Ngomqondo onesifo sikashukela kuyadingeka ukuqonda ubunzima nemiphumela yesifo sikashukela. Lesi simo sikhula ngokuqhamile futhi singashintshwa kalula. Kukholelwa ukuthi izinga likashukela ngokweqile kumuntu ogulayo (i-hyperglycemic state) kungaholela ekutheni abe nesifo sikashukela. Ngaphezu kwalokho, ngalesi sifo, i-coma iyabonakala:
- i-hyperosmolar;
- i-hypoglycemic (kwenzeka ngesifo sikashukela sohlobo 2);
- i-hyperlactacidemic;
- i-ketoacidotic (evame ukubonwa ngesifo sikashukela sohlobo 1).
Izimbangela eziphambili zokuthuthukiswa kwesimo se-pathological
Izici eziyinhloko eziholela ekuqaleni kokuthuthukiswa kokukhathazeka kwesifo sikashukela kufaka phakathi ngokushesha ukwanda kokuqukethwe ushukela egazini lomuntu ogulayo. Lokhu kungabangelwa, ngokwesibonelo, njengomphumela wokungahambisani nokudla okwelashwa. Iziguli ziyazi ukuthi isifo sikashukela siqala kanjani, kunzima ukuthi ungaziboni izimpawu zaso, kepha sivame ukungakunaki ukubonakaliswa kwaso, okugcwala i-coma.
Ukuntuleka kwe-insulin yangaphakathi kanye nerejimeni engalungile yokwelashwa kungadala ne-hyperglycemic coma. Imiphumela yalokhu - i-insulin ayingeni, engavumeli ukuthi ushukela acutshungulwe ezintweni ezibalulekile emzimbeni womuntu.
Isibindi esimweni esinjalo siqala ukukhiqizwa okungagunyaziwe kwe-glucose, sikholelwa ukuthi izinto ezidingekayo azange zingene emzimbeni ngokufanele ngenxa yezinga laso elinganele. Ngaphezu kwalokhu, ukukhiqizwa okusebenzayo kwezidumbu ze-ketone kuqala, okuthi, inqobo nje uma ushukela uqongelelwe ngokweqile emzimbeni, kuholela ekulahlekelweni kokungazi kahle komzimba.
Ezimweni ezinjalo, ukuba khona kwezidumbu ze-ketone kanye ne-glucose kusilinganiso esikhulu kangangokuba umzimba womuntu ogulayo umane ungakwazi ukuphendula ngokwenele enqubweni enjalo. Umphumela walokhu yi-ketoacidotic coma.
Kunezimo lapho, kanye noshukela, umzimba uqongelela ama-lactates nezinye izinto, okuvuse ukuqala komqondo we-hyperlactacidemic (hyperosmolar).
Kubalulekile ukuqaphela ukuthi akuwona wonke amacala lapho ikomidi yesifo sikashukela ibonwa khona ngesifo sikashukela esibangelwa i-glucose yegazi ngokweqile, ngoba kwesinye isikhathi kungaba ne-insulin ngokweqile. Ezimweni ezinjalo, kukhona ukwehla okubukhali koshukela wegazi kuye ngezinga elingaphansi kokujwayelekile, futhi isiguli siwela esimweni se-hypoglycemic coma.
Izimpawu zokuqalwa kwekoma
Izimpawu ze-coma ku-mellitus yesifo sikashukela ziyefana, ezisiphoqa ukuthi sifinyelele eziphethweni ezinembile kuphela ngemuva kwezifundo ezifanele zaselebhu. Ukuqala ukuthuthukiswa kwesifo sikashukela, kudingeka ushukela wegazi ongaphezulu kwama-33 mmol / ilitha (3.3-5.5 mmol / litre kuthathwa njengokujwayelekile).
Izimpawu zokuqalwa kwe-coma:
- ukuchama njalo;
- ikhanda;
- ukwehla kwesifiso sokudla;
- ukoma okwandayo;
- obuthakathaka obujwayelekile
- isasasa lezinzwa, elijika lijuluke, izimpawu okunzima ukuzazi;
- isicanucanu
- ukuhlanza (hhayi njalo).
Uma lezo zimpawu zihlala emahoreni ayi-12 kuye kwangama-24 ngaphandle kokunakekelwa okwanele nangesikhathi esifanele kwesokwelapha, isiguli singase siwele ekhefini leqiniso. Kuyinto isimilo ngaye:
- ukungabi nandaba ngokuphelele nabantu abasondele nokuthi kwenzekani;
- ukungazi kahle;
- isikhumba esomile;
- ukungazi ngokuphelele nokuphendula kunoma yisiphi isikhuthazo;
- amehlo athambile;
- ukuncipha kwenhliziyo;
- iphunga le-acetone elivela emlonyeni wesiguli;
- kwehle umfutho wegazi.
Uma sikhuluma nge-hypoglycemic coma, khona-ke kuzohlukahluka, kunciphise ezinye izimpawu. Ezimweni ezinjalo, kuzoba nomuzwa obukhali wendlala, ukwesaba, ukukhathazeka, ukuthuthumela emzimbeni, umuzwa osheshayo wokukhanya wobuthakathaka, ukujuluka.
Ungamisa ukuqala kwalesi simo ngokudla inani elincane lekapa, njengoshukela. Uma lokhu kungenziwa, khona-ke kungahle kube nokulahleka kokuqwashisa kanye nokuqala kokuquleka. Imisipha izoba sesimweni esihle isikhumba sibe mnyama.
Kutholakala kanjani ukuthi unesifo sikashukela?
Ukubona i-coma ku-mellitus yesifo sikashukela, awudingi kuphela ukuhlolwa ngodokotela, kodwa nokuhlolwa okubalulekile kwelebhu. Lokhu kufaka phakathi ukuhlolwa kwegazi okujwayelekile, i-biochemistry yomchamo, igazi, kanye nokuhlaziywa kwezinga likashukela.
Noma yiluphi uhlobo lokukhohlakala okugula kuzobonakaliswa ukuthi kukhona ushukela egazini ngaphezu kwama-33 mmol / ilitha, bese kuthi glucose kutholakale umchamo. Nge-hyperglycemic coma, ngeke zibe khona ezinye izimpawu zesimo sakho.
I-ketoacidotic coma ibonakala ngokuba khona kwezidumbu ze-ketone emchameni. Okwe-hyperosmolar, izinga eleqile le-osmolarity ye-plasma. I-Hyperlactacidemic ibonakala ngokwanda kwamazinga wegazi e-lactic acid.
Ukwelashwa kanjani?
Noma iyiphi i-coma yesifo sikashukela ifaka ukwelashwa kwayo, okokuqala, kuyadingeka ukubuyisela izinga elifanele likashukela egazini, izimpawu eziqondile zibalulekile lapha.
Lokhu kungatholakala kalula ngokuphatha i-insulin (noma i-glucose ye-hypoglycemia). Ngaphezu kwalokho, benza inkambo yokwelashwa kokungenelela, okubandakanya ama-droppers nemijovo ngezixazululo ezikhethekile ezingasusa ukwephulwa kokwakheka kwegazi, kunciphise ukomisa futhi kulethe i-acidity kunejwayelekile.
Zonke lezi zinqubo zenziwa ngokunakekelwa okuqinile kwezinsuku eziningana. Ngemuva kwalokho, isiguli singadluliselwa emnyangweni we-endocrinology, lapho isimo sakhe sizolungiswa khona, khona-ke kufanele sinamathele ngokucacile esimweni lapho ushukela, ushukela wegazi uzoba sesimweni esijwayelekile.
Isifo sikashukela sikashukela - imiphumela
Njengakwezinye izimo, uma nje ufuna usizo lwezokwelashwa olufanele ngesikhathi esifanele, kuzokwazi ukugwema ukungasebenzi kanye nokulahlekelwa ukwazi kuphela, kepha futhi nokubuyisela isimo somuntu ogulayo ezigabeni zokuqala zokukhula komqondo wesifo sikashukela. Uma lokhu kungenziwa, khona manjalo isiguli singafa. Ngokwezibalo zamanje zezokwelapha, ukushona kwabantu ekwakhiweni kwezinkinga ezinjengalesi sikashukela cishe amaphesenti ayishumi enani lilonke leziguli ezinalesi sifo.
