I-cholesterol iyi-lipid ebalulekile, ukuba khona kwayo kubalulekile kunoma yisiphi isidalwa esiphilayo. Ama-molecule e-cholesterol angama-hydrophobic subunits otshwala be-polyhydric, amaningi awo ahlanganiswa kakhulu emzimbeni. Ukukhetha okungafanele kanye nokudla okune-cholesterol kwansuku zonke kungandisa ama-lipids endo native. Ngokungalingani kwe-lipids esegazini, i-pathologies ye-cardiological and vascular grow. Esikhathini sokuqala, lesi sifo siyasentuleka. Izikhalazo ezifakiwe zivela kuphela ezigabeni eziphinyisiwe.
Izimpawu zokuqala ze-atherosulinosis zivele kuphela ngokuvinjezelwa komkhumbi ngamaphesenti angaphezu kwangama-50. Sekuyisibonakaliso esincane kunazo zonke sesifo, kubalulekile ukufuna izeluleko zikadokotela onguchwepheshe. Akuzona zonke iziguli ezaziyo ukuthi yimuphi udokotela ophatha i-cholesterol. Lokhu kubuye kube nomthelela ekufuneni kokugcina kwezokwelashwa.
Izinkomba zokuxhumana nodokotela
Izinhlobo eziningi ezahlukene ze-lipids zizungeza emzimbeni womuntu.
Emzimbeni ophilile, i-lipid metabolism ejwayelekile yenzeka, ngenxa yokuthi kugcinwa ibhalansi yezinto ezinamafutha ezinhlobonhlobo.
Ngokuphazamiseka kwe-metabolic, ukwephulwa kwesilinganiso samafutha ahlukahlukene kuthuthuka, okucasulayo ukuthuthukiswa kwe-atherossteosis namanye ama-pathologies aqatha.
Imvamisa, izinhlobo ezilandelayo zama-lipid zijikeleza egazini:
- inani lama-molecule we-cholesterol;
- izingxenyana ezahlukahlukene ze-lipoprotein;
- triglycerides.
Izinga elikhulile noma elehlisiwe lanoma iyiphi yalezi zinto zedatha libonisa inqubo kungenzeka.
Izingxenyana ezilandelayo ze-lipoprotein ziyahlukaniswa:
- Ama-lipoprotein aphezulu kakhulu futhi aphezulu kakhulu anezindawo eziphikisiwe ze-antiatherosclerotic. Ukwehla kwe-HDL / HDL kuholela ekukhuleni kwe-atherosulinosis noma ukwanda kwaso.
- Ama-lipoprotein aphansi futhi aphansi kakhulu anomphumela ophambene ohambelana nalezi zinto ezingenhla. Ukwanda kokuhlushwa kwe-LDL / VLDL kuholela ekuqaleni komshini we-atherosulinotic kanye nasekuqalekeni kokusungulwa kwama-cholesterol plaques. I-Atherossteotic plaque iyingxenye ye-morphological of atherosulinosis.
Ukushintsha lezi zinhlaka kuyisizathu esihle sokubona udokotela.
Ngaphezu kwalokho, ukwelashwa kudinga izimo ezihambisana nokuba khona kwezimpawu ezisetshenziswayo, kanye nokwephulwa kwamanye paramitha we-metabolic emzimbeni.
Amaqembu anobungozi e-atherossteosis
I-Atherossteosis yisifo se-polyetiological futhi, ngokwezinga elithile, i-idiopathic.
Lokhu kusho ukuthi ngasikhathi sinye izici eziningi ziholela isiguli izehlakalo, futhi ngasikhathi sinye, azikho kulezi zinto eziyimbangela ye-100% imbangela ye-atherosclerosis.
Amaqembu alandelayo asengozini yeziguli ahlukaniswa:
- abantu abathanda indlela yokuphila engaphansi;
- ababhemayo
- umuntu okudla kwakhe kugcwele ama-carbohydrate namafutha emvelaphi yesilwane;
- izici zobulili nezobulili: amadoda amadala kuneminyaka engama-50;
- abantu abanezakhi zofuzo;
- iziguli ezihlushwa isifo senhliziyo;
- iziguli ezinesifo sikashukela
- isiguli nge-rheumatic pathology.
Abantu abasengozini badinga ukuvimbela ekuqaleni kwe-atherossteosis.
I-prophylaxis eyisisekelo isho ukusetshenziswa kwe-non-ethize engacacisi izidakamizwa kanye ne-prophylaxis ehlobene nezidakamizwa ngaphambi kokubonakaliswa komtholampilo wenqubo ye-pathological.
Ukuvimbela okuyinhloko kufaka izindlela zokuphila kanye nokuguqulwa kokudla, ukusetshenziswa kwezithako zokudla, kanye nokuqapha njalo ukuhlolwa kwegazi.
Ukuvinjwa kwesibili kusho iqoqo lezinyathelo ezihlose ukuvikela ukuthuthukiswa kwezinkinga kanye nokuqhubeka kwalesi sifo.
Lolu hlobo lokuvimbela lufanele abantu abanesizathu esimisiwe sokukhula kwe-cholesterol.
Izimpawu zokuqala ze-atherosulinosis
Funa usizo lwezokwelashwa ngisho nasezingeni lokugula. Udokotela anganikeza ukunakekelwa okugcwele kwezokwelapha futhi abe neqhaza ekwelashweni okuphelele kuphela ezigabeni ezisusiwe neziqala zalesi sifo.
Isici sesifundo sesi sifo isikhathi eside esedlule noma esingezansi. Okwamanje, kunenkambiso yokungalingani kwama-lipids, kepha akukho zikhalazo.
Ukwelapha lesi sigaba kuhle kakhulu ngokuya ngezibikezelo. Ukwelashwa kusenesikhathi kungakhuphula kakhulu amathuba okululama okugcwele futhi, ngokuvamile, kuthuthukise ikhwalithi yempilo.
Izimpawu zivele ngokuqhekeka okukhulu komkhumbi, futhi kuya ngokuqondile endaweni okuso sifo.
Izimpawu ezilandelayo ziyimpawu ye-atherosulinosis:
- Ubuthakathaka, ukukhathala, ukozela.
- Ukwephula ukunakwa, inkumbulo, imisebenzi yengqondo.
- Isiyezi nokufelwa.
- Ubuhlungu ngemuva kwe-sternum kanye nemilenze.
- Ukuzwa kokubanda, ukudonsa ezingxenyeni ezikude zezitho nezinyawo.
- Ku-atherosclerosis yesifo sikashukela semikhawulo engezansi, ukubhekelwa kwesinye isikhathi kuyaqapheleka.
- Isiguli, kwezinye izimo, singakhalaza ngomfutho wegazi ophakeme. Ukwanda komfutho wegazi ngaphezulu kwe-140 ne-90 mm RT. Ubuciko. idinga ukwelashwa kwe-hypotonic.
Izimpawu zincike ngqo ekutholakaleni kwesifo kwifomu lesifundo salo. Kodwa-ke, ukwelapha i-atherosclerosis akukazeephuze kakhulu. Noma unamafomu anzima, ungasiza isiguli siphile ngaphandle kobuhlungu nokuhlupheka.
Ezigabeni zakamuva, kunzima ukwelapha i-atherosulinosis, futhi ikhwalithi yokwelashwa ixhomeke ngqo ekuzibopheeleni kwesiguli ekwelashweni, iziqu zobudokotela kanye nesimo sezinto ezibonakalayo zesiguli.
Ithuba lokusinda lingakhuphuka ngokungenelela kokuhlinzwa engxenyeni etholwayo yomkhumbi.
Ochwepheshe be-Atherossteosis
Ukuqala ukwelashwa, okokuqala, udinga ukuqonda ukuthi yimuphi udokotela owenza i-cholesterol. Ngenxa yokuthi i-atherosclerosis yisifo se-etiology esingaziwa, odokotela bezinto ezikhethekile abangahlanganyela ekwelapheni lesi sifo.
Uma kusolwa ukuthi i-atherossteosis ne-cholesterol ephezulu iyasolwa, kungcono kakhulu ukuya kudokotela ohambele lapho uhlala khona. Umelaphi uyadingeka ukuthatha igazi ngephrofayili ye-lipid. Lesi sinyathelo isinyathelo sokuqala senqubo yokuxilonga.
Futhi, odokotela abavela kubuchwepheshe abahlobene nabo bayabandakanyeka kule pathology.
Isinyathelo esengeziwe ekuxilongeni lezi zindlela ezichazayo zokuhlolwa. Njengezindlela ezingeziwe zokuhlolwa, izinqubo ezinamazinga ahlukile wokuhlaseleka zingasebenza. Akuyona yonke imisebenzi engenziwa ngaphandle kwesikhashana.
Odokotela abalandelayo bangasiza iziguli ezine-atherosulinosis:
- udokotela womndeni anganikeza isiguli ukuhlolwa kwegazi okune-biochemical, okuzobonisa ngokunembile ukuba khona kokuphazamiseka kwe-metabolic;
- lapho eqinisekisa ukwephulwa kwe-lipid metabolism, udokotela womndeni uthumela isiguli ukubonisana nodokotela wezinhliziyo;
- udokotela wenhliziyo uchaza ukwelashwa okuphezulu;
- ukubonisana nodokotela wezokudla kuzosiza isiguli ukuthi zijwayele uhlobo lokudla kwazo ukuvimbela ukwakheka kwama-cholesterol plaque;
- ngosizo lwe-endocrinologist, ungabheka ukusebenza kwama-pancreas, kanye nezinye izitho zokucasha kwangaphakathi;
- Ukubonisana nodokotela wezitho zomzimba kuyadingeka ukukhipha isifo sesibindi.
Ukwazi ukuthi yiluphi udokotela odinga ukudinga ukuxhumana ne-cholesterol elengisiwe, kungenzeka ukuthi ugweme ukuqhubeka kwesifo bese uqala ngokushesha ukwelashwa.
Ungayelapha kanjani i-atherosclerosis izotshela uchwepheshe kule vidiyo ekulesi sihloko.
