Isifo sikashukela esingakhokhelwe: siyini?

Isifo sikashukela yisifo esidala ukwedlula zonke kubantu abaningi. Lesi sifo senzeka ngenxa yokushoda kwe-pancreatic hormone.

Ukwelashwa kwe-pathology, kusetshenziswa imithi ekhethekile esiza ekuqiniseni izinga le-glucose egazini. Kepha kukhona amacala ukuthi ukwelashwa akunawo umphumela owufunayo.

Kulokhu, isifo sikashukela esibolile siyavela (abaningi basibiza nge-decompression sikashukela). Le ndlela yalesi sifo iyingozi enkulu. Ngokulashwa okungafanele, nokufa kungenzeka.

Izizathu zokuthuthuka kwe-pathology

Abantu abaningi bezwe ngesifo sikashukela esibolile - ukuthi imaphi amayunithi angakha lokhu. Uma ukholelwa imibhalo ye-anatomy, khona-ke lesi yisimo lapho izinga le-glucose egazini lingenakushintshwa.

Kalula nje, isifo sikashukela esigabeni sokunciphiswa yisifo lapho i-insulin noma amaphilisi kungasebenzi ukukwenza kube noshukela ngokwejwayelekile. Kungani i-pathology ikhula? Njengomthetho, ukubola kweshukela kukhula ngenxa yokudla ngokweqile. Uma umuntu edla ama-carbohydrate amaningi ayinkimbinkimbi, khona-ke i-insulin nezinye izidakamizwa azisakwazi ukuqinisa amazinga kashukela.

Futhi isifo sikashukela esibolile singakhula ngenxa ye:

1. Amasu okuphatha angalungile. Isibonelo, uma umuntu enqunywe imithamo ephansi kakhulu yezidakamizwa ezithile, khona-ke ukuthuthukiswa kokubuyiselwa kwemali akunakugwenywa. Futhi, i-pathology ingathuthuka ngenxa yokuphazamiseka kokwelashwa kwezidakamizwa.
2. Ukusetshenziswa kwezithasiselo zokudla. Ama-supplements angasebenza kuphela ngezinjongo ezisizayo. Izinto ekwakhekeni kwazo zithinta ngokungangabazeki izinga le-glucose egazini. Ukuqinisa ushukela, kufanele kusetshenziswe kuphela umuthi.
3. Ukusetshenziswa kwemithi yendabuko esikhundleni sezidakamizwa.
4. Ukuba khona kwezifo ezithathelanayo eziyingozi. I-mellitus yeshukela ebolile ingaba ngumphumela wezifo ezithile ezihambisana nokwehla komzimba nokungasebenzi komzimba.

Kuyaqapheleka ukuthi ukuwohloka kwesifo sikashukela kungakhula ngenxa yokuqothuka ngokweqile kwengqondo noma ukucindezelwa okungapheli.

Izimpawu zesifo sikashukela esibolile

Ngabe kunqunywa kanjani uhlobo 2 noma isifo sikashukela sokuqala? Ukuhlonza lesi sifo kulula. Cishe i-90% yamacala, isiguli somile.

Kuhambisana nomlomo owomile. Isiguli singaphuza kuze kufike kumalitha ayi-2,5 amanzi, kepha ukoma akupheli. Ngokuhamba kwesikhathi, lesi sibonakaliso somtholampilo singakhuphuka, noma okuphambene nalokho - ukuziqothula.

Futhi, ngesifo sikashukela esigabeni sokubuyiselwa kwemali, kuvela izimpawu ezilandelayo:

• Ukuchama ngokweqile. Izifiso ezingalungile nazo zinendawo. Kunezimo ukuthi kungakapheli ihora isiguli sinokunxusa okungaphezu kuka-2-3 ukuthi uchame. Imvamisa, lolu phawu lwenzeka ngenxa yokwanda kokuphuza kwamanzi.
• Ukudonsa ngomunwe noma ukuzindla kweminwe. Lokhu kungenxa yokuthi ngokuncipha kwesifo sikashukela, imikhumbi emincane iyathinteka.
• Ukulunywa kwesikhumba okukhulu. Ngokukhuphuka kweglucose yegazi, kuyanda.

Ngokwenyuka okubukhali koshukela wegazi, ubulukhuni balezi zibonakaliso zomtholampilo buphakeme kakhulu. Kepha ngesifo sikashukela sohlobo 2 esigabeni sokubuyiselwa kwemali, izimpawu ezingenhla aziphinyiswa.

Ngaphezu kwalokho, kunezimo zokuthi lapho esesimweni sokunciphiswa kwanoma yiziphi izimpawu zesifo sikashukela zingekho ngokuphelele.

Yingakho i-pathology ivame ukutholwa sekwephuze kakhulu.

Izindlela zokutholwa kwe-pathology

Ungabona kanjani ukubola kweshukela? Khomba le ndlela yokusebenza usebenzisa izinqubo ezithile. Kunezindlela ezi-3 eziphambili zokubuyiswa kwemali - izinga ushukela emchameni, izinga loshukela egazini esiswini esingenalutho nangemva kokudla, i-glycated hemoglobin.

Kufanele futhi unake ngokukhethekile izinga lama-triglycerides, umfutho wegazi, i-cholesterol yegazi, inkomba yomzimba (efushaniswa ne-BMI).

Isigaba esinqunyiwe sikashukela sibonisa izinkomba ezinjalo:

1. Ukusheshisa amazinga kashukela egazi angaphezu kuka-7.8 mmol / L.
2. Izinkomba zikashukela wegazi ngemuva kokudla ngaphezu kwama-10 mmol l.
3. Amazinga kashukela we-urine adlula i-0.5%.
4. I-Glycosylated hemoglobin ingaphezulu kuka-7.5%. Ngaphezu kwalokho, isimo sale nkomba singu-6%.
5. I-cholesterol ephelele nayo iphakeme. Njengomthetho, lidlula i-6.5-6.6 mmol l.
6. Izinga le-triglycerides lenyuka kakhulu - ngu-2.2 mmol l.
7. Umfutho wegazi kumaphesenti ayi-100 wamacala ukhuphuka kakhulu. Njengoba wazi, isimo sale nkomba singu-120 80 mm Hg. Uma isiguli sinesifo sikashukela sohlobo loku-1 noma sohlobo 2 esigabeni sokubuyiselwa kwemali, khona-ke inkomba yomfutho wegazi ingu-160 95 mm Hg.
8. BMI nayo iyanda. Ngokuvamile, ngokubola, isiguli sikhula ukukhuluphala.

Ungalawula izinkomba ezibaluleke kakhulu ekhaya. Ukuze wenze lokhu, kwanele ukuba ne-glucometer. Ngalo, ungabheka njalo amazinga kashukela wegazi. Kunconywa ukukala le nkomba esiswini esingenalutho, nangemva kwamahora ayi-1.5-2 ngemuva kokudla.

Kungenzeka futhi ukuthola izinga likashukela kanye ne-acetone kumchamo ekhaya. Ukuze wenze lokhu, sebenzisa imichilo ekhethekile yokuhlola. Zingathengwa kunoma iyiphi ikhemisi ngaphandle kwencwadi kadokotela.

Ukwelashwa kanye nezinkinga zesifo sikashukela esibolile

Ayikho indlela eqondile yokwelapha i-pathology, ngoba ikhula njengomphumela wokungalandelani kwezinkambiso nemithetho ethile. Ukuze sinciphise ubungozi bokuqhubeka kwezifo, kufanele kulandelwe imithetho ethile.

Okokuqala, udinga ukudla ukudla okulinganiselayo. Uma isiguli sidla inani elikhulu lokudla okuphezulu kuma-carbohydrate, lapho-ke ingozi yokuthola isifo sikashukela ebolile iyakhula. Isiguli sidinga ukusebenzisa inani elilawulwa ngokuqinile lama-carbohydrate. Ukudla kumele kuhlanganiswe nokuzivocavoca okulinganiselayo.

Ukuze ugweme ukuthuthukiswa kokubola, kuyadingeka ukuthi uhlole izinga likashukela esegazini ngezikhathi ezithile, sebenzisa izidakamizwa ngendlela esifike ngesikhathi futhi ungangeni esikhundleni semithi yokwenziwa ngemithako yokudla.

Uma ukwelashwa kungenziwa ngendlela yesikhathi, khona-ke ukuxhomekeka kwe-insulin (uhlobo lokuqala) nokungathembeki kwe-insulin (uhlobo lwesibili) isifo sikashukela esigabeni sokubuyiselwa kwemali kungadala:

• I-ketoacidosis. Le nkinga isongela impilo kakhulu esigulini. I-Ketoacidosis ihambisana nokoma ngokweqile, ikhanda, ukudinwa, isicanucanu. Ezimweni ezinzima, isiguli siphelelwa amandla. Futhi, i-ketoacidosis ebukhali ihambisana nokulahleka kokukhishwa kanye nokubonakala kwephunga le-acetone emlonyeni. Uma ungayeki le nkinga ngendlela esheshayo, isiguli siwela ebuthakathakeni. I-ketoacidosis ingabulala.
• I-Hypoglycemic noma i-hyperglycemic attack. Lezi zinkinga futhi ziyingozi kakhulu. Ngokukhululeka okungalindelekile, ukuhlaselwa okufana ne-ketoacidosis, kungaholela ekwehliseni kwesifo sikashukela. Ngokuhlasela kwe-hyperglycemic, kusetshenziswa izidakamizwa ezisiza ukwehlisa ushukela wegazi. Nge-hypoglycemia, kunalokho, kusetshenziswa izidakamizwa ezibandakanya ushukela.
• Ukuphazamiseka ohlelweni lwesisu. Ngendlela ebunjiwe yesifo sikashukela, ubungozi bokukhula kwamathambo kanye ne-osteoarthropathy luyakhula. Lokhu kuhlobene ngqo neqiniso lokuthi i-pathology ihambisana nokwephulwa kwezinhlelo zokujikeleza kwegazi kanye ne-endocrine.
• Izilonda Trophic kanye dermatosis. Lezi zingqinamba zivela ngenxa yokuphazamiseka kokuhamba kwegazi. Imvamisa ekwelashweni okungazelelwe, isiguli sikhula nge-necrosis yezingalo ezingalweni noma emilenzeni. Kulokhu, ukunqunywa kwezingalo kungadingeka.
• Ukukhuluphala kwe-dysplastic. Le nkinga iyivelakancane. Isifo sokukhuluphala kwe-dysplastic yisifo lapho amanoni asemzimbeni ophezulu aqokelelwa kakhulu. Kulokhu, imilenze yesiguli incipha isisindo.
• I-Lipodystrophy. Le pathology ihambisana nokunyamalala kwezicubu ezinamafutha endaweni yomjovo we-insulin.
• Ukuphazamiseka kokugaya ukudla. Isigaba esibunjiwe sikashukela sigqokwa ngokubonakala kwe-hemorrhagic diathesis, i-peritonitis eyingozi kanye nokopha kwangaphakathi.
• I-Cataract ne-renopathy. Ukungasebenzi kwezitho zombono ezibangelwa ushukela kungaholela ekulahlekelweni ngokuphelele kombono.
• Ukwephulwa kohlelo lwezinzwa oluphakathi. Babonakala ngesimo sokusebenza okunciphile, ukudangala, ukukhuphuka kokukhathazeka. Imemori ivama ukwanda.
• Isifo sokuqina kwemithambo yegazi

Noma ngabe welashwa ngokungafanele, ingozi yokwanda kwe-pathologies yesifo senhliziyo iyanda. Imvamisa, isifo sikashukela esibolile sibangela ukwehluleka kwenhliziyo nesifo senhliziyo. Lezi zifo ziba ezingamahlalakhona. Ezimweni ezimbi kakhulu, kuba nokuhlaselwa yinhliziyo. Uchwepheshe kule vidiyo ekulesi sihloko uzosho ngengozi ephelele yesifo sikashukela.