaholela ekukhubazekeni noma ngisho nasekufeni. Ingozi yezinkinga ingancishiswa uma lesi sifo sinxeshezelwe. Kusho ukuthini leli gama?

Ukukhethwa kwenqubo yokuthola isinxephezelo kuya ngezinto ezinjalo:

• iminyaka yesiguli;
• "isipiliyoni" sikashukela;
• ukuba khona kwezinkinga nezifo ezihlangene.

Ukuqapha isimo kanye noshukela wegazi kuyadingeka kunoma yisiphi isimo. Ngokudabukisayo, ngokweqile okuncane kokugxila kwe-glucose, kufanele kube okuxinzekayo, ngoba ubungozi be-hypoglycemia buyanda, yize kungenjalo ngesimo esinzima kakhulu.

Uma isifo sikashukela sihambisana nezifo ze-vascular, khona-ke "ukuswayipha" okunjalo kuyingozi.

Nawa amanani "kashukela" okuhlosiwe "wezigaba ezahlukahlukene zeziguli.

• akukho zinkinga zesifo sikashukela; akukho bungozi be-hypoglycemia - 6.5-7.5%;
• kunezinkinga kanye nengozi yokwehla okubukhali koshukela - 7.0-8.0%.

Izindlela zokuvimbela nazo zibaluleke kakhulu:

• ukudla okufanele. Akukhulunywa ngokudla okusha okusha. Futhi ukudla okuvunyelenwe ngakho-ke okondlayo kanye ne-endocrinologist, ngaphandle kokudla okusheshayo namabha we-chocolate, kutholakala kuwo wonke umuntu;
• umsebenzi womzimba. Ngokusho kochwepheshe, izinga layo lesonto lesifo sikashukela kufanele libe amahora angama-150. Lokhu ukuhamba nsuku zonke imizuzu engama-30, imishini yokuzivocavoca yasekuseni yokuhlanzeka, ukubhukuda, ukuhamba ngamabhayisekili, ukuqina, njll. Yebo, imithwalo eyeqile kufanele igwenywe;
• ukwenqaba ngokuphelele utshwala nogwayi;
• Ukuzilawula njalo kwengcindezi, isisindo somzimba futhi, yebo, ushukela. Kungcono ukugcina idayari yesifo sikashukela, ukurekhoda kuyo hhayi izinkomba zokucindezela kwegazi, ishayela kanye ne-glucose, kepha futhi nokudla okudliwayo. Lokhu kuzovumela udokotela ukuthi akhombe izimbangela zokwehluleka ukwelashwa futhi azilungise.

Amavidiyo ahlobene

Mayelana nemiphumela yesifo sikashukela kule vidiyo:

Ngokwezibalo, inani leziguli ezinesifo sikashukela emhlabeni selwedlule izigidi ezingama-300. Ukubonakaliswa kwalesi sifo kumuntu ngamunye kuyindlela yomuntu ngamunye. Ukubambisana kuphela nodokotela ohambelayo, ukuhambisana nazo zonke izidingo kuzogwema ukuqhubeka kwesifo kanye nezinkinga ezihlobene.