I-Atherossteosis iwukunciphisa kwemithambo ebangelwa ukugcotshwa kwamafutha okufaka odongeni lomthambo. Lama-deposits angamafutha aholela ekunciphiseni kokugeleza kwegazi kwezicubu. Ngaphezu kwalokho, izingcezu zamafutha zingaqhuma futhi zivimbe isitsha segazi. Yonke imithambo ingathinteka, kepha imithambo ye-coronary and cerebral ibaluleke kakhulu, ngoba ukuhamba kwegazi okunganele kuya enhliziyweni nasengqondweni kungafaka ingozi enkulu empilweni yomuntu. I-Ear atherossteosis nayo ingeyolu hlu.
I-Atherossteosis kanye nezinkinga zayo (isifo senhliziyo esine-coronary, stroke) zingenye zezimbangela ezinkulu zokufa. Ukuhlaselwa yinhliziyo kukodwa kuhlanganisa ama-20% abo bonke ukufa minyaka yonke.
Lapho kufe abantu abafa imivimbo kanye nesifo senhliziyo esisanda kufakwa, inani eliphelele lokufa okubangelwa yi-atherosclerosis lenyuka lifinyelela cishe ku-50% wenani. Ukwelapha lesi sifo kubiza ngaphezu kwama- $ 60 ezigidigidi ngonyaka.
Izimpawu nezimpawu kuncike kubungako bokuvinjwa nemithambo ethintekayo. Lokhu kungafaka:
- izinhlungu esifubeni
- imilenze emincane (ikakhulukazi lapho uhamba);
- ubuthakathaka
- Isiyezi
- ukuwohloka kancane kancane.
Ezinye izimpawu "ezincane", ezivame ukubangelwa ukuncipha kokugeleza kwegazi kwe-atherossteosis, kufaka phakathi i-tinnitus (tinnitus), ukungabi namandla, ukulahleka kokuzwa, ukungasebenzi kahle kwamehlo. Imvamisa ngaphambi kokuhlaselwa yisifo senhliziyo, unhlangothi, akunazimpawu.
Izimbangela zokukhula kwalesi sifo
Njengoba kushiwo ngenhla, amadiphozithi angakheka kunoma iyiphi ingxenye yomzimba.
I-ararososososis yendlebe ivame ukutholakala, lapho ukuhlinzwa ngakhona nokwelashwa okufanelekile kusiza kuzosiza.
Imiphumela yesifo ingaholela ekukhuleni kwezithulu noma ekuxilongeni kakhulu (ngokwesibonelo, unhlangothi).
Izimbangela ze-atherosclerosis zaziwa kakhulu:
- Indlela yokuphila yokwehlisa ukuthula.
- Ukubhema.
- Ukungalingani kokudla.
- Ukuxineka
Futhi uma zonke lezi zizathu zihlanganiswa nomunye, lapho-ke ubungozi bokuthola ukugula buye bukhuphuke ngezikhathi ezithile. Njengoba konke lokhu kuyizici zokulawulwa kwengozi, umuntu angenza yonke imizamo ukuvimba le nqubo yokuwohloka.
Kusukela ngo-1973, kuyaziwa ukuthi i-diagonal fold ye-earlobe iwuphawu lwe-atherosclerosis. Ucwaningo lwakamuva luye lwabonisa ukuthi lokhu, empeleni, kungenye yezinkomba ezinembile kakhulu ze-atherosclerosis - ethembekile kakhulu kunanoma iyiphi enye into eyaziwayo ebeka engcupheni, kufaka phakathi iminyaka yobudala, impilo yokuhlala phansi, cholesterol ephezulu nokubhema.
E-earlobe kunemithambo yegazi emincane eminingi eyaziwa ngokuthi ama-capillaries. Ukwehla kokugeleza kwegazi okubangelwa i-atherossteosis kubangela "ukuwa" kombhede we-vascular - futhi kukhona isibaya ku-earlobe.
Ngakho-ke, lapho kuxilongwa isibaya se-atherosclerotic endlebeni, odokotela basikisela ukuthi baxilongeke ngokujulile futhi banqume ukuba khona kwalesi sifo, noma basiphike.
Izindlela zokwelapha lesi sifo
Okokuqala, kufanele uvuselele kabusha uhlelo lwakho lokudla okunempilo, kanye nendlela yakho yokuphila. Kubalulekile ukubona inani elanele lokuzivocavoca ngokomzimba, kanye nokunamathela ekudleni okufanele.
Udinga ukudla ukudla okuphezulu kwezakhi zomzimba kanye nefayibha.
Amakilasi ejwayelekile aerobics (ngemvume kadokotela) azosiza inqubo ejwayelekile yokujikeleza kwegazi nokunciphisa cholesterol yegazi. Futhi, ukuzivocavoca kusiza ukubuyisela ukusebenza kwemisipha yenhliziyo. Kuyasiza futhi umzimba ukusebenzisa amafutha athe xaxa ne-cholesterol yamandla.
Odokotela batusa ukulandela lecebo:
- Phuza izingilazi ezi-8 zamanzi ahlanzekile nsuku zonke.
- Gcina isisindo somzimba esijwayelekile.
- Ungabhemi. Isimo sentuthu kagwayi singadala ukugoqeka kwemithambo yegazi.
- Khawulela i-caffeine kuphela izinkomishi ezi-2 (kufaka phakathi iziphuzo ezingekho fat nama-carbonated) ngosuku. Vikela ngokuphelele uma i-arrhythmia ikhona.
Ungaphuza nemithi ekhethekile ngokwelashwa noma ngamakhambi. Kukhona izakhiwo ezikhethekile zamavithamini ezinama-antioxidants asebenza kahle kakhulu.
Imithamo efanelekile (hhayi mincane) yamavithamini we-antioxidant, kufaka phakathi uC, E ne-beta-carotene, nama-vithamini B ayinkimbinkimbi, i-magnesium, i-selenium ne-bioflavonoids abaluleke kakhulu enhliziyweni.
Kungakho nge-atherosclerosis yendlebe kubaluleke kakhulu ukuthatha amavithamini B ongeziwe. Amavithamini B (ikakhulukazi i-B6, B12 ne-folic acid) anciphisa i-homocysteine, ingozi ezimele yesifo senhliziyo, okuyinto, ngokusho kwabacwaningi abaningi, ebaluleke kakhulu kune-cholesterol.
Kodwa-ke, indlela ephumelela kunazo zonke ukuhlinzwa. Kuyinto ukungenelela kokuhlinzwa okuzosiza ukugwema imiphumela futhi kuvimbele ukulimala kokuzwa esikhathini esizayo.
Ukusetshenziswa kwamavithamini
Uma sikhuluma ngama-vitamin complexes azosiza ukunciphisa umphumela omubi we-atherosulinosis yendlebe, khona-ke ungaba ufulawa wefilakisi.
2 wezipuni ngosuku ngokudla kungenza ngokwejwayelekile inani lamavithamini emzimbeni.
Ungasebenzisa amaphilisi wembewu wefilakisi. Amaphilisi ama-2-4 izikhathi ezi-3 ngosuku, uhla lwamthamo oluvunyelwe lusuka kumaphilisi ayi-6 kuye kwayi-12 ngosuku, lungaqhakambisa ukwakheka kwamavithamini emzimbeni.
Odokotela batusa ukusebenzisa uwoyela wembewu yefilakisi isipuni esisodwa ngosuku, uwoyela wezinhlanzi emaphabhulini ama-1-2 amaphilisi, izikhathi ezi-3 ngosuku ngokudla (umthamo wokuqondisa: amaphilisi angama-3-6 ngosuku).
Ungasebenzisa i-CoQ10 ngesikhathi sokwelashwa: 50-300 mg ngosuku. Kuyi-antioxidant enamandla ekhiqizwa umzimba, futhi ngeminyaka, ukukhiqizwa kwale ngxenye esebenzayo kuyancipha.
I-CoQ10 ibaluleke kakhulu uma izinkinga zendlebe zihambisana nesifo senhliziyo.
Umthamo uzoncika ebubini besifo. Imithamo ephansi ingasetshenziswa ukugcina impilo, imithamo ephezulu ye-arrhythmias, angina pectoris, and atherossteosis.
Njengokwelashwa okwengeziwe, ungasebenzisa:
- I-L-carnitine: i-1 cap (250 mg), amahlandla ama-3 nsuku zonke nokudla.
- I-Bromelain: I-1 cap (ama-microns angama-2400), amahlandla ama-3 ngosuku phakathi kokudla.
Kepha, ngeqiniso, ukuthatha konke lokhu okungezelelweyo akuyona indawo yokuhlinzwa. Le ndlela yokwelashwa kungenzeka ukuthi isetshenziswe njenge-prophylaxis, kunokuba njengezokwelapha eziphambili.
Kungani i-arteriosulinosis yenzeka?
Omunye umbono uthi i-atherosclerosis iba khona ngenxa yokonakala okuphindaphindayo kokufakwa kwangaphakathi kwethambo lomthambo.
I-Trauma ingavusa ukukhula kweseli njengengxenye yenqubo yokuvuvukala.
Lokhu kujwayelekile, impendulo yokwelashwa ebuhlungwini empeleni kungaholela ekwandeni kwethala lamakhemikhali.
Lokhu kulimala kungabangelwa noma yimuphi umcimbi, kufaka phakathi:
- Ukucindezela ngokomzimba ezicutshini zomkhumbi okwaziwa ukuthi udalwe ngumfutho wegazi ophakeme.
- Impendulo yokutheleleka odongeni lwe-artery.
- Umonakalo we-oxterative we-Arterial. Ukulimala kwe-Oxidative kubhekisa ekulimaleni okubangelwa ama-molecule angazinzile abizwa ngokuthi yi-free radicals. Ama-radicals wamahhala akhiwa ngesikhathi sokuphendula phakathi kwe-oxygen ne-LDL ("bad" cholesterol noma low density lipoprotein).
I-cholesterol ye-Oxidized LDL ingadala ukulimala odongeni lomgogodla wegazi futhi inikele ekuphenduleni okunethonya ekwakhiweni kwama-cholesterol deposits.
Kungani amazinga aphezulu e-cholesterol efaka isandla ekwakhekeni kwamakhemikhali e-cholesterol akwaziwa ngokuqinisekile.
I-cholesterol ijwayele ukutholakala kuwo wonke ama-membrane amaseli, kepha ingaguqula izindawo ezibonakalayo odongeni lomkhumbi wegazi, okwenza umkhumbi onjalo ube sengozini enkulu futhi uvame ukulimala.
Ukubhema kudlala indima enkulu ekuthuthukiseni i-atherosclerosis. I-Carbon monoxide ne-nicotine equkethe intuthu kagwayi kuthinta ukuhamba kwegazi, okungukuthi.
- lula inqubo yokungena kwe-cholesterol lipoproteins ezindongeni zemithambo yegazi;
- neqhaza ekwakhekeni kwesikali se-fibrous;
Ngaphezu kwalokho, izakhi zentuthu kagwayi zinegalelo ekwakhekeni kweziqeshana zegazi, ezingavimba ngokuphelele i-lumen yemithambo yegazi.
Ngabe isifo samathambo sibangela kanjani i-aneurysm ye-aortic?
I-Atherossteosis ingenye yezimbangela eziphambili ze-aneurysm yesisu. Udonga lwe-aorta (nayo yonke imithambo yegazi) luyisicubu esisebenzayo esakhiwa amaseli aphilayo adinga izakhamzimba nomoya-mpilo.
Iningi lalezi zakhi lingena lisuka ngaphakathi liqonde odongeni ukuze ligcwalise lonke elinye isitsha segazi.
Lapho ingaphakathi lomkhumbi limbozwe nge-atherosselotic plaque, izakhi zomzimba azisakwazi ukungena ngamanani anele.
Amaseli awatholi i-oksijini - i-hypoxia iyakhula, okuholela ekufeni kwamanye amaseli. Njengoba i-atherosulinosis iqhubeka, amangqamuzana ayaqhubeka nokufa, okuholela kumonakalo omkhulu odongeni lwe-vascular.
Ngesinye isikhathi, ubudlelwano obubucayi bufinyeleleka phakathi kwengcindezi etholakala emthanjeni wegazi, ukungezwani kodonga namandla odonga uqobo.
Lapho leli phuzu lifinyelelwa, udonga luqala ukwanda (landa) endaweni ye-plaque. Njengoba ububanzi bomkhumbi banda, ukucindezela kodonga kuyanda, okuholela ekunwetshisweni okukhulu kakhulu. Umphumela wokuphela kwenqubo enjalo ukwakhiwa kwe-aneurysm.
Kungenxa yalolu hlelo ukuthi isibambo esingeziwe endlebeni sakhiwa, okukhombisa ukuba khona kwe-pathology emzimbeni.
Yini okufanele ikhunjulwe lapho kukhonjwa isifo?
E-Russia, inani elikhulu labantu libulawa yisifo i-atherosranceotic of the coronary and carotid arriers ngaphandle kwezici nezimpawu ezivele zakhona. Kodwa-ke, iningi leziguli alifinyeleli eminyakeni engamashumi ayisithupha.
Ukuchazwa kwe-Diagonal folds ye-earlobe (DELC) kuchazwe ezincwadini zezokwelapha njengophawu lwe-surrogate olungakhomba iziguli ezisengozini enkulu ene-atherossteosis. Kodwa-ke, lesi sihloko asifundiswanga ngemininingwane eminingi.
Iningi lemibiko yemitholampilo, i-angiographic, ne-post-mortem isekela isiphakamiso sokuthi i-DELC iyisici esibalulekile esivelanayo somzimba esingakhomba iziguli ezisengozini enkulu ye-coronary artery atherossteosis.
Abanye abacwaningi abayisekeli le nkulumo. Muva nje, izifundo ezisebenzisa i-B-mode ultrasound zixhumanise i-DELC ne-carotid arteriosulinosis noma zingakhombisa ukuxhumana phakathi kwe-DELC ne-atheroscopy ye-carotid artery ebalulwe kuma-radiographs wephaneli.
Ngokuhambisana nomlando wezokwelapha wesiguli kanye ne-X-ray ye-panoramic, i-DELC kungaba wubufakazi bokwanda kwengozi ye-atherosclerotic.
Akusizi ngalutho ukusho ngokusobala ukuthi ukungabikho kwalayini kusho ukungabikho kokugula. Ukuqinisekisa ngokunembile ukutholwa, noma ukungatholakali kwayo, kubalulekile ukuthi uhlolwe ngokuphelele. Kungemva kwalokhu kuphela lapho kudingeka ukunquma ukwelashwa futhi, ngaphezu kwalokho, ukwenza ukungenelela kokuhlinzwa.
Kepha ushintsho ngendlela yokuphila ejwayelekile ukuze uthuthuke kahle lwamukeleka kahle ngaphandle kokuxilongwa. Isibonelo, uma uyeka ukubhema, ngena kwezemidlalo udle ngakwesokudla, ungakuqinisa kahle inhlala-kahle yakho.
Ungayelapha kanjani i-atherosclerosis izotshela uchwepheshe kule vidiyo ekulesi sihloko.
